Ewald H. Hettema
- Molecular Biology top 2%
- Peroxisome Proliferator-Activated Receptors 37
- RNA Research and Splicing 18
- RNA modifications and cancer 16
- Fungal and yeast genetics research 8
- Biochemical and Molecular Research 5
- Microbial Metabolic Engineering and Bioproduction 5
- Metabolism, Diabetes, and Cancer 5
- Biochemistry top 1%
- Cell Biology top 2%
- Clinical Biochemistry top 2%
- Physiology top 5%
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- Autophagy in Disease and Therapy 6
- Co-authors
- Alison M. MotleyHenk F. TabakMarlene van den BergBen DistelJames M. NuttallRonald J. A. WandersCarlo W.T. van RoermundDominic Hoepfner
- Partner nations
- United KingdomNetherlandsGermany
In The Last Decade
Ewald H. Hettema
50 papers receiving 3.5k citations
Peers
Comparison fields: 5 of 92
- Molecular Biology 3.1k
- Biochemistry 322
- Cell Biology 624
- Clinical Biochemistry 249
- Physiology 97
Countries citing papers authored by Ewald H. Hettema
This map shows the geographic impact of Ewald H. Hettema's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ewald H. Hettema with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ewald H. Hettema more than expected).
Fields of papers citing papers by Ewald H. Hettema
This network shows the impact of papers produced by Ewald H. Hettema. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ewald H. Hettema. The network helps show where Ewald H. Hettema may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Ewald H. Hettema, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2023 | 3 | |
| 2 | 2023 | 1 | |
| 3 | 2023 | 1 | |
| 4 | 2023 | 0 | |
| 5 | 2022 | 3 | |
| 6 | 2017 | 31 | |
| 7 | 2015 | 109 | |
| 8 | 2014 | 60 | |
| 9 | 2014 | 53 | |
| 10 | 2010 | 90 | |
| 11 | 2009 | 53 | |
| 12 | 2007 | 208 | |
| 13 | 2004 | 124 | |
| 14 | 2003 | 171 | |
| 15 | 2001 | 264 | |
| 16 | 2000 | 94 | |
| 17 | 2000 | 14 | |
| 18 | 1999 | 116 | |
| 19 | 1999 | 21 | |
| 20 | 1995 | 14 |
About Ewald H. Hettema
Ewald H. Hettema is a scholar working on Molecular Biology, Aging and Cell Biology, having authored 51 papers that have together received 3.6k indexed citations. Recurring topics across this work include Peroxisome Proliferator-Activated Receptors (37 papers), RNA Research and Splicing (18 papers), RNA modifications and cancer (16 papers), Fungal and yeast genetics research (8 papers), Autophagy in Disease and Therapy (6 papers), Biochemical and Molecular Research (5 papers), Microbial Metabolic Engineering and Bioproduction (5 papers) and Metabolism, Diabetes, and Cancer (5 papers). The work is most often cited by research in Molecular Biology (3.1k citations), Biochemistry (322 citations) and Cell Biology (624 citations). Ewald H. Hettema has collaborated with scholars based in United Kingdom, Netherlands and Germany. Frequent co-authors include Alison M. Motley, Henk F. Tabak, Marlene van den Berg, Ben Distel, James M. Nuttall, Ronald J. A. Wanders, Carlo W.T. van Roermund, Dominic Hoepfner, Peter Philippsen and Marc Ouellette. Their work appears in journals such as Nature Genetics, The Journal of Cell Biology and The EMBO Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.