Eva‐Maria Gehring

451 total citations
11 papers, 392 citations indexed

About

Eva‐Maria Gehring is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Surgery. According to data from OpenAlex, Eva‐Maria Gehring has authored 11 papers receiving a total of 392 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 4 papers in Cellular and Molecular Neuroscience and 3 papers in Surgery. Recurrent topics in Eva‐Maria Gehring's work include Ion channel regulation and function (7 papers), Drug Transport and Resistance Mechanisms (2 papers) and Neuroscience and Neuropharmacology Research (2 papers). Eva‐Maria Gehring is often cited by papers focused on Ion channel regulation and function (7 papers), Drug Transport and Resistance Mechanisms (2 papers) and Neuroscience and Neuropharmacology Research (2 papers). Eva‐Maria Gehring collaborates with scholars based in Germany, Austria and United States. Eva‐Maria Gehring's co-authors include Florian Läng, Michael Föller, Mentor Sopjani, Michael Föller, Sylvia Aust, Walter Jäger, Adrian M. Senderowicz, Christos Stournaras, Ekaterina Shumilina and Shuchen Gu and has published in prestigious journals such as Genetics, The FASEB Journal and Biochemical and Biophysical Research Communications.

In The Last Decade

Eva‐Maria Gehring

11 papers receiving 388 citations

Peers

Eva‐Maria Gehring
Guoyu Ling United States
Chee Gee See United Kingdom
Laura L. Wootton United Kingdom
Sufang Yao United States
Guoyu Ling United States
Eva‐Maria Gehring
Citations per year, relative to Eva‐Maria Gehring Eva‐Maria Gehring (= 1×) peers Guoyu Ling

Countries citing papers authored by Eva‐Maria Gehring

Since Specialization
Citations

This map shows the geographic impact of Eva‐Maria Gehring's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva‐Maria Gehring with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva‐Maria Gehring more than expected).

Fields of papers citing papers by Eva‐Maria Gehring

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva‐Maria Gehring. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva‐Maria Gehring. The network helps show where Eva‐Maria Gehring may publish in the future.

Co-authorship network of co-authors of Eva‐Maria Gehring

This figure shows the co-authorship network connecting the top 25 collaborators of Eva‐Maria Gehring. A scholar is included among the top collaborators of Eva‐Maria Gehring based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva‐Maria Gehring. Eva‐Maria Gehring is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Gehring, Eva‐Maria, Nicole Heise, Ekaterina Shumilina, et al.. (2011). Stimulation of Ca 2+ ‐channel Orai1/STIM1 by serum‐and glucocorticoid‐inducible kinase 1 (SGK1). The FASEB Journal. 25(6). 2012–2021. 82 indexed citations
2.
3.
Rexhepaj, Rexhep, Miribane Dërmaku‐Sopjani, Eva‐Maria Gehring, et al.. (2010). Stimulation of Electrogenic Glucose Transport by Glycogen Synthase Kinase 3. Cellular Physiology and Biochemistry. 26(4-5). 641–646. 37 indexed citations
4.
Föller, Michael, Hasan Mahmud, Yuliya Kucherenko, et al.. (2009). Modulation of suicidal erythrocyte cation channels by an AMPA antagonist. Journal of Cellular and Molecular Medicine. 13(9b). 3680–3686. 19 indexed citations
5.
Gu, Shuchen, Natalia Papadopoulou, Eva‐Maria Gehring, et al.. (2009). Functional membrane androgen receptors in colon tumors trigger pro-apoptotic responses in vitro and reduce drastically tumor incidence in vivo. Molecular Cancer. 8(1). 114–114. 67 indexed citations
6.
Rexhepaj, Rexhep, Anand Rotte, Daniela S. Kempe, et al.. (2009). Stimulation of electrogenic intestinal dipeptide transport by the glucocorticoid dexamethasone. Pflügers Archiv - European Journal of Physiology. 459(1). 191–202. 15 indexed citations
7.
Gehring, Eva‐Maria, Fabian Klaus, Mentor Sopjani, et al.. (2009). Regulation of the Glutamate Transporter EAAT2 by PIKfyve. Cellular Physiology and Biochemistry. 24(5-6). 361–368. 50 indexed citations
8.
Fedorenko, Olga, Cai Tang, Mentor Sopjani, et al.. (2009). PIP5K2A-dependent regulation of excitatory amino acid transporter EAAT3. Psychopharmacology. 206(3). 429–435. 23 indexed citations
9.
Gehring, Eva‐Maria, Rebecca S. Lam, Gulab Dattrao Siraskar, et al.. (2009). PIKfyve upregulates CFTR activity. Biochemical and Biophysical Research Communications. 390(3). 952–957. 17 indexed citations
10.
Jäger, Walter, et al.. (2003). Biliary excretion of flavopiridol and its glucuronides in the isolated perfused rat liver: role of multidrug resistance protein 2 (Mrp2). Life Sciences. 73(22). 2841–2854. 39 indexed citations
11.
Jäger, Walter, et al.. (2003). The role of hepatic Mrp2 in the interaction of flavopiridol and bilirubin: impact on therapy. International Journal of Clinical Pharmacology and Therapeutics. 41(12). 610–611. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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