Eva Medina

8.8k total citations · 1 hit paper
128 papers, 6.6k citations indexed

About

Eva Medina is a scholar working on Infectious Diseases, Immunology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Eva Medina has authored 128 papers receiving a total of 6.6k indexed citations (citations by other indexed papers that have themselves been cited), including 64 papers in Infectious Diseases, 47 papers in Immunology and 42 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Eva Medina's work include Antimicrobial Resistance in Staphylococcus (48 papers), Streptococcal Infections and Treatments (40 papers) and Neonatal and Maternal Infections (27 papers). Eva Medina is often cited by papers focused on Antimicrobial Resistance in Staphylococcus (48 papers), Streptococcal Infections and Treatments (40 papers) and Neonatal and Maternal Infections (27 papers). Eva Medina collaborates with scholars based in Germany, United States and Sweden. Eva Medina's co-authors include Oliver Goldmann, Manfred Rohde, Carlos A. Guzmán, Gursharan S. Chhatwal, Dietmar H. Pieper, Maren von Köckritz‐Blickwede, Georg Peters, R J North, Anna Norrby‐Teglund and Andreas Beineke and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Angewandte Chemie International Edition and Nature Communications.

In The Last Decade

Eva Medina

126 papers receiving 6.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Immune-responsive gene 1 protein links metabolism to immunity by catalyzing itaconic acid production

2013 857 citations Oliver Goldmann, Eva Medina et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Eva Medina Germany	44	2.7k	2.1k	2.0k	1.1k	1.1k	128	6.6k
Marcelo B. Sztein United States	60	3.7k 1.3×	4.0k 1.9×	1.9k 0.9×	1.5k 1.4×	1.4k 1.3×	235	10.1k
Luisa Martı́nez-Pomares United Kingdom	48	4.5k 1.7×	1.2k 0.6×	2.7k 1.3×	1.3k 1.2×	630 0.6×	88	8.8k
David H. Dockrell United Kingdom	50	2.6k 1.0×	1.2k 0.6×	2.0k 1.0×	2.2k 2.0×	483 0.5×	143	7.3k
Reinhard Würzner Austria	50	3.6k 1.3×	2.3k 1.1×	886 0.4×	1.9k 1.7×	529 0.5×	255	7.4k
Trine H. Mogensen Denmark	36	3.9k 1.4×	1.3k 0.6×	2.1k 1.0×	1.9k 1.7×	399 0.4×	116	7.0k
Adeline M. Hajjar United States	34	4.5k 1.7×	864 0.4×	1.9k 0.9×	1.4k 1.3×	675 0.6×	62	7.4k
Harm HogenEsch United States	49	3.0k 1.1×	1.2k 0.6×	2.4k 1.2×	946 0.9×	255 0.2×	166	6.8k
Kohtaro Fujihashi United States	53	5.0k 1.9×	1.3k 0.6×	1.5k 0.7×	1.5k 1.4×	336 0.3×	169	8.3k
Sérgio C. Oliveira Brazil	50	2.8k 1.0×	1.3k 0.6×	2.5k 1.2×	1.7k 1.5×	987 0.9×	266	8.6k
Klaus Heeg Germany	54	7.5k 2.8×	1.1k 0.5×	2.8k 1.4×	1.7k 1.6×	859 0.8×	223	11.5k

Countries citing papers authored by Eva Medina

Since Specialization

Citations

This map shows the geographic impact of Eva Medina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Medina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Medina more than expected).

Fields of papers citing papers by Eva Medina

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Medina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Medina. The network helps show where Eva Medina may publish in the future.

Co-authorship network of co-authors of Eva Medina

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Medina. A scholar is included among the top collaborators of Eva Medina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Medina. Eva Medina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Hosseini, Shirin, Niklas Lonnemann, Wei He, et al.. (2024). The potential therapeutic role of itaconate and mesaconate on the detrimental effects of LPS-induced neuroinflammation in the brain. Journal of Neuroinflammation. 21(1). 207–207. 10 indexed citations

Goldmann, Oliver & Eva Medina. (2024). Metabolic pathways fueling the suppressive activity of myeloid-derived suppressor cells. Frontiers in Immunology. 15. 1461455–1461455. 5 indexed citations

Horst, Sarah A., Sudip Das, Bruno Hüettel, et al.. (2020). Identification of a Novel LysR-Type Transcriptional Regulator in Staphylococcus aureus That Is Crucial for Secondary Tissue Colonization during Metastatic Bloodstream Infection. mBio. 11(4). 11 indexed citations

Zhao, Weining, Caillan Crowe‐McAuliffe, Joanna Krysiak, et al.. (2019). Der Naturstoff Elegaphenon verstärkt antibiotische Effekte gegen Pseudomonas aeruginosa. Angewandte Chemie. 131(25). 8670–8674. 1 indexed citations

Zhao, Weining, Caillan Crowe‐McAuliffe, Joanna Krysiak, et al.. (2019). The Natural Product Elegaphenone Potentiates Antibiotic Effects against Pseudomonas aeruginosa. Angewandte Chemie International Edition. 58(25). 8581–8584. 16 indexed citations

Formaglio, Pauline, Oliver Goldmann, Susanne Engelmann, et al.. (2019). Longitudinal proliferation mapping in vivo reveals NADPH oxidase-mediated dampening of Staphylococcus aureus growth rates within neutrophils. Scientific Reports. 9(1). 5703–5703. 7 indexed citations

Medina, Eva & Dominik Hartl. (2018). Myeloid-Derived Suppressor Cells in Infection: A General Overview. Journal of Innate Immunity. 10(5-6). 407–413. 86 indexed citations

Fetzer, Christian, Vadim S. Korotkov, Robert Thänert, et al.. (2017). Verringerung der Virulenz von multiresistentem Staphylococcus aureus mithilfe eines chemischen Disruptors des ClpX‐Chaperon‐Komplexes. Angewandte Chemie. 129(49). 15952–15957. 2 indexed citations

Leech, John, Keenan A. Lacey, Michelle E. Mulcahy, Eva Medina, & Rachel M. McLoughlin. (2017). IL-10 Plays Opposing Roles during Staphylococcus aureus Systemic and Localized Infections. The Journal of Immunology. 198(6). 2352–2365. 70 indexed citations

10.

Fetzer, Christian, Vadim S. Korotkov, Robert Thänert, et al.. (2017). A Chemical Disruptor of the ClpX Chaperone Complex Attenuates the Virulence of Multidrug‐Resistant Staphylococcus aureus. Angewandte Chemie International Edition. 56(49). 15746–15750. 33 indexed citations

11.

Goldmann, Oliver & Eva Medina. (2017). Staphylococcus aureus strategies to evade the host acquired immune response. International Journal of Medical Microbiology. 308(6). 625–630. 53 indexed citations

12.

Tsatsaronis, James A., Aleta Pupovac, Oliver Goldmann, et al.. (2015). Group A <b><i>Streptococcus</i></b> Modulates Host Inflammation by Manipulating Polymorphonuclear Leukocyte Cell Death Responses. Journal of Innate Immunity. 7(6). 612–622. 6 indexed citations

13.

Bergmann, Simone, et al.. (2013). Lung dendritic cells facilitate extrapulmonary bacterial dissemination during pneumococcal pneumonia. Frontiers in Cellular and Infection Microbiology. 3. 21–21. 25 indexed citations

14.

Wos‐Oxley, Melissa L., André Bleich, Andrew P. A. Oxley, et al.. (2012). Comparative evaluation of establishing a human gut microbial community within rodent models. Gut Microbes. 3(3). 234–249. 108 indexed citations

15.

Köckritz‐Blickwede, Maren von, Oliver Goldmann, Pontus Thulin, et al.. (2008). Phagocytosis-independent antimicrobial activity of mast cells by means of extracellular trap formation. Blood. 111(6). 3070–3080. 443 indexed citations

16.

Dinkla, Katrin, et al.. (2007). Upregulation of capsule enables Streptococcus pyogenes to evade immune recognition by antigen-specific antibodies directed to the G-related α2-macroglobulin-binding protein GRAB located on the bacterial surface. Microbes and Infection. 9(8). 922–931. 30 indexed citations

17.

Rasmussen, Magnus, et al.. (2003). Contribution of Protein G–Related α₂‐Macroglobulin–Binding Protein to Bacterial Virulence in a Mouse Skin Model of Group A Streptococcal Infection. The Journal of Infectious Diseases. 187(11). 1694–1703. 23 indexed citations

18.

Medina, Eva, Paola Paglia, Manfred Rohde, Mario P. Colombo, & Carlos A. Guzmán. (2000). Modulation of host immune responses stimulated bySalmonella vaccine carrier strains by using different promoters to drive the expression of the recombinant antigen. European Journal of Immunology. 30(3). 768–777. 24 indexed citations

19.

Medina, Eva, et al.. (2000). Modulation of immune responses following antigen administration by mucosal route. FEMS Immunology & Medical Microbiology. 27(4). 305–311. 40 indexed citations

20.

Borthwick, Nicola, Margarita Bofill, Wendy M. Gombert, et al.. (1994). Lymphocyte activation in HIV-1 infection. II. Functional defects of CD28− T cells. AIDS. 8(4). 431–442. 180 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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