Eva M. Jiménez‐Mateos

4.2k total citations
73 papers, 3.2k citations indexed

About

Eva M. Jiménez‐Mateos is a scholar working on Molecular Biology, Cancer Research and Cellular and Molecular Neuroscience. According to data from OpenAlex, Eva M. Jiménez‐Mateos has authored 73 papers receiving a total of 3.2k indexed citations (citations by other indexed papers that have themselves been cited), including 47 papers in Molecular Biology, 31 papers in Cancer Research and 17 papers in Cellular and Molecular Neuroscience. Recurrent topics in Eva M. Jiménez‐Mateos's work include MicroRNA in disease regulation (31 papers), Circular RNAs in diseases (15 papers) and Neuroscience and Neuropharmacology Research (12 papers). Eva M. Jiménez‐Mateos is often cited by papers focused on MicroRNA in disease regulation (31 papers), Circular RNAs in diseases (15 papers) and Neuroscience and Neuropharmacology Research (12 papers). Eva M. Jiménez‐Mateos collaborates with scholars based in Ireland, Spain and United States. Eva M. Jiménez‐Mateos's co-authors include David C. Henshall, Tobías Engel, Takanori Sano, Jochen H.M. Prehn, Ross C. McKiernan, Jesús Ávila, Raymond L. Stallings, Genshin Mouri, Christian González‐Billault and Katsuhiro Tanaka and has published in prestigious journals such as Nature Medicine, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Eva M. Jiménez‐Mateos

69 papers receiving 3.2k citations

Peers

Eva M. Jiménez‐Mateos
Travis L. Unger United States
Jasper J. Anink Netherlands
Monika A. Davare United States
Stefanie Robel United States
Nadine Ruderisch Switzerland
Susan C. Su United States
James Smith United Kingdom
Eva M. Jiménez‐Mateos
Citations per year, relative to Eva M. Jiménez‐Mateos Eva M. Jiménez‐Mateos (= 1×) peers Cristina Richichi

Countries citing papers authored by Eva M. Jiménez‐Mateos

Since Specialization
Citations

This map shows the geographic impact of Eva M. Jiménez‐Mateos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva M. Jiménez‐Mateos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva M. Jiménez‐Mateos more than expected).

Fields of papers citing papers by Eva M. Jiménez‐Mateos

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva M. Jiménez‐Mateos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva M. Jiménez‐Mateos. The network helps show where Eva M. Jiménez‐Mateos may publish in the future.

Co-authorship network of co-authors of Eva M. Jiménez‐Mateos

This figure shows the co-authorship network connecting the top 25 collaborators of Eva M. Jiménez‐Mateos. A scholar is included among the top collaborators of Eva M. Jiménez‐Mateos based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva M. Jiménez‐Mateos. Eva M. Jiménez‐Mateos is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mooney, Catherine, María Luisa Scattoni, Laura Ricceri, et al.. (2025). Deregulated mRNA and microRNA Expression Patterns in the Prefrontal Cortex of the BTBR Mouse Model of Autism. Molecular Neurobiology. 62(8). 10614–10634. 1 indexed citations
2.
Murphy, Ronan P., Amaya Sanz‐Rodriguez, Tobías Engel, et al.. (2025). Shared and distinct microRNA profiles between HT22, N2A and SH-SY5Y cell lines and primary mouse hippocampal neurons. PLoS ONE. 20(12). e0326401–e0326401.
3.
Jiménez‐Mateos, Eva M., et al.. (2024). Contribution of microglia to the epileptiform activity that results from neonatal hypoxia. Neuropharmacology. 253. 109968–109968.
4.
Alves, Mariana, Alberto Parras, Giorgia Conte, et al.. (2023). The P2X7 receptor contributes to seizures and inflammation‐driven long‐lasting brain hyperexcitability following hypoxia in neonatal mice. British Journal of Pharmacology. 180(13). 1710–1729. 15 indexed citations
5.
Jiménez‐Mateos, Eva M., et al.. (2018). Regulation of P2X7 receptor expression and function in the brain. Brain Research Bulletin. 151. 153–163. 52 indexed citations
6.
Reynolds, James R., Eva M. Jiménez‐Mateos, Li Cao, et al.. (2017). Proteomic Analysis After Status Epilepticus Identifies UCHL1 as Protective Against Hippocampal Injury. Neurochemical Research. 42(7). 2033–2054. 7 indexed citations
7.
Groß, Christina, Xiaodi Yao, Tobías Engel, et al.. (2016). MicroRNA-Mediated Downregulation of the Potassium Channel Kv4.2 Contributes to Seizure Onset. Cell Reports. 17(1). 37–45. 65 indexed citations
8.
Reynolds, James R., Suzanne F. C. Miller-Delaney, Eva M. Jiménez‐Mateos, et al.. (2015). Transcriptional Response of Polycomb Group Genes to Status Epilepticus in Mice is Modified by Prior Exposure to Epileptic Preconditioning. Frontiers in Neurology. 6. 46–46. 15 indexed citations
9.
Jiménez‐Mateos, Eva M., et al.. (2015). Effects of hypoxia-induced neonatal seizures on acute hippocampal injury and later-life seizure susceptibility and anxiety-related behavior in mice. Neurobiology of Disease. 83. 100–114. 45 indexed citations
10.
Dávila, David, Eva M. Jiménez‐Mateos, Claire Mooney, et al.. (2014). Hsp27 binding to the 3′UTR ofbimmRNA prevents neuronal death during oxidative stress–induced injury: a novel cytoprotective mechanism. Molecular Biology of the Cell. 25(21). 3413–3423. 17 indexed citations
11.
Engel, Tobías, Eva M. Jiménez‐Mateos, Caoimhín G. Concannon, et al.. (2013). CHOP regulates the p53–MDM2 axis and is required for neuronal survival after seizures. Brain. 136(2). 577–592. 77 indexed citations
12.
Brennan, Gary P., Eva M. Jiménez‐Mateos, Ross C. McKiernan, et al.. (2013). Transgenic Overexpression of 14-3-3 Zeta Protects Hippocampus against Endoplasmic Reticulum Stress and Status Epilepticus In Vivo. PLoS ONE. 8(1). e54491–e54491. 44 indexed citations
13.
Miller-Delaney, Suzanne F. C., Sudipto Das, Takanori Sano, et al.. (2012). Differential DNA Methylation Patterns Define Status Epilepticus and Epileptic Tolerance. Journal of Neuroscience. 32(5). 1577–1588. 87 indexed citations
14.
Sano, Takanori, James R. Reynolds, Eva M. Jiménez‐Mateos, et al.. (2012). MicroRNA-34a upregulation during seizure-induced neuronal death. Cell Death and Disease. 3(3). e287–e287. 86 indexed citations
15.
McKiernan, Ross C., Eva M. Jiménez‐Mateos, Isabella Bray, et al.. (2012). Reduced Mature MicroRNA Levels in Association with Dicer Loss in Human Temporal Lobe Epilepsy with Hippocampal Sclerosis. PLoS ONE. 7(5). e35921–e35921. 105 indexed citations
16.
Jiménez‐Mateos, Eva M. & David C. Henshall. (2009). Seizure preconditioning and epileptic tolerance: models and mechanisms.. PubMed Central. 26 indexed citations
17.
Jiménez‐Mateos, Eva M., Seiji Hatazaki, Martha B. Johnson, et al.. (2008). Hippocampal transcriptome after status epilepticus in mice rendered seizure damage-tolerant by epileptic preconditioning features suppressed calcium and neuronal excitability pathways. Neurobiology of Disease. 32(3). 442–453. 59 indexed citations
18.
Hatazaki, Seiji, Eva M. Jiménez‐Mateos, Robert Meller, et al.. (2007). Microarray profile of seizure damage-refractory hippocampal CA3 in a mouse model of epileptic preconditioning. Neuroscience. 150(2). 467–477. 41 indexed citations
19.
González‐Billault, Christian, Eva M. Jiménez‐Mateos, Alfredo Cáceres, et al.. (2003). Microtubule‐associated protein 1B function during normal development, regeneration, and pathological conditions in the nervous system. Journal of Neurobiology. 58(1). 48–59. 83 indexed citations
20.
González‐Billault, Christian, Michael Engelke, Eva M. Jiménez‐Mateos, et al.. (2002). Participation of structural microtubule‐associated proteins (MAPs) in the development of neuronal polarity. Journal of Neuroscience Research. 67(6). 713–719. 62 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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