Eva Balada

2.0k total citations
45 papers, 1.4k citations indexed

About

Eva Balada is a scholar working on Rheumatology, Immunology and Molecular Biology. According to data from OpenAlex, Eva Balada has authored 45 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Rheumatology, 22 papers in Immunology and 10 papers in Molecular Biology. Recurrent topics in Eva Balada's work include Systemic Lupus Erythematosus Research (22 papers), Inflammatory Myopathies and Dermatomyositis (8 papers) and T-cell and B-cell Immunology (8 papers). Eva Balada is often cited by papers focused on Systemic Lupus Erythematosus Research (22 papers), Inflammatory Myopathies and Dermatomyositis (8 papers) and T-cell and B-cell Immunology (8 papers). Eva Balada collaborates with scholars based in Spain, Palestinian Territory and United States. Eva Balada's co-authors include Miquel Vilardell‐Tarrés, Josep Ordi‐Ros, Moisés Labrador‐Horrillo, Albert Selva-O’Callaghan, Luis Martínez‐Lostao, María Ángeles Martínez, Ernesto Trallero‐Araguás, C. Juárez, José Pardos‐Gea and Jesús Castro‐Marrero and has published in prestigious journals such as PLoS ONE, Annals of the New York Academy of Sciences and The American Journal of Medicine.

In The Last Decade

Eva Balada

44 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Eva Balada Spain 21 619 608 426 370 175 45 1.4k
H. Mann Czechia 23 639 1.0× 513 0.8× 559 1.3× 503 1.4× 74 0.4× 89 1.5k
María Francisca González‐Escribano Spain 25 498 0.8× 727 1.2× 260 0.6× 343 0.9× 303 1.7× 64 1.6k
Jason W. Bauer United States 9 935 1.5× 978 1.6× 293 0.7× 366 1.0× 142 0.8× 10 1.6k
Hajime Yamagata Japan 16 529 0.9× 437 0.7× 347 0.8× 248 0.7× 139 0.8× 35 1.5k
Hemlata Varsani United Kingdom 20 571 0.9× 891 1.5× 399 0.9× 607 1.6× 139 0.8× 31 1.7k
Yoshiaki Tokano Japan 22 760 1.2× 929 1.5× 207 0.5× 145 0.4× 103 0.6× 71 1.4k
Masashi Akizuki Japan 19 568 0.9× 302 0.5× 235 0.6× 456 1.2× 94 0.5× 42 1.2k
Takaki Nojima Japan 13 426 0.7× 279 0.5× 237 0.6× 360 1.0× 48 0.3× 32 898
Yoshinori Katada Japan 17 529 0.9× 462 0.8× 391 0.9× 132 0.4× 73 0.4× 46 1.4k
Maryvonne Dueymes France 21 539 0.9× 592 1.0× 288 0.7× 83 0.2× 131 0.7× 60 1.5k

Countries citing papers authored by Eva Balada

Since Specialization
Citations

This map shows the geographic impact of Eva Balada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Balada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Balada more than expected).

Fields of papers citing papers by Eva Balada

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Balada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Balada. The network helps show where Eva Balada may publish in the future.

Co-authorship network of co-authors of Eva Balada

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Balada. A scholar is included among the top collaborators of Eva Balada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Balada. Eva Balada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Marín, Ana, María Ángeles Martínez, Laura Martínez‐Martínez, et al.. (2016). Statin-associated autoimmune myopathy: A distinct new IFL pattern can increase the rate of HMGCR antibody detection by clinical laboratories. Autoimmunity Reviews. 15(12). 1161–1166. 26 indexed citations
2.
Balada, Eva, Albert Selva-O’Callaghan, Josep Ordi‐Ros, et al.. (2015). Sequence analysis ofTMEM173exon 5 in patients with systemic autoimmune diseases. Autoimmunity. 49(1). 12–16. 8 indexed citations
3.
Balada, Eva, M. Ramentol, Josep Ordi‐Ros, et al.. (2014). Prevalence of HHV-8 in systemic autoimmune diseases. Journal of Clinical Virology. 62. 84–88. 5 indexed citations
4.
5.
Balada, Eva, et al.. (2011). Enhanced transcript levels of CD48 in CD4+ T cells from systemic lupus erythematosus patients. Immunobiology. 216(9). 1034–1037. 2 indexed citations
6.
Pardos‐Gea, José, Jesús Castro‐Marrero, Josefina Cortés-Hernández, et al.. (2011). Beta2-glycoprotein I gene polymorphisms Val247Leu and Trp316Ser in Spanish patients with primary antiphospholipid syndrome. Rheumatology International. 32(4). 927–932. 3 indexed citations
7.
Castro‐Marrero, Jesús, Eva Balada, Miquel Vilardell‐Tarrés, & Josep Ordi‐Ros. (2011). The PTPN22*R620W polymorphism does not confer genetic susceptibility to antiphospholipid syndrome in the Spanish population. International Journal of Immunogenetics. 38(6). 529–531. 7 indexed citations
8.
Balada, Eva, Miquel Vilardell‐Tarrés, & Josep Ordi‐Ros. (2010). Implication of Human Endogenous Retroviruses in the Development of Autoimmune Diseases. International Reviews of Immunology. 29(4). 351–370. 79 indexed citations
9.
Balada, Eva, et al.. (2009). Serum Levels of Soluble CD40 Ligand at Flare and at Remission in Patients with Systemic Lupus Erythematosus. The Journal of Rheumatology. 36(5). 953–960. 12 indexed citations
10.
Castro‐Marrero, Jesús, Eva Balada, Miquel Vilardell‐Tarrés, & Josep Ordi‐Ros. (2009). Genetic risk factors of thrombosis in the antiphospholipid syndrome. British Journal of Haematology. 147(3). 289–296. 23 indexed citations
11.
Cortés-Hernández, Josefina, et al.. (2009). Systemic lupus erythematosus as a first presentation of common variable immunodeficiency associated with infrequent mannose-binding lectin gene polymorphisms. Rheumatology International. 31(4). 537–541. 6 indexed citations
12.
Balada, Eva, et al.. (2009). Changes in Lipid Profile Between Flare and Remission of Patients with Systemic Lupus Erythematosus: A Prospective Study. The Journal of Rheumatology. 36(8). 1639–1645. 11 indexed citations
13.
Balada, Eva, et al.. (2008). The complex immunogenetic basis of systemic lupus erythematosus. Autoimmunity Reviews. 7(5). 345–351. 44 indexed citations
14.
Balada, Eva, et al.. (2008). DNase 1 and systemic lupus erythematosus. Autoimmunity Reviews. 7(5). 359–363. 97 indexed citations
15.
16.
Balada, Eva, Carmen Pilar Simeón‐Aznar, José Ordi-Ros, et al.. (2008). Anti-PDGFR-α antibodies measured by non-bioactivity assays are not specific for systemic sclerosis. Annals of the Rheumatic Diseases. 67(7). 1027–1029. 24 indexed citations
17.
Balada, Eva, Josep Ordi‐Ros, & Miquel Vilardell‐Tarrés. (2007). DNA Methylation and Systemic Lupus Erythematosus. Annals of the New York Academy of Sciences. 1108(1). 127–136. 72 indexed citations
18.
Pardos‐Gea, José, et al.. (2007). Protein Z levels and anti-protein Z antibodies in patients with arterial and venous thrombosis. Thrombosis Research. 121(6). 727–734. 20 indexed citations
19.
González, Antonio, Isabel Ferreirós-Vidal, Eva Balada, et al.. (2006). Association of a non-synonymous single-nucleotide polymorphism of DNASEI with SLE susceptibility. Lara D. Veeken. 45(7). 819–823. 43 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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