Eva Arranz

787 total citations
24 papers, 545 citations indexed

About

Eva Arranz is a scholar working on Hematology, Genetics and Pathology and Forensic Medicine. According to data from OpenAlex, Eva Arranz has authored 24 papers receiving a total of 545 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Hematology, 9 papers in Genetics and 8 papers in Pathology and Forensic Medicine. Recurrent topics in Eva Arranz's work include Acute Myeloid Leukemia Research (10 papers), Lymphoma Diagnosis and Treatment (8 papers) and Chronic Myeloid Leukemia Treatments (7 papers). Eva Arranz is often cited by papers focused on Acute Myeloid Leukemia Research (10 papers), Lymphoma Diagnosis and Treatment (8 papers) and Chronic Myeloid Leukemia Treatments (7 papers). Eva Arranz collaborates with scholars based in Spain and United States. Eva Arranz's co-authors include Javier Benı́tez, Carmen Rivas, Mercedes Robledo, Beatriz Martı́nez-Delgado, María J. García, Françesc Solé, Juan C. Cigudosa, Marı́a José Calasanz, José Cervera and Elisa Luño and has published in prestigious journals such as SHILAP Revista de lepidopterología, Blood and PLoS ONE.

In The Last Decade

Eva Arranz

23 papers receiving 535 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Eva Arranz Spain 14 252 231 172 162 129 24 545
Stéphanie Struski France 16 171 0.7× 94 0.4× 134 0.8× 264 1.6× 112 0.9× 30 596
T Flores Spain 9 124 0.5× 126 0.5× 112 0.7× 232 1.4× 228 1.8× 21 570
Aleksandra Rizo United States 10 337 1.3× 147 0.6× 215 1.3× 314 1.9× 193 1.5× 28 655
Françoise Brizard France 14 343 1.4× 102 0.4× 252 1.5× 278 1.7× 93 0.7× 33 680
GJ van Ommen Netherlands 9 156 0.6× 254 1.1× 155 0.9× 167 1.0× 156 1.2× 13 494
JL Huret France 6 154 0.6× 301 1.3× 128 0.7× 126 0.8× 166 1.3× 100 521
J. Treleaven United Kingdom 10 354 1.4× 141 0.6× 135 0.8× 177 1.1× 214 1.7× 17 636
Dominique Mühlematter Switzerland 14 365 1.4× 102 0.4× 160 0.9× 212 1.3× 43 0.3× 25 528
Valère Cacheux France 10 174 0.7× 159 0.7× 111 0.6× 175 1.1× 121 0.9× 19 526
C Mecucci Italy 16 392 1.6× 176 0.8× 272 1.6× 293 1.8× 123 1.0× 35 765

Countries citing papers authored by Eva Arranz

Since Specialization
Citations

This map shows the geographic impact of Eva Arranz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Arranz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Arranz more than expected).

Fields of papers citing papers by Eva Arranz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Arranz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Arranz. The network helps show where Eva Arranz may publish in the future.

Co-authorship network of co-authors of Eva Arranz

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Arranz. A scholar is included among the top collaborators of Eva Arranz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Arranz. Eva Arranz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Loscertales, Javier, Eva Arranz, Javier Ortíz, et al.. (2025). T-cell prolymphocytic leukemia, a case report and review of the literature. Oncology Research Featuring Preclinical and Clinical Cancer Therapeutics. 33(3). 505–517. 1 indexed citations
2.
Rubio, Montserrat López, et al.. (2020). Jumping Translocation in a Patient with Acute Leukemia and Fatal Evolution. Case Reports in Oncology. 13(2). 1026–1030. 1 indexed citations
3.
López, Francisco, et al.. (2019). Comparison and cost analysis of three protocols for mobilization and apheresis of haematopoietic progenitor cells. Journal of Clinical Apheresis. 34(4). 461–467.
4.
5.
Cuesta‐Domínguez, Álvaro, C. Ormazábal, Marta Galán-Díez, et al.. (2012). Transforming and Tumorigenic Activity of JAK2 by Fusion to BCR: Molecular Mechanisms of Action of a Novel BCR-JAK2 Tyrosine-Kinase. PLoS ONE. 7(2). e32451–e32451. 26 indexed citations
6.
Font, Patricia, Dolores Subirá, Susana Castañón, et al.. (2006). Evaluation of CD7 and terminal deoxynucleotidyl transferase (TdT) expression in CD34+ myeloblasts from patients with myelodysplastic syndrome. Leukemia Research. 30(8). 957–963. 13 indexed citations
7.
Cigudosa, Juan C., María D. Odero, Marı́a José Calasanz, et al.. (2003). De novo erythroleukemia chromosome features include multiple rearrangements, with special involvement of chromosomes 11 and 19. Genes Chromosomes and Cancer. 36(4). 406–412. 29 indexed citations
8.
Steegmann, Juan Luis, Gemma Moreno, Santiago Osorio, et al.. (2003). Chronic myeloid leukemia patients resistant to or intolerant of interferon alpha and subsequently treated with imatinib show reduced immunoglobulin levels and hypogammaglobulinemia.. PubMed. 88(7). 762–8. 34 indexed citations
9.
10.
Renedo, Mónica, Beatriz Martı́nez-Delgado, Eva Arranz, et al.. (2001). Chromosomal changes pattern and gene amplification in T cell non-Hodgkin's lymphomas. Leukemia. 15(10). 1627–1632. 26 indexed citations
11.
Arranz, Eva, Beatriz Martı́nez-Delgado, Ana Osório, et al.. (2000). Identification by Comparative Genomic Hybridization of Genetic Changes Involved in Tumoral Progression of a T-Cell Non-Hodgkin Lymphoma. Cancer Genetics and Cytogenetics. 117(1). 41–44. 2 indexed citations
12.
Martı́nez-Delgado, Beatriz, Mercedes Robledo, Eva Arranz, et al.. (1998). Hypermethylation of p15/ink4b/MTS2 gene is differentially implicated among non-Hodgkin’s lymphomas. Leukemia. 12(6). 937–941. 33 indexed citations
13.
14.
Arranz, Eva, Mercedes Robledo, Beatriz Fernández-Martínez, et al.. (1997). Detection of trisomy 8 using conventional cytogenetic techniques and interphase FISH analysis in 34 myeloid disorders: A comparative study. Cancer Genetics and Cytogenetics. 94(2). 103–105. 3 indexed citations
15.
Martı́nez-Delgado, Beatriz, et al.. (1997). Correlation between mutations in p53 gene and protein expression in human lymphomas. American Journal of Hematology. 55(1). 1–8. 34 indexed citations
16.
Martı́nez-Delgado, Beatriz, José Fernández‐Piqueras, María J. García, et al.. (1997). Hypermethylation of a 5′ CpG island of p16 is a frequent event in non-Hodgkin’s lymphoma. Leukemia. 11(3). 425–428. 71 indexed citations
17.
Renedo, Mónica, et al.. (1997). Cytogenetic and molecular studies of siblings with ataxia telangiectasia followed for 7 years. Cancer Genetics and Cytogenetics. 95(2). 178–182. 2 indexed citations
18.
Arranz, Eva, et al.. (1996). Incidence of homogeneously staining regions in non-Hodgkin lymphomas. Cancer Genetics and Cytogenetics. 87(1). 1–3. 15 indexed citations
19.
Arranz, Eva, et al.. (1996). Rearrangement of one RAR-α gene in an acute promyelocytic leukemia case with t(15;17) and t(6;17) involving chromosomes 17 band q21. Cancer Genetics and Cytogenetics. 87(2). 172–175. 1 indexed citations
20.
Robledo, Mercedes, et al.. (1995). Genetic instability of microsatellites in hematological neoplasms.. PubMed. 9(6). 960–4. 60 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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