Eugen Mengel
- Physiology top 5%
- Molecular Biology
- Epidemiology
- Rheumatology top 10%
- Cell Biology
- Co-authors
- Roberto GiuglianiSimon JonesNathalie GuffonPaul HarmatzMelissa WassersteinMargaret M. McGovernBruno BembiBarbara K. Burton
- Topics
- Lysosomal Storage Disorders Research (19 papers)Cellular transport and secretion (7 papers)Trypanosoma species research and implications (4 papers)
- Cited by
- PhysiologyRheumatologyHematology
- Partner nations
- United StatesFranceGermany
In The Last Decade
Eugen Mengel
20 papers receiving 477 citations
Peers
Comparison fields: 5 of 52
- Physiology 430
- Molecular Biology 144
- Epidemiology 132
- Rheumatology 98
- Cell Biology 61
Countries citing papers authored by Eugen Mengel
This map shows the geographic impact of Eugen Mengel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eugen Mengel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eugen Mengel more than expected).
Fields of papers citing papers by Eugen Mengel
This network shows the impact of papers produced by Eugen Mengel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eugen Mengel. The network helps show where Eugen Mengel may publish in the future.
Co-authorship network of co-authors of Eugen Mengel
This figure shows the co-authorship network connecting the top 25 collaborators of Eugen Mengel. A scholar is included among the top collaborators of Eugen Mengel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eugen Mengel. Eugen Mengel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 1 | |
| 3 | 7 | |
| 4 | 1 | |
| 5 | 26 | |
| 6 | 50 | |
| 7 | 31 | |
| 8 | 1 | |
| 9 | 4 | |
| 10 | 3 | |
| 11 | 0 | |
| 12 | NEO1 AND NEO-EXT STUDIES: PHARMACODYNAMIC/EXPLORATORY BIOMARKER AND SAFETY ASSESSMENTS FOLLOWING REPEAT AVALGLUCOSIDASE ALFA DOSING FOR UP TO 4.5 YEARS IN PATIENTS WITH LATE-ONSET POMPE DISEASE | 1 |
| 13 | 25 | |
| 14 | 43 | |
| 15 | 66 | |
| 16 | 74 | |
| 17 | 2 | |
| 18 | 31 | |
| 19 | 110 | |
| 20 | 4 |
About Eugen Mengel
Eugen Mengel is a scholar working on Physiology, Cell Biology and Pediatrics, Perinatology and Child Health, having authored 21 papers that have together received 482 indexed citations. Recurring topics across this work include Lysosomal Storage Disorders Research (19 papers), Cellular transport and secretion (7 papers) and Trypanosoma species research and implications (4 papers). The work is most often cited by research in Physiology (430 citations), Rheumatology (98 citations) and Hematology (56 citations). Eugen Mengel has collaborated with scholars based in United States, France and Germany. Frequent co-authors include Roberto Giugliani, Simon Jones, Nathalie Guffon, Paul Harmatz, Melissa Wasserstein, Margaret M. McGovern, Bruno Bembi, Barbara K. Burton, Christian J. Hendriksz and Vassili Valayannopoulos. Their work appears in journals such as Brain, The Journal of Pediatrics and Archives of Disease in Childhood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.