Esther Gillert

506 total citations
8 papers, 400 citations indexed

About

Esther Gillert is a scholar working on Molecular Biology, Public Health, Environmental and Occupational Health and Hematology. According to data from OpenAlex, Esther Gillert has authored 8 papers receiving a total of 400 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 4 papers in Public Health, Environmental and Occupational Health and 3 papers in Hematology. Recurrent topics in Esther Gillert's work include Advanced biosensing and bioanalysis techniques (5 papers), Acute Lymphoblastic Leukemia research (4 papers) and Acute Myeloid Leukemia Research (3 papers). Esther Gillert is often cited by papers focused on Advanced biosensing and bioanalysis techniques (5 papers), Acute Lymphoblastic Leukemia research (4 papers) and Acute Myeloid Leukemia Research (3 papers). Esther Gillert collaborates with scholars based in Germany, United States and Slovakia. Esther Gillert's co-authors include Rolf Marschalek, Georg H. Fey, Johann Greil, Martin Reichel, Reinald Repp, Sieglinde Angermüller, Gabriele Margareta Siegler, Thomas Leis, Frank Griesinger and Ivan D. Horak and has published in prestigious journals such as Oncogene, Leukemia and Journal of Cellular Biochemistry.

In The Last Decade

Esther Gillert

8 papers receiving 384 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Esther Gillert Germany 8 250 178 148 51 44 8 400
F. Mitelman United Kingdom 4 156 0.6× 223 1.3× 108 0.7× 24 0.5× 45 1.0× 4 321
Maria Gracia Gonzalez Spain 7 182 0.7× 202 1.1× 70 0.5× 68 1.3× 19 0.4× 8 380
Meike Fischer Germany 9 167 0.7× 137 0.8× 85 0.6× 41 0.8× 22 0.5× 11 307
MJ Thirman United States 8 251 1.0× 205 1.2× 158 1.1× 26 0.5× 39 0.9× 8 383
Deborah Ruggeri Italy 8 172 0.7× 281 1.6× 73 0.5× 41 0.8× 19 0.4× 9 378
Lene Olesen Denmark 11 378 1.5× 353 2.0× 142 1.0× 29 0.6× 21 0.5× 13 535
C Guidal France 8 91 0.4× 163 0.9× 150 1.0× 54 1.1× 19 0.4× 11 309
Michael Hadler United States 7 255 1.0× 233 1.3× 261 1.8× 48 0.9× 24 0.5× 12 531
AJ Carroll United States 7 119 0.5× 298 1.7× 232 1.6× 26 0.5× 29 0.7× 9 377

Countries citing papers authored by Esther Gillert

Since Specialization
Citations

This map shows the geographic impact of Esther Gillert's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Esther Gillert with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Esther Gillert more than expected).

Fields of papers citing papers by Esther Gillert

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Esther Gillert. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Esther Gillert. The network helps show where Esther Gillert may publish in the future.

Co-authorship network of co-authors of Esther Gillert

This figure shows the co-authorship network connecting the top 25 collaborators of Esther Gillert. A scholar is included among the top collaborators of Esther Gillert based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Esther Gillert. Esther Gillert is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Zech, Juergen, Peter L. Oliver, Edith Pfitzner, et al.. (2006). Transcription linked to recombination: a gene-internal promoter coincides with the recombination hot spot II of the human MLL gene. Oncogene. 26(10). 1361–1371. 40 indexed citations
2.
3.
Reichel, Martin, Esther Gillert, Sieglinde Angermüller, et al.. (2001). Rapid isolation of chromosomal breakpoints from patients with t(4;11) acute lymphoblastic leukemia: implications for basic and clinical research. Leukemia. 15(2). 286–288. 10 indexed citations
4.
Reichel, Martin, Esther Gillert, Sieglinde Angermüller, et al.. (2001). Biased distribution of chromosomal breakpoints involving the MLL gene in infants versus children and adults with t(4;11) ALL. Oncogene. 20(23). 2900–2907. 68 indexed citations
5.
Gillert, Esther, Thomas Leis, Reinald Repp, et al.. (1999). A DNA damage repair mechanism is involved in the origin of chromosomal translocations t(4;11) in primary leukemic cells. Oncogene. 18(33). 4663–4671. 95 indexed citations
6.
Reichel, Martin, Esther Gillert, Reinald Repp, et al.. (1999). Rapid isolation of chromosomal breakpoints from patients with t(4;11) acute lymphoblastic leukemia: implications for basic and clinical research.. PubMed. 59(14). 3357–62. 30 indexed citations
7.
Reichel, Martin, Esther Gillert, Gabriele Margareta Siegler, et al.. (1998). Fine structure of translocation breakpoints in leukemic blasts with chromosomal translocation t(4;11): the DNA damage-repair model of translocation. Oncogene. 17(23). 3035–3044. 83 indexed citations
8.
Autenrieth, Ingo B., et al.. (1996). Interleukin-2 Is Indispensable for Development of Immunological Self-Tolerance. Clinical Immunology and Immunopathology. 81(3). 282–286. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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