Esra Gündüz

1.8k citations
70 papers · 1.3k indexed · h-index 21
  • Oncology top 10%
    • Cancer-related Molecular Pathways 8
    • Cancer-related gene regulation 22
    • RNA modifications and cancer 14
    • Genomics and Chromatin Dynamics 7
    • Chromatin Remodeling and Cancer 6
    • Wnt/β-catenin signaling in development and cancer 6
    • Epigenetics and DNA Methylation 5
    • Cell Adhesion Molecules Research 5

Esra Gündüz

69 papers receiving 1.3k citations

Peers

Esra Gündüz
Comparison fields: 5 of 92
  • Cancer Research 240
  • Otorhinolaryngology 69
  • Oncology 397
  • Molecular Biology 858
  • Immunology and Allergy 65
Replace Norishige Yamada with:
Norishige Yamada Japan
Jianming Hu China
Seth B. Krantz United States
Hiroshi Ogawa Japan
Maria Cristina Curia Italy
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M Korabiowska Germany
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Esra Gündüz relative to Norishige Yamada Japan Norishige Yamada's profile →
Citations per field
00.5×8.1×
Norishige Yamada · 1×
Citations per year

Countries citing papers authored by Esra Gündüz

Since Specialization
Citations

This map shows the geographic impact of Esra Gündüz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Esra Gündüz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Esra Gündüz more than expected).

Fields of papers citing papers by Esra Gündüz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Esra Gündüz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Esra Gündüz. The network helps show where Esra Gündüz may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Esra Gündüz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Esra Gündüz Line = papers co-authored together Esra Gündüz links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 202137
2 20168
3 20156
4 20122
5 201048
6 200916
7 200919
8 200912
9 20085
10 200846
11 200886
12 200834
13 200827
14 200817
15 200822
16 200530
17 200534
18 20043
19 200374
20 20028

About Esra Gündüz

Esra Gündüz is a scholar working on Immunology and Allergy, Otorhinolaryngology and Molecular Biology, having authored 70 papers that have together received 1.3k indexed citations. Recurring topics across this work include Cancer-related gene regulation (22 papers), RNA modifications and cancer (14 papers), Cancer-related Molecular Pathways (8 papers), Genomics and Chromatin Dynamics (7 papers), Chromatin Remodeling and Cancer (6 papers), Wnt/β-catenin signaling in development and cancer (6 papers), Epigenetics and DNA Methylation (5 papers) and Cell Adhesion Molecules Research (5 papers). The work is most often cited by research in Cancer Research (240 citations), Otorhinolaryngology (69 citations) and Oncology (397 citations). Esra Gündüz has collaborated with scholars based in Japan, Türkiye and Finland. Frequent co-authors include Hitoshi Nagatsuka, Mehmet Gündüz, Mehmet Gündüz, Levent Beder, Kenji Shimizu, Noriyuki Nagai, Kunihiro Fukushima, Beyhan Cengiz, Ryo Tamamura and Noboru Yamanaka. Their work appears in journals such as Biochemical and Biophysical Research Communications, International Journal of Cancer and Gene.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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