Emre E. Turer

3.1k total citations · 1 hit paper
19 papers, 2.3k citations indexed

About

Emre E. Turer is a scholar working on Molecular Biology, Immunology and Cancer Research. According to data from OpenAlex, Emre E. Turer has authored 19 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 9 papers in Immunology and 8 papers in Cancer Research. Recurrent topics in Emre E. Turer's work include Immune Response and Inflammation (8 papers), NF-κB Signaling Pathways (8 papers) and interferon and immune responses (4 papers). Emre E. Turer is often cited by papers focused on Immune Response and Inflammation (8 papers), NF-κB Signaling Pathways (8 papers) and interferon and immune responses (4 papers). Emre E. Turer collaborates with scholars based in United States, Portugal and Sweden. Emre E. Turer's co-authors include Averil Ma, Osamu Hitotsumatsu, Barbara A. Malynn, Rommel Advincula, Rizwan Ahmad, Sophia Chai, David L. Boone, Cecile M. Pickart, Matthew T. Wheeler and Paula J. Hurley and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Emre E. Turer

19 papers receiving 2.2k citations

Hit Papers

The ubiquitin-modifying enzyme A20 is required for termin... 2004 2026 2011 2018 2004 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Emre E. Turer United States 12 1.6k 997 995 248 210 19 2.3k
Rommel Advincula United States 17 1.8k 1.1× 1.0k 1.0× 817 0.8× 457 1.8× 265 1.3× 21 2.6k
James P. Lodolce United States 14 2.4k 1.5× 784 0.8× 727 0.7× 545 2.2× 391 1.9× 20 3.1k
Andreas Gewies Germany 18 1.1k 0.7× 785 0.8× 513 0.5× 352 1.4× 354 1.7× 31 2.3k
Marcia Chien United States 11 2.4k 1.5× 1.1k 1.1× 1.1k 1.1× 520 2.1× 341 1.6× 16 3.3k
Arnulf Hertweck United Kingdom 14 1.4k 0.9× 983 1.0× 724 0.7× 298 1.2× 83 0.4× 22 2.4k
Beichu Guo United States 22 2.4k 1.5× 1.2k 1.2× 685 0.7× 639 2.6× 381 1.8× 28 3.4k
Brian Skaug United States 14 1.4k 0.9× 1.4k 1.4× 525 0.5× 292 1.2× 326 1.6× 22 2.4k
Jeannie Q. He United States 14 2.0k 1.3× 1.2k 1.2× 1.1k 1.1× 507 2.0× 276 1.3× 14 2.8k
Reiko Shinkura Japan 29 1.9k 1.2× 1.3k 1.3× 438 0.4× 416 1.7× 344 1.6× 48 3.1k
Osamu Hitotsumatsu United States 5 1.1k 0.7× 580 0.6× 640 0.6× 164 0.7× 157 0.7× 5 1.5k

Countries citing papers authored by Emre E. Turer

Since Specialization
Citations

This map shows the geographic impact of Emre E. Turer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emre E. Turer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emre E. Turer more than expected).

Fields of papers citing papers by Emre E. Turer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Emre E. Turer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emre E. Turer. The network helps show where Emre E. Turer may publish in the future.

Co-authorship network of co-authors of Emre E. Turer

This figure shows the co-authorship network connecting the top 25 collaborators of Emre E. Turer. A scholar is included among the top collaborators of Emre E. Turer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emre E. Turer. Emre E. Turer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Singla, Amika, Ho Yee Joyce Fung, Ran Song, et al.. (2024). Structural basis for Retriever-SNX17 assembly and endosomal sorting. Nature Communications. 15(1). 10193–10193. 5 indexed citations
2.
Song, Ran, Xiaohong Li, Miao Tang, et al.. (2023). The dual lipid desaturase/hydroxylase DEGS2 controls phytoceramide levels necessary to counter intestinal inflammation. Disease Models & Mechanisms. 16(9). 2 indexed citations
3.
Song, Ran, William McAlpine, Evan Nair‐Gill, et al.. (2023). Trans-Golgi protein TVP23B regulates host-microbe interactions via Paneth cell homeostasis and Goblet cell glycosylation. Nature Communications. 14(1). 3652–3652. 8 indexed citations
4.
Yue, Tao, Xiaoming Zhan, Duanwu Zhang, et al.. (2021). SLFN2 protection of tRNAs from stress-induced cleavage is essential for T cell–mediated immunity. Science. 372(6543). 59 indexed citations
5.
Pais, Lynn, Casie A. Genetti, John M. Schreiber, et al.. (2021). Genetic Study in a Cohort of Children With ROHHAD Syndrome. Journal of the Endocrine Society. 5(Supplement_1). A503–A504. 3 indexed citations
6.
Wang, Kuan-Wen, Xiaoming Zhan, William McAlpine, et al.. (2019). Enhanced susceptibility to chemically induced colitis caused by excessive endosomal TLR signaling in LRBA-deficient mice. Proceedings of the National Academy of Sciences. 116(23). 11380–11389. 17 indexed citations
7.
McAlpine, William, Jamie L. Russell, Anne R. Murray, Bruce Beutler, & Emre E. Turer. (2019). Research Techniques Made Simple: Forward Genetic Screening to Uncover Genes Involved in Skin Biology. Journal of Investigative Dermatology. 139(9). 1848–1853.e1. 3 indexed citations
8.
McAlpine, William, Jin Huk Choi, Sara Ludwig, et al.. (2018). The class I myosin MYO1D binds to lipid and protects against colitis. Disease Models & Mechanisms. 11(9). 9 indexed citations
9.
Turer, Emre E., William McAlpine, Feiya Ou, et al.. (2018). A viable hypomorphic Arnt2 mutation causes hyperphagic obesity, diabetes and hepatic steatosis. Disease Models & Mechanisms. 11(12). 5 indexed citations
10.
Miyata, Naoteru, Qing Chen, Curtis A. Thorne, et al.. (2018). Microbial Sensing by Intestinal Myeloid Cells Controls Carcinogenesis and Epithelial Differentiation. Cell Reports. 24(9). 2342–2355. 15 indexed citations
11.
Turer, Emre E., William McAlpine, Kuan-Wen Wang, et al.. (2017). Creatine maintains intestinal homeostasis and protects against colitis. Proceedings of the National Academy of Sciences. 114(7). E1273–E1281. 56 indexed citations
12.
Zhang, Zhao, Emre E. Turer, Xiaohong Li, et al.. (2016). Insulin resistance and diabetes caused by genetic or diet-induced KBTBD2 deficiency in mice. Proceedings of the National Academy of Sciences. 113(42). E6418–E6426. 33 indexed citations
13.
Lu, Timothy T., Michio Onizawa, Gianna Hammer, et al.. (2013). Dimerization and Ubiquitin Mediated Recruitment of A20, a Complex Deubiquitinating Enzyme. Immunity. 38(5). 896–905. 117 indexed citations
14.
Hammer, Gianna, Emre E. Turer, Kimberly E. Taylor, et al.. (2011). Expression of A20 by dendritic cells preserves immune homeostasis and prevents colitis and spondyloarthritis. Nature Immunology. 12(12). 1184–1193. 212 indexed citations
15.
Tavares, Rita M., Emre E. Turer, Rommel Advincula, et al.. (2010). The Ubiquitin Modifying Enzyme A20 Restricts B Cell Survival and Prevents Autoimmunity. Immunity. 33(2). 181–191. 201 indexed citations
16.
Turer, Emre E., Rita M. Tavares, Erwan Mortier, et al.. (2008). Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20. The Journal of Experimental Medicine. 205(2). 451–464. 224 indexed citations
17.
Hitotsumatsu, Osamu, Rizwan Ahmad, Rita M. Tavares, et al.. (2008). The Ubiquitin-Editing Enzyme A20 Restricts Nucleotide-Binding Oligomerization Domain Containing 2-Triggered Signals. Immunity. 28(3). 381–390. 280 indexed citations
18.
Oshima, Shigeru, Emre E. Turer, Sophia Chai, et al.. (2008). ABIN-1 is a ubiquitin sensor that restricts cell death and sustains embryonic development. Nature. 457(7231). 906–909. 135 indexed citations
19.
Boone, David L., Emre E. Turer, Rizwan Ahmad, et al.. (2004). The ubiquitin-modifying enzyme A20 is required for termination of Toll-like receptor responses. Nature Immunology. 5(10). 1052–1060. 889 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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