Emma L. Wise

711 total citations
11 papers, 573 citations indexed

About

Emma L. Wise is a scholar working on Immunology, Oncology and Molecular Biology. According to data from OpenAlex, Emma L. Wise has authored 11 papers receiving a total of 573 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Immunology, 8 papers in Oncology and 3 papers in Molecular Biology. Recurrent topics in Emma L. Wise's work include T-cell and B-cell Immunology (7 papers), Chemokine receptors and signaling (7 papers) and Immune Cell Function and Interaction (5 papers). Emma L. Wise is often cited by papers focused on T-cell and B-cell Immunology (7 papers), Chemokine receptors and signaling (7 papers) and Immune Cell Function and Interaction (5 papers). Emma L. Wise collaborates with scholars based in United Kingdom, United States and Austria. Emma L. Wise's co-authors include James E. Pease, Robert E. Tigelaar, Adrian Hayday, Daniel J. Pennington, Timothy J. Williams, Sarah J. Petit, Anja Müeller, Bruno Silva‐Santos, Daniel M. Davis and Ellen M. McDonagh and has published in prestigious journals such as Journal of Biological Chemistry, Nature Immunology and The Journal of Immunology.

In The Last Decade

Emma L. Wise

11 papers receiving 566 citations

Peers

Countries citing papers authored by Emma L. Wise

Since Specialization

Citations

This map shows the geographic impact of Emma L. Wise's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emma L. Wise with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emma L. Wise more than expected).

Fields of papers citing papers by Emma L. Wise

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Emma L. Wise. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emma L. Wise. The network helps show where Emma L. Wise may publish in the future.

Co-authorship network of co-authors of Emma L. Wise

This figure shows the co-authorship network connecting the top 25 collaborators of Emma L. Wise. A scholar is included among the top collaborators of Emma L. Wise based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emma L. Wise. Emma L. Wise is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

#	Work	Indexed citations
1	Small molecule chemokine mimetics suggest a molecular basis for the observation that CXCL10 and CXCL11 are allosteric ligands of CXCR3 2011 ·British Journal of Pharmacology ·Belinda Nedjai, Hubert Li, Ilana L. Stroke, Emma L. Wise, Maria L. Webb, J. Robert Merritt, Ian Henderson, Anthony E. Klon, Andrew G. Cole, Richard Horuk, Nagarajan Vaidehi, James E. Pease	35
2	A distinct subset of human NK cells expressing HLA‐DR expand in response to IL‐2 and can aid immune responses to BCG 2011 ·European Journal of Immunology ·(unknown), Amir Horowitz, Maryam Mehrabi, Emma L. Wise, James E. Pease, Eleanor M. Riley, Daniel M. Davis	81
3	The CXCL16 A181V Mutation Selectively Inhibits Monocyte Adhesion to CXCR6 but Is Not Associated With Human Coronary Heart Disease 2011 ·Arteriosclerosis Thrombosis and Vascular Biology ·Sarah J. Petit, Emma L. Wise, John C. Chambers, (unknown), Naomi E. Chayen, Jaspal S. Kooner, James E. Pease	16
4	A single nucleotide polymorphism in the CCR3 gene ablates receptor export to the plasma membrane 2010 ·Journal of Allergy and Clinical Immunology ·Emma L. Wise, (unknown), Timothy J. Williams, James E. Pease	8
5	Elucidation of Binding Sites of Dual Antagonists in the Human Chemokine Receptors CCR2 and CCR5 2009 ·Molecular Pharmacology ·Spencer E. Hall, Allen Mao, Vicky Nicolaidou, Mattéa J. Finelli, Emma L. Wise, Belinda Nedjai, (unknown), Paymann Harirchian, (unknown), (unknown), (unknown), (unknown), James E. Pease, Richard Horuk, Nagarajan Vaidehi	47
6	The Chemokine Receptor CXCR3 Is Degraded following Internalization and Is Replenished at the Cell Surface by De Novo Synthesis of Receptor 2008 ·The Journal of Immunology ·Andrea Meiser, Anja Müeller, Emma L. Wise, Ellen M. McDonagh, Sarah J. Petit, (unknown), Peter Clark, Timothy J. Williams, James E. Pease	106
7	Structure and Function of A41, a Vaccinia Virus Chemokine Binding Protein 2008 ·PLoS Pathogens ·Mohammad W. Bahar, Julia C. Kenyon, Mike M. Pütz, Nicola G. A. Abrescia, James E. Pease, Emma L. Wise, David I. Stuart, Geoffrey L. Smith, Jonathan M. Grimes	62
8	Small Molecule Receptor Agonists and Antagonists of CCR3 Provide Insight into Mechanisms of Chemokine Receptor Activation 2007 ·Journal of Biological Chemistry ·Emma L. Wise, (unknown), da Fonseca, Rodger A. Allen, Timothy J. Williams, James E. Pease	46
9	Unravelling the mechanisms underpinning chemokine receptor activation and blockade by small molecules: a fine line between agonism and antagonism? 2007 ·Biochemical Society Transactions ·Emma L. Wise, James E. Pease	3
10	The Integration of Conventional and Unconventional T Cells that Characterizes Cell‐Mediated Responses 2005 ·Advances in immunology ·Daniel J. Pennington, David Vermijlen, Emma L. Wise, Sarah L. Clarke, Robert E. Tigelaar, Adrian Hayday	62
11	The inter-relatedness and interdependence of mouse T cell receptor γδ+ and αβ+ cells 2003 ·Nature Immunology ·Daniel J. Pennington, Bruno Silva‐Santos, John Shires, Efstathios Theodoridis, Christopher C. Pollitt, Emma L. Wise, Robert E. Tigelaar, Michael J. Owen, Adrian Hayday	107

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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