Eloise Peet

425 total citations
7 papers, 345 citations indexed

About

Eloise Peet is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Psychiatry and Mental health. According to data from OpenAlex, Eloise Peet has authored 7 papers receiving a total of 345 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Cellular and Molecular Neuroscience, 3 papers in Molecular Biology and 2 papers in Psychiatry and Mental health. Recurrent topics in Eloise Peet's work include Neurotransmitter Receptor Influence on Behavior (3 papers), Neuroscience and Neuropharmacology Research (2 papers) and Receptor Mechanisms and Signaling (2 papers). Eloise Peet is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (3 papers), Neuroscience and Neuropharmacology Research (2 papers) and Receptor Mechanisms and Signaling (2 papers). Eloise Peet collaborates with scholars based in United States and China. Eloise Peet's co-authors include Stephen B. Manuck, Douglas E. Williamson, Ronald E. Dahl, R E Ferrell, Ahmad R. Hariri, Sarah M. Brown, Michael W. Shiflett, Edda Thiels, Jocelyn C. Mauna and Ruth G. Perez and has published in prestigious journals such as Journal of Neuroscience, PLoS ONE and Journal of Neurochemistry.

In The Last Decade

Eloise Peet

7 papers receiving 338 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Eloise Peet United States	6	168	92	77	62	56	7	345
Yolanda Peña‐Oliver United Kingdom	11	309 1.8×	174 1.9×	93 1.2×	39 0.6×	54 1.0×	14	448
Aaron Tan United States	4	120 0.7×	142 1.5×	52 0.7×	44 0.7×	25 0.4×	4	324
Michael M. Barrus Canada	8	175 1.0×	78 0.8×	40 0.5×	24 0.4×	95 1.7×	10	299
Anke Zimmer Germany	7	242 1.4×	179 1.9×	53 0.7×	95 1.5×	119 2.1×	8	501
Shilpa K Gandhi United States	6	202 1.2×	157 1.7×	93 1.2×	61 1.0×	65 1.2×	7	512
Mahesh Darna United States	12	280 1.7×	141 1.5×	118 1.5×	35 0.6×	39 0.7×	16	452
Hershel Mehta United States	2	172 1.0×	268 2.9×	51 0.7×	58 0.9×	22 0.4×	2	416
Laura J. Mosher United States	13	131 0.8×	64 0.7×	58 0.8×	23 0.4×	128 2.3×	18	316
Chelsea A. Vadnie United States	9	87 0.5×	73 0.8×	56 0.7×	83 1.3×	29 0.5×	15	319
Samantha J. Podurgiel United States	11	282 1.7×	104 1.1×	91 1.2×	18 0.3×	23 0.4×	14	447

Countries citing papers authored by Eloise Peet

Since Specialization

Citations

This map shows the geographic impact of Eloise Peet's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eloise Peet with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eloise Peet more than expected).

Fields of papers citing papers by Eloise Peet

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eloise Peet. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eloise Peet. The network helps show where Eloise Peet may publish in the future.

Co-authorship network of co-authors of Eloise Peet

This figure shows the co-authorship network connecting the top 25 collaborators of Eloise Peet. A scholar is included among the top collaborators of Eloise Peet based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eloise Peet. Eloise Peet is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Karunanidhi, Anuradha, Kimimasa Tobita, Charles L. Hoppel, et al.. (2020). ACAD10 protein expression and Neurobehavioral assessment of Acad10-deficient mice. PLoS ONE. 15(12). e0242445–e0242445. 1 indexed citations

2.

Villanueva, Ernesto, et al.. (2013). Non‐motor parkinsonian pathology in aging A53T α‐Synuclein mice is associated with progressive synucleinopathy and altered enzymatic function. Journal of Neurochemistry. 128(4). 536–546. 52 indexed citations

3.

Neumann, Serina A., Whittemore G. Tingley, Bruce R. Conklin, et al.. (2009). AKAP10 (I646V) functional polymorphism predicts heart rate and heart rate variability in apparently healthy, middle‐aged European‐Americans. Psychophysiology. 46(3). 466–472. 19 indexed citations

4.

Shiflett, Michael W., Jocelyn C. Mauna, Amanda M. Chipman, Eloise Peet, & Edda Thiels. (2009). Appetitive Pavlovian conditioned stimuli increase CREB phosphorylation in the nucleus accumbens. Neurobiology of Learning and Memory. 92(3). 451–454. 17 indexed citations

5.

Shiflett, Michael W., et al.. (2008). Cue-Elicited Reward-Seeking Requires Extracellular Signal-Regulated Kinase Activation in the Nucleus Accumbens. Journal of Neuroscience. 28(6). 1434–1443. 32 indexed citations

6.

Brown, Sarah M., Eloise Peet, Stephen B. Manuck, et al.. (2005). A regulatory variant of the human tryptophan hydroxylase-2 gene biases amygdala reactivity. Molecular Psychiatry. 10(9). 884–888. 197 indexed citations

7.

Brown, Sarah M., Eloise Peet, Stephen B. Manuck, et al.. (2005). A regulatory variant of the human tryptophan hydroxylase-2 gene biases amygdala reactivity. Molecular Psychiatry. 10(9). 805–805. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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