Élodie Bal

2.3k total citations
15 papers, 517 citations indexed

About

Élodie Bal is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Genetics. According to data from OpenAlex, Élodie Bal has authored 15 papers receiving a total of 517 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 5 papers in Pathology and Forensic Medicine and 4 papers in Genetics. Recurrent topics in Élodie Bal's work include Genetic and rare skin diseases. (4 papers), Lymphoma Diagnosis and Treatment (4 papers) and Cancer-related gene regulation (3 papers). Élodie Bal is often cited by papers focused on Genetic and rare skin diseases. (4 papers), Lymphoma Diagnosis and Treatment (4 papers) and Cancer-related gene regulation (3 papers). Élodie Bal collaborates with scholars based in France, United States and Italy. Élodie Bal's co-authors include Asma Smahi, Arnold Münnich, Christine Bodemer, Céline Cluzeau, Jean‐Paul Bonnefont, Katia Basso, Riccardo Dalla‐Favera, Sofija Vlasevska, Antony B. Holmes and Laura Pasqualucci and has published in prestigious journals such as Nature, Immunity and Journal of Allergy and Clinical Immunology.

In The Last Decade

Élodie Bal

13 papers receiving 511 citations

Peers

Countries citing papers authored by Élodie Bal

Since Specialization

Citations

This map shows the geographic impact of Élodie Bal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Élodie Bal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Élodie Bal more than expected).

Fields of papers citing papers by Élodie Bal

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Élodie Bal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Élodie Bal. The network helps show where Élodie Bal may publish in the future.

Co-authorship network of co-authors of Élodie Bal

This figure shows the co-authorship network connecting the top 25 collaborators of Élodie Bal. A scholar is included among the top collaborators of Élodie Bal based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Élodie Bal. Élodie Bal is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

#	Work	Indexed citations
1	SUPER‐ENHANCER HYPERMUTATION IN DLBCL: ROLE OF ALTERATIONS IN THE GLUCOCORTICOID RECEPTOR PATHWAY 2023 ·Hematological Oncology ·Élodie Bal, Clarissa Corinaldesi, Antony B. Holmes, Laura Pasqualucci, Katia Basso, Riccardo Dalla‐Favera	0
2	BTG2 SUPER‐ENHANCER MUTATIONS DISRUPT TFAP4 BINDING AND DYSREGULATE BTG2 EXPRESSION IN DIFFUSE LARGE B‐CELL LYMPHOMA 2023 ·Hematological Oncology ·Élodie Bal, Paola De Simone, Andrew B. Holmes, Lisette Hilton, Katia Basso, Rajesh K. Soni, Ryan D. Morin, Riccardo Dalla‐Favera, Laura Pasqualucci	0
3	Super-enhancer hypermutation alters oncogene expression in B cell lymphoma 2022 ·Nature ·Élodie Bal, Rahul Kumar, Mohammad Hadigol, Antony B. Holmes, Laura K. Hilton, (unknown), Kostiantyn Dreval, Jasper Wong, Sofija Vlasevska, Clarissa Corinaldesi, Rajesh K. Soni, Katia Basso, Ryan D. Morin, Hossein Khiabanian, Laura Pasqualucci, Riccardo Dalla‐Favera	74
4	PERVASIVE HYPERMUTATION OF SUPER‐ENHANCER REGIONS DYSREGULATES ONCOGENE EXPRESSION IN DIFFUSE LARGE B‐CELL LYMPHOMA 2021 ·Hematological Oncology ·Élodie Bal, Rahul Kumar, Mohammad Hadigol, Antony B. Holmes, Katia Basso, Hossein Khiabanian, Laura Pasqualucci, Riccardo Dalla‐Favera	1
5	Unique and Shared Epigenetic Programs of the CREBBP and EP300 Acetyltransferases in Germinal Center B Cells Reveal Targetable Dependencies in Lymphoma 2019 ·Immunity ·Stefanie N. Meyer, Claudio Scuoppo, Sofija Vlasevska, Élodie Bal, Antony B. Holmes, (unknown), Laura Garcia‐Ibanez, Sarah Nataraj, Romain Duval, (unknown), Katia Basso, Nigel Brooks, Riccardo Dalla‐Favera, Laura Pasqualucci	88
6	Turkish Ectodermal Dysplasia Cohort: From Phenotype to Genotype in 17 Families 2019 ·Cytogenetic and Genome Research ·Yeliz Güven, Élodie Bal, Umut Altunoğlu, (unknown), S. Hadj‐Rabia, (unknown), Elif Bahar Tuna, (unknown), Oya Aktören, Christine Bodemer, Zehra Oya Uyguner, Asma Smahi, Hülya Kayserili	8
7	Lack of interaction between NEMO and SHARPIN impairs linear ubiquitination and NF-κB activation and leads to incontinentia pigmenti 2017 ·Journal of Allergy and Clinical Immunology ·Élodie Bal, Emmanuel Laplantine, Yamina Hamel, Virginie Dubosclard, Bertrand Boisson, Alessandra Pescatore, Capucine Pïcard, S. Hadj‐Rabia, (unknown), Julie Steffann, Jean‐Paul Bonnefont, (unknown), P. Vabres, Arnold Münnich, Jean‐Laurent Casanova, Christine Bodemer, Robert Weil, Fabrice Agou, Asma Smahi	13
8	Severe neuroimaging anomalies are usually associated with random X inactivation in leucocytes circulating DNA in X-linked dominant Incontinentia Pigmenti 2017 ·Molecular Genetics and Metabolism ·Volodia Dangouloff‐Ros, S. Hadj‐Rabia, (unknown), Élodie Bal, I. Desguerre, Manoëlle Kossorotoff, Isabelle An, Asma Smahi, Christine Bodemer, Arnold Münnich, Julie Steffann, Nathalie Boddaert	14
9	IL36RN Mutations Affect Protein Expression and Function: A Basis for Genotype-Phenotype Correlation in Pustular Diseases 2016 ·Journal of Investigative Dermatology ·M. Tauber, Élodie Bal, Xue-Yuan Pei, Marine Madrange, Amel Haj Khelil, (unknown), (unknown), Mohamed Makrelouf, (unknown), (unknown), (unknown), (unknown), (unknown), Slaheddine Marrakchi, H. Turki, E. Bourrat, M. Viguier, Yamina Hamel, H. Bachelez, Asma Smahi	71
10	Genomic architecture at the Incontinentia Pigmenti locus favours de novo pathological alleles through different mechanisms 2011 ·Human Molecular Genetics ·Francesca Fusco, Mariateresa Paciolla, (unknown), Alessandra Pescatore, (unknown), Élodie Bal, Maria Brigida Lioi, Asma Smahi, Maria Giuseppina Miano, Matilde Valeria Ursini	46
11	Mutations inEDARADDaccount for a small proportion of hypohidrotic ectodermal dysplasia cases 2010 ·British Journal of Dermatology ·Nicolas Chassaing, Céline Cluzeau, Élodie Bal, Philippe Guigue, (unknown), Géraldine Viot, (unknown), (unknown), Asma Smahi, Patrick Calvas	20
12	Alterations of the IKBKG locus and diseases: an update and a report of 13 novel mutations 2008 ·Human Mutation ·Francesca Fusco, Alessandra Pescatore, Élodie Bal, Aïda Ghoul, Mariateresa Paciolla, Maria Brigida Lioi, Michele D’Urso, (unknown), Christine Bodemer, Jean‐Paul Bonnefont, Arnold Münnich, Maria Giuseppina Miano, Asma Smahi, Matilde Valeria Ursini	76
13	Autosomal dominant anhidrotic ectodermal dysplasias at the EDARADD locus 2007 ·Human Mutation ·Élodie Bal, Lekbir Baala, Céline Cluzeau, F. El Kerch, Karim Ouldim, S. Hadj‐Rabia, Christine Bodemer, Arnold Münnich, Gilles Courtois, Abdelaziz Sefiani, Asma Smahi	61
14	Father-to-daughter transmission of Cornelia de Lange syndrome caused by a mutation in the 5′ untranslated region of theNIPBL Gene 2006 ·Human Mutation ·Guntram Borck, (unknown), Céline Cluzeau, Élodie Bal, Jean‐Paul Bonnefont, Arnold Munnich, Valérie Cormier‐Daire, Laurence Colleaux	44
15	Efforts to chemically induce mutations for cytoplasmic male sterility in petunia. I. Field experiments 1985 ·Euphytica ·Ami Harten, (unknown), Élodie Bal	1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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