Elisa Boscolo

3.1k total citations
47 papers, 2.2k citations indexed

About

Elisa Boscolo is a scholar working on Surgery, Neurology and Oncology. According to data from OpenAlex, Elisa Boscolo has authored 47 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Surgery, 24 papers in Neurology and 17 papers in Oncology. Recurrent topics in Elisa Boscolo's work include Vascular Malformations and Hemangiomas (30 papers), Vascular Malformations Diagnosis and Treatment (24 papers) and Vascular Tumors and Angiosarcomas (14 papers). Elisa Boscolo is often cited by papers focused on Vascular Malformations and Hemangiomas (30 papers), Vascular Malformations Diagnosis and Treatment (24 papers) and Vascular Tumors and Angiosarcomas (14 papers). Elisa Boscolo collaborates with scholars based in United States, Italy and France. Elisa Boscolo's co-authors include Joyce Bischoff, John B. Mulliken, Shoshana Greenberger, Irit Adini, Zia A. Khan, Juan M. Melero‐Martin, Miikka Vikkula, Nisha Limaye, Arnaud Picard and Sarah P. Psutka and has published in prestigious journals such as New England Journal of Medicine, Journal of Clinical Investigation and Nature Medicine.

In The Last Decade

Elisa Boscolo

43 papers receiving 2.2k citations

Peers

Elisa Boscolo

SURGE

NEURO

ONCOL

PFM

Pascal Brouillard Belgium

Andrew Horvai United States

Göran Stenman Sweden

Kazuaki Misugi Japan

Marialaura Del Basso De Italy

Gian Luigi Taddei Italy

M. Raphael Pfeffer Israel

Kanji Mori Japan

Eileen Boye United States

Andréï Tchirkov France

Pascal Brouillard Belgium

Elisa Boscolo

870 ×0.6

SURGE

438 ×0.6

NEURO

776 ×1.0

ONCOL

298 ×0.5

418 ×1.5

PFM

Citations per year, relative to Elisa Boscolo Elisa Boscolo (= 1×) peers Pascal Brouillard

Countries citing papers authored by Elisa Boscolo

Since Specialization

Citations

This map shows the geographic impact of Elisa Boscolo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elisa Boscolo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elisa Boscolo more than expected).

Fields of papers citing papers by Elisa Boscolo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Elisa Boscolo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elisa Boscolo. The network helps show where Elisa Boscolo may publish in the future.

Co-authorship network of co-authors of Elisa Boscolo

This figure shows the co-authorship network connecting the top 25 collaborators of Elisa Boscolo. A scholar is included among the top collaborators of Elisa Boscolo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elisa Boscolo. Elisa Boscolo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Boscolo, Elisa, et al.. (2025). Semaphorin 3A and 3F Promote Lumen Expansion in TIE2-Mutated Venous Malformation. Arteriosclerosis Thrombosis and Vascular Biology. 45(12). 2243–2260.

Liu, Bingxin, et al.. (2025). PBAE-PEG/Lipid Nanoparticle Delivery of RNA for the Creation of Genetically Engineered Lung Cancer Mouse Models. Nano Letters. 25(51). 17578–17583.

Cai, Yuqi, Shruti Vemaraju, Yoshinobu Odaka, et al.. (2023). MEK inhibition reduced vascular tumor growth and coagulopathy in a mouse model with hyperactive GNAQ. Nature Communications. 14(1). 1929–1929. 14 indexed citations

Boscolo, Elisa, et al.. (2020). A Xenograft Model for Venous Malformation. Methods in molecular biology. 2206. 179–192. 5 indexed citations

Cai, Yuqi, et al.. (2019). Constitutive Active Mutant TIE2 Induces Enlarged Vascular Lumen Formation with Loss of Apico-basal Polarity and Pericyte Recruitment. Scientific Reports. 9(1). 12352–12352. 19 indexed citations

Bai, Shoumei, Patrick Ingram, Yu‐Chih Chen, et al.. (2016). EGFL6 Regulates the Asymmetric Division, Maintenance, and Metastasis of ALDH+ Ovarian Cancer Cells. Cancer Research. 76(21). 6396–6409. 51 indexed citations

Rossi, Elisa, David M. Smadja, Elisa Boscolo, et al.. (2015). Endoglin regulates mural cell adhesion in the circulatory system. Cellular and Molecular Life Sciences. 73(8). 1715–1739. 59 indexed citations

Boscolo, Elisa, Nisha Limaye, Lan Huang, et al.. (2015). Rapamycin improves TIE2-mutated venous malformation in murine model and human subjects. Journal of Clinical Investigation. 125(9). 3491–3504. 159 indexed citations

Boscolo, Elisa, Sílvia Coma, Valerie L. Luks, et al.. (2014). AKT hyper-phosphorylation associated with PI3K mutations in lymphatic endothelial cells from a patient with lymphatic malformation. Angiogenesis. 18(2). 151–162. 94 indexed citations

10.

Dorfmüller, Peter, Coralie L. Guérin, Ivan Bièche, et al.. (2014). Cooperation between human fibrocytes and endothelial colony-forming cells increases angiogenesis via the CXCR4 pathway. Thrombosis and Haemostasis. 112(11). 1002–1013. 31 indexed citations

11.

Wong, Alvin, Karen A. Tong, Elisa Boscolo, et al.. (2011). Expression of HES and HEY genes in infantile hemangiomas. PubMed. 3(1). 19–19. 22 indexed citations

12.

Wu, June K., et al.. (2010). A switch in Notch gene expression parallels stem cell to endothelial transition in infantile hemangioma. Angiogenesis. 13(1). 15–23. 44 indexed citations

13.

Greenberger, Shoshana, Elisa Boscolo, Irit Adini, John B. Mulliken, & Joyce Bischoff. (2010). Corticosteroid Suppression of VEGF-A in Infantile Hemangioma-Derived Stem Cells. New England Journal of Medicine. 362(11). 1005–1013. 192 indexed citations

14.

Greenberger, Shoshana, Irit Adini, Elisa Boscolo, John B. Mulliken, & Joyce Bischoff. (2010). Targeting NF-κB in infantile hemangioma-derived stem cells reduces VEGF-A expression. Angiogenesis. 13(4). 327–335. 52 indexed citations

15.

Boscolo, Elisa & Joyce Bischoff. (2009). Vasculogenesis in infantile hemangioma. Angiogenesis. 12(2). 197–207. 135 indexed citations

16.

Picard, Arnaud, et al.. (2008). IGF-2 and FLT-1/VEGF-R1 mRNA Levels Reveal Distinctions and Similarities Between Congenital and Common Infantile Hemangioma. Pediatric Research. 63(3). 263–267. 38 indexed citations

17.

Khan, Zia A., Elisa Boscolo, Arnaud Picard, et al.. (2008). Multipotential stem cells recapitulate human infantile hemangioma in immunodeficient mice. Journal of Clinical Investigation. 118(7). 2592–9. 231 indexed citations

18.

Jinnin, Masatoshi, Damian Medici, Nisha Limaye, et al.. (2008). Suppressed NFAT-dependent VEGFR1 expression and constitutive VEGFR2 signaling in infantile hemangioma. Nature Medicine. 14(11). 1236–1246. 257 indexed citations

19.

Yu, Ying, Jill Wylie‐Sears, Elisa Boscolo, John B. Mulliken, & Joyce Bischoff. (2004). Genomic Imprinting of IGF2 Is Maintained in Infantile Hemangioma despite its High Level of Expression. Molecular Medicine. 10(7-12). 117–123. 24 indexed citations

20.

Conconi, Maria Teresa, Silvano Lora, Silvia Baiguera, et al.. (2004). In vitro culture of rat neuromicrovascular endothelial cells on polymeric scaffolds. Journal of Biomedical Materials Research Part A. 71A(4). 669–674. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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