Élida G. Campos
- Ecology top 5%
- Health, Toxicology and Mutagenesis top 10%
- Molecular Biology
- Epidemiology
- Aquatic Science top 5%
- Co-authors
- Marcelo Hermes‐LimaDaniel C. MoreiraAlexis F. WelkerMaximiliano Giraud‐BilloudGeorgina A. Rivera‐IngrahamThiago C. Genaro‐MattosFranklin David RumjanekLuís Afonso
- Topics
- Physiological and biochemical adaptations (8 papers)Parasites and Host Interactions (5 papers)Environmental Toxicology and Ecotoxicology (4 papers)
In The Last Decade
Élida G. Campos
26 papers receiving 728 citations
Peers
Comparison fields: 5 of 102
- Ecology 305
- Health, Toxicology and Mutagenesis 132
- Molecular Biology 131
- Epidemiology 113
- Aquatic Science 95
Countries citing papers authored by Élida G. Campos
This map shows the geographic impact of Élida G. Campos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Élida G. Campos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Élida G. Campos more than expected).
Fields of papers citing papers by Élida G. Campos
This network shows the impact of papers produced by Élida G. Campos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Élida G. Campos. The network helps show where Élida G. Campos may publish in the future.
Co-authorship network of co-authors of Élida G. Campos
This figure shows the co-authorship network connecting the top 25 collaborators of Élida G. Campos. A scholar is included among the top collaborators of Élida G. Campos based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Élida G. Campos. Élida G. Campos is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 10 | |
| 2 | 9 | |
| 3 | 8 | |
| 4 | 35 | |
| 5 | 181 | |
| 6 | 155 | |
| 7 | 19 | |
| 8 | 22 | |
| 9 | 14 | |
| 10 | 32 | |
| 11 | 3 | |
| 12 | 57 | |
| 13 | 8 | |
| 14 | 22 | |
| 15 | 7 | |
| 16 | Immunocytochemical study of giant cell fibroma. | 19 |
| 17 | 14 | |
| 18 | 10 | |
| 19 | 4 | |
| 20 | 44 |
About Élida G. Campos
Élida G. Campos is a scholar working on Parasitology, Applied Microbiology and Biotechnology and Ecology, having authored 26 papers that have together received 738 indexed citations. Recurring topics across this work include Physiological and biochemical adaptations (8 papers), Parasites and Host Interactions (5 papers) and Environmental Toxicology and Ecotoxicology (4 papers). The work is most often cited by research in Aquatic Science (95 citations), Ecology (305 citations) and Health, Toxicology and Mutagenesis (132 citations). Élida G. Campos has collaborated with scholars based in Brazil, Canada and Argentina. Frequent co-authors include Marcelo Hermes‐Lima, Daniel C. Moreira, Alexis F. Welker, Maximiliano Giraud‐Billoud, Georgina A. Rivera‐Ingraham, Thiago C. Genaro‐Mattos, Franklin David Rumjanek, Luís Afonso, Maria Sueli Soares Felipe and Rosângela Vieira de Andrade. Their work appears in journals such as Scientific Reports, Free Radical Biology and Medicine and Journal of Ethnopharmacology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.