Elena Vendramini

935 total citations
10 papers, 232 citations indexed

About

Elena Vendramini is a scholar working on Molecular Biology, Genetics and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Elena Vendramini has authored 10 papers receiving a total of 232 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Genetics and 5 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Elena Vendramini's work include Chronic Lymphocytic Leukemia Research (5 papers), Acute Lymphoblastic Leukemia research (5 papers) and RNA modifications and cancer (4 papers). Elena Vendramini is often cited by papers focused on Chronic Lymphocytic Leukemia Research (5 papers), Acute Lymphoblastic Leukemia research (5 papers) and RNA modifications and cancer (4 papers). Elena Vendramini collaborates with scholars based in Italy, Switzerland and Germany. Elena Vendramini's co-authors include Valter Gattei, Tamara Bittolo, Antonella Zucchetto, Riccardo Bomben, Federico Pozzo, Erika Tissino, Shai Izraeli, Giuseppe Basso, Geertruy te Kronnie and Marco Giordan and has published in prestigious journals such as Blood, Cancer Cell and Leukemia.

In The Last Decade

Elena Vendramini

9 papers receiving 231 citations

Peers

Elena Vendramini
Jane Churpek United States
Barbara Neistadt United States
Olaf Hopfer Germany
Sonali Narang United States
Jane Churpek United States
Elena Vendramini
Citations per year, relative to Elena Vendramini Elena Vendramini (= 1×) peers Jane Churpek

Countries citing papers authored by Elena Vendramini

Since Specialization
Citations

This map shows the geographic impact of Elena Vendramini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elena Vendramini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elena Vendramini more than expected).

Fields of papers citing papers by Elena Vendramini

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Elena Vendramini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elena Vendramini. The network helps show where Elena Vendramini may publish in the future.

Co-authorship network of co-authors of Elena Vendramini

This figure shows the co-authorship network connecting the top 25 collaborators of Elena Vendramini. A scholar is included among the top collaborators of Elena Vendramini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elena Vendramini. Elena Vendramini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Vendramini, Elena, Riccardo Bomben, Federico Pozzo, et al.. (2022). KRAS and RAS-MAPK Pathway Deregulation in Mature B Cell Lymphoproliferative Disorders. Cancers. 14(3). 666–666. 19 indexed citations
2.
Pozzo, Federico, Tamara Bittolo, Erika Tissino, et al.. (2020). <i>SF3B1</i>-mutated chronic lymphocytic leukemia shows evidence of NOTCH1 pathway activation including CD20 downregulation. Haematologica. 106(12). 3125–3135. 13 indexed citations
3.
Pozzo, Federico, Tamara Bittolo, Elena Vendramini, et al.. (2017). NOTCH1-mutated chronic lymphocytic leukemia cells are characterized by a MYC-related overexpression of nucleophosmin 1 and ribosome-associated components. Leukemia. 31(11). 2407–2415. 46 indexed citations
4.
Pozzo, Federico, Tamara Bittolo, Elena Vendramini, et al.. (2017). NOTCH1 MUTATED CHRONIC LYMPHOCYTIC LEUKEMIA CELLS ARE CHARACTERIZED BY a MYC‐RELATED OVEREXPRESSION OF NUCLEOPHOSMIN‐1 AND RIBOSOME ASSOCIATED COMPONENTS. Hematological Oncology. 35(S2). 57–58.
5.
Vendramini, Elena, Marco Giordan, Emanuela Giarin, et al.. (2017). High expression of miR-125b-2 and SNORD116 noncoding RNA clusters characterize ERG-related B cell precursor acute lymphoblastic leukemia. Oncotarget. 8(26). 42398–42413. 19 indexed citations
6.
Vendramini, Elena, Yubin Ge, Yaron Goren, et al.. (2014). MicroRNA-486-5p is an erythroid oncomiR of the myeloid leukemias of Down syndrome. Blood. 125(8). 1292–1301. 64 indexed citations
7.
Kronnie, Geertruy te, Daniela Silvestri, Elena Vendramini, et al.. (2013). Philadelphia-Like Signature In Childhood Acute Lymphoblastic Leukemia: The AIEOP Experience. Blood. 122(21). 353–353. 9 indexed citations
8.
Meyer, Lüder Hinrich, Manon Queudeville, Johann M. Kraus, et al.. (2011). Early Relapse in ALL Is Identified by Time to Leukemia in NOD/SCID Mice and Is Characterized by a Gene Signature Involving Survival Pathways. Cancer Cell. 19(2). 206–217. 60 indexed citations
9.
Palmi, Chiara, Daniela Silvestri, Giulia Longinotti, et al.. (2011). Poor Prognosis for IKZF1 Intra-Gene Deletions in Pediatric Ph− B-Cell Precursor Acute Lymphoblastic Leukemia,. Blood. 118(21). 3518–3518. 1 indexed citations
10.
Cazzaniga, Giovanni, Elena Vendramini, Chiara Palmi, et al.. (2010). Biological Features and Prognostic Impact of CRLF2 Overexpression In Childhood ALL. Blood. 116(21). 271–271. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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