Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
HYPOCATALASEMIA: A NEW GENETIC CARRIER STATE*
1960598 citationsShigeo Takahara, Howard B. Hamilton et al.Journal of Clinical Investigationprofile →
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late)
cites ·
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Countries citing papers authored by Edwin T. Nishimura
Since
Specialization
Citations
This map shows the geographic impact of Edwin T. Nishimura's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edwin T. Nishimura with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edwin T. Nishimura more than expected).
Fields of papers citing papers by Edwin T. Nishimura
This network shows the impact of papers produced by Edwin T. Nishimura. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edwin T. Nishimura. The network helps show where Edwin T. Nishimura may publish in the future.
Co-authorship network of co-authors of Edwin T. Nishimura
This figure shows the co-authorship network connecting the top 25 collaborators of Edwin T. Nishimura.
A scholar is included among the top collaborators of Edwin T. Nishimura based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Edwin T. Nishimura. Edwin T. Nishimura is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Nishimura, Edwin T., et al.. (1972). Appearance of abnormal catalase antigen and concurrent deletion of normal antigen in leukocytes of myelogenous leukemia.. PubMed. 32(11). 2353–8.8 indexed citations
4.
Hokama, Y., et al.. (1970). Relationship of Cx-reactive protein response to induction of liver and kidney catalases following hematin administration into rabbits.. PubMed. 30(5). 1319–26.7 indexed citations
5.
Nishimura, Edwin T., et al.. (1970). Epizootic reticulum cell sarcoma in a sequestered colony of Japanese quails.. PubMed. 30(8). 2119–26.2 indexed citations
6.
Hokama, Y., et al.. (1969). The metabolic interrelationship and physicochemical analysis of C-reactive protein and hepatic catalase.. PubMed. 29(3). 542–8.6 indexed citations
Kaltenbach, John P., et al.. (1966). Hepatic catalase changes and leucine incorporation in talc-injected mice.. PubMed. 26(4). 580–4.5 indexed citations
Nishimura, Edwin T., et al.. (1966). Immunoelectrophoretic identification of catalase subcomponents in the homogenates of rat tissues.. PubMed. 26(1). 92–6.15 indexed citations
Nishimura, Edwin T., et al.. (1964). Isozymes of human and rat catalases. Archives of Biochemistry and Biophysics. 108(3). 452–459.24 indexed citations
14.
Nishimura, Edwin T., Sidney Rosenheim, & Louis Klein. (1963). Depression of hepatic catalase in mice after subcutaneous injury.. PubMed. 12. 415–21.7 indexed citations
15.
Ogata, Masana, et al.. (1962). The "catalase protein" of acatalasemic red blood cells. An electrophoretic and immunologic study.. PubMed. 11. 782–90.8 indexed citations
Takahara, Shigeo, Howard B. Hamilton, James V. Neel, et al.. (1960). HYPOCATALASEMIA: A NEW GENETIC CARRIER STATE*. Journal of Clinical Investigation. 39(4). 610–619.598 indexed citations breakdown →
18.
Wolf, Norman S. & Edwin T. Nishimura. (1960). Repression of methylcholanthrene-induced epidermal hyperplasia by hydrocortisone.. PubMed. 20. 1299–302.2 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.