Edwin H. Goodwin

3.7k total citations
50 papers, 3.1k citations indexed

About

Edwin H. Goodwin is a scholar working on Molecular Biology, Plant Science and Physiology. According to data from OpenAlex, Edwin H. Goodwin has authored 50 papers receiving a total of 3.1k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Molecular Biology, 19 papers in Plant Science and 13 papers in Physiology. Recurrent topics in Edwin H. Goodwin's work include DNA Repair Mechanisms (19 papers), Telomeres, Telomerase, and Senescence (13 papers) and Carcinogens and Genotoxicity Assessment (12 papers). Edwin H. Goodwin is often cited by papers focused on DNA Repair Mechanisms (19 papers), Telomeres, Telomerase, and Senescence (13 papers) and Carcinogens and Genotoxicity Assessment (12 papers). Edwin H. Goodwin collaborates with scholars based in United States, United Kingdom and Germany. Edwin H. Goodwin's co-authors include Bruce E. Lehnert, Susan M. Bailey, Michael N. Cornforth, Padma Kumar Narayanan, Atul Deshpande, Akihiro Kurimasa, Munira Kadhim, Julianne Meyne, J. Meyne and Babetta L. Marrone and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Oncogene.

In The Last Decade

Edwin H. Goodwin

49 papers receiving 3.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Edwin H. Goodwin United States 26 1.9k 1.2k 899 758 647 50 3.1k
Michael N. Cornforth United States 31 2.4k 1.3× 819 0.7× 789 0.9× 492 0.6× 1.4k 2.2× 73 3.3k
Sally A. Lorimore United Kingdom 30 1.5k 0.8× 2.4k 2.0× 1.4k 1.5× 231 0.3× 813 1.3× 50 4.1k
Hatsumi Nagasawa United States 29 1.9k 1.0× 1.8k 1.5× 1.2k 1.3× 142 0.2× 964 1.5× 65 3.4k
Seiji Kodama Japan 27 1.2k 0.7× 662 0.6× 383 0.4× 324 0.4× 495 0.8× 83 2.0k
Hiroshi Utsumi Japan 22 1.6k 0.8× 469 0.4× 350 0.4× 93 0.1× 624 1.0× 62 2.3k
Carl N. Sprung Australia 26 752 0.4× 344 0.3× 245 0.3× 328 0.4× 207 0.3× 50 1.4k
Adayabalam S. Balajee United States 28 2.2k 1.2× 307 0.3× 323 0.4× 195 0.3× 662 1.0× 82 2.9k
M.S. Sasaki Japan 29 1.3k 0.7× 587 0.5× 407 0.5× 81 0.1× 830 1.3× 69 2.4k
Carmel Mothersill Ireland 19 607 0.3× 1.3k 1.1× 891 1.0× 73 0.1× 322 0.5× 58 2.0k
K. M. S. Townsend United Kingdom 17 835 0.4× 477 0.4× 294 0.3× 106 0.1× 454 0.7× 33 1.7k

Countries citing papers authored by Edwin H. Goodwin

Since Specialization
Citations

This map shows the geographic impact of Edwin H. Goodwin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edwin H. Goodwin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edwin H. Goodwin more than expected).

Fields of papers citing papers by Edwin H. Goodwin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edwin H. Goodwin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edwin H. Goodwin. The network helps show where Edwin H. Goodwin may publish in the future.

Co-authorship network of co-authors of Edwin H. Goodwin

This figure shows the co-authorship network connecting the top 25 collaborators of Edwin H. Goodwin. A scholar is included among the top collaborators of Edwin H. Goodwin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edwin H. Goodwin. Edwin H. Goodwin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
McKenna, Miles J., Joel S. Bedford, Edwin H. Goodwin, et al.. (2019). Directional Genomic Hybridization (dGH) for Detection of Intrachromosomal Rearrangements. Methods in molecular biology. 1984. 107–116. 10 indexed citations
2.
McKenna, Miles J., et al.. (2017). Telomeres and NextGen CO-FISH: Directional Genomic Hybridization (Telo-dGH™). Methods in molecular biology. 1587. 103–112. 5 indexed citations
3.
Ray, F. Andrew, Erin Zimmerman, Bruce Robinson, et al.. (2013). Directional genomic hybridization for chromosomal inversion discovery and detection. Chromosome Research. 21(2). 165–174. 24 indexed citations
4.
Williams, Eli S., Michael N. Cornforth, Edwin H. Goodwin, & Susan M. Bailey. (2011). CO-FISH, COD-FISH, ReD-FISH, SKY-FISH. Methods in molecular biology. 735. 113–124. 19 indexed citations
5.
Hagelstrom, R. Tanner, Lila Ramaiah, Christian Desaintes, et al.. (2008). DNA-PKcs and ATM influence generation of ionizing radiation-induced bystander signals. Oncogene. 27(53). 6761–6769. 30 indexed citations
6.
Blagoev, Krastan B. & Edwin H. Goodwin. (2007). Telomere exchange and asymmetric segregation of chromosomes can account for the unlimited proliferative potential of ALT cell populations. DNA repair. 7(2). 199–204. 16 indexed citations
7.
Blagoev, Krastan B., Boian S. Alexandrov, Edwin H. Goodwin, & A. R. Bishop. (2006). Ultra-violet light induced changes in DNA dynamics may enhance TT-dimer recognition. DNA repair. 5(7). 863–867. 24 indexed citations
8.
Bailey, Susan M. & Edwin H. Goodwin. (2004). DNA and telomeres: beginnings and endings. Cytogenetic and Genome Research. 104(1-4). 109–115. 21 indexed citations
9.
Kadhim, Munira, Stephen Moore, & Edwin H. Goodwin. (2004). Interrelationships amongst radiation-induced genomic instability, bystander effects, and the adaptive response. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 568(1). 21–32. 120 indexed citations
10.
Donoho, Gregory P., Mark A. Brenneman, Tracy X. Cui, et al.. (2003). Deletion of Brca2 exon 27 causes hypersensitivity to DNA crosslinks, chromosomal instability, and reduced life span in mice. Genes Chromosomes and Cancer. 36(4). 317–331. 83 indexed citations
11.
Bailey, Susan M., Mark A. Brenneman, James Halbrook, et al.. (2003). The kinase activity of DNA-PK is required to protect mammalian telomeres. DNA repair. 3(3). 225–233. 69 indexed citations
12.
13.
Lehnert, Bruce E. & Edwin H. Goodwin. (1997). A new mechanism for DNA alterations induced by alpha particles such as those emitted by radon and radon progeny.. Environmental Health Perspectives. 105(suppl 5). 1095–1101. 41 indexed citations
14.
Meyne, J., et al.. (1996). A new method for detecting pericentric inversions using COD-FISH. Cytogenetic and Genome Research. 75(4). 248–253. 25 indexed citations
15.
Goodwin, Edwin H., Julianne Meyne, Susan M. Bailey, & Denise I. Quigley. (1996). On the origin of lateral asymmetry. Chromosoma. 104(5). 345–347. 15 indexed citations
16.
Goodwin, Edwin H. & Michael N. Cornforth. (1994). RBE: Mechanisms inferred from cytogenetics. Advances in Space Research. 14(10). 249–255. 5 indexed citations
17.
Meyne, Julianne, Edwin H. Goodwin, & Robert K. Moyzis. (1994). Chromosome localization and orientation of the simple sequence repeat of human satellite I DNA. Chromosoma. 103(2). 99–103. 26 indexed citations
18.
Goodwin, Edwin H. & J. Meyne. (1993). Strand-specific FISH reveals orientation of chromosome 18 alphoid DNA. Cytogenetic and Genome Research. 63(2). 126–127. 43 indexed citations
19.
Goodwin, Edwin H. & Eleanor A. Blakely. (1992). Heavy ion-induced chromosomal damage and repair. Advances in Space Research. 12(2-3). 81–89. 14 indexed citations
20.
Goodwin, Edwin H.. (1988). The LET Dependence of Interphase Chromosome Breakage and Rejoining in Two Mammalian Cell Lines. eScholarship (California Digital Library). 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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