Edel Kavanagh

2.2k total citations
19 papers, 1.7k citations indexed

About

Edel Kavanagh is a scholar working on Molecular Biology, Neurology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Edel Kavanagh has authored 19 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 7 papers in Neurology and 6 papers in Cellular and Molecular Neuroscience. Recurrent topics in Edel Kavanagh's work include Neuroinflammation and Neurodegeneration Mechanisms (7 papers), Cell death mechanisms and regulation (7 papers) and Nerve injury and regeneration (3 papers). Edel Kavanagh is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (7 papers), Cell death mechanisms and regulation (7 papers) and Nerve injury and regeneration (3 papers). Edel Kavanagh collaborates with scholars based in Sweden, Spain and Ireland. Edel Kavanagh's co-authors include Bertrand Joseph, Miguel Ángel Burguillos, José L. Venero, Jens Füllgrabe, Nabil Hajji, Tomas Deierborg, Annette Persson, Elisabet Englund, Albert García-Quintanilla and Patrik Brundin and has published in prestigious journals such as Nature, Nature Immunology and Oncogene.

In The Last Decade

Edel Kavanagh

19 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Edel Kavanagh Sweden 15 858 568 461 216 208 19 1.7k
Roser Gorina Spain 15 507 0.6× 761 1.3× 570 1.2× 195 0.9× 173 0.8× 15 1.7k
Lin Jia China 24 1.1k 1.2× 604 1.1× 417 0.9× 232 1.1× 359 1.7× 77 2.2k
Xingchun Gou China 25 793 0.9× 452 0.8× 280 0.6× 244 1.1× 298 1.4× 86 2.1k
Miguel Ángel Burguillos Spain 21 828 1.0× 800 1.4× 701 1.5× 263 1.2× 302 1.5× 33 2.1k
Tsuyoshi Nishioku Japan 26 624 0.7× 654 1.2× 244 0.5× 206 1.0× 290 1.4× 43 1.8k
Alexander M. Herrmann Germany 23 673 0.8× 535 0.9× 371 0.8× 294 1.4× 170 0.8× 51 1.6k
Kyungmin Ji United States 19 840 1.0× 521 0.9× 266 0.6× 204 0.9× 167 0.8× 32 1.9k
Yueting Zhang China 16 581 0.7× 647 1.1× 222 0.5× 169 0.8× 128 0.6× 33 1.6k
Fabìola Puentes United Kingdom 14 672 0.8× 626 1.1× 601 1.3× 197 0.9× 304 1.5× 26 2.1k

Countries citing papers authored by Edel Kavanagh

Since Specialization
Citations

This map shows the geographic impact of Edel Kavanagh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edel Kavanagh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edel Kavanagh more than expected).

Fields of papers citing papers by Edel Kavanagh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edel Kavanagh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edel Kavanagh. The network helps show where Edel Kavanagh may publish in the future.

Co-authorship network of co-authors of Edel Kavanagh

This figure shows the co-authorship network connecting the top 25 collaborators of Edel Kavanagh. A scholar is included among the top collaborators of Edel Kavanagh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edel Kavanagh. Edel Kavanagh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Ilkhanizadeh, Shirin, Andreas Åslund, Marcus Bäck, et al.. (2023). Live Detection of Neural Progenitors and Glioblastoma Cells by an Oligothiophene Derivative. ACS Applied Bio Materials. 6(9). 3790–3797. 1 indexed citations
2.
Kavanagh, Edel, et al.. (2022). Cell Death Related Proteins Beyond Apoptosis in the CNS. Frontiers in Cell and Developmental Biology. 9. 825747–825747. 8 indexed citations
3.
Rodríguez‐Gómez, José A., Edel Kavanagh, Pinelopi Engskog‐Vlachos, et al.. (2020). Microglia: Agents of the CNS Pro-Inflammatory Response. Cells. 9(7). 1717–1717. 261 indexed citations
4.
Watson, Paul, et al.. (2017). Bioengineered 3D Glial Cell Culture Systems and Applications for Neurodegeneration and Neuroinflammation. SLAS DISCOVERY. 22(5). 583–601. 55 indexed citations
5.
Shen, Xianli, Miguel Ángel Burguillos, Ahmed M. Osman, et al.. (2016). Glioma-induced inhibition of caspase-3 in microglia promotes a tumor-supportive phenotype. Nature Immunology. 17(11). 1282–1290. 74 indexed citations
6.
Burguillos, Miguel Ángel, Rocío M. de Pablos, Edel Kavanagh, et al.. (2016). Spatio-temporal activation of caspase-8 in myeloid cells upon ischemic stroke. Acta Neuropathologica Communications. 4(1). 92–92. 20 indexed citations
7.
Burguillos, Miguel Ángel, Martina Svensson, Tim Schulte, et al.. (2015). Microglia-Secreted Galectin-3 Acts as a Toll-like Receptor 4 Ligand and Contributes to Microglial Activation. Cell Reports. 10(9). 1626–1638. 278 indexed citations
8.
Kavanagh, Edel, Miguel Ángel Burguillos, Alejandro Carrillo-Jiménez, et al.. (2015). Deletion of caspase-8 in mouse myeloid cells blocks microglia pro-inflammatory activation and confers protection in MPTP neurodegeneration model. Aging. 7(9). 673–689. 26 indexed citations
9.
Mnich, Katarzyna, Laura A. Carleton, Edel Kavanagh, et al.. (2014). Nerve growth factor-mediated inhibition of apoptosis post-caspase activation is due to removal of active caspase-3 in a lysosome-dependent manner. Cell Death and Disease. 5(5). e1202–e1202. 46 indexed citations
10.
Kavanagh, Edel, et al.. (2014). Regulation of caspase-3 processing by cIAP2 controls the switch between pro-inflammatory activation and cell death in microglia. Cell Death and Disease. 5(12). e1565–e1565. 61 indexed citations
11.
Kavanagh, Edel. (2013). Quantification of Active Caspase-3 and Active Caspase-8 in Microglia Cells. Methods in molecular biology. 1041. 113–120. 3 indexed citations
12.
Kavanagh, Edel, et al.. (2013). TAp73β-mediated suppression of cell migration requires p57Kip2 control of actin cytoskeleton dynamics. Oncotarget. 4(2). 289–297. 13 indexed citations
13.
Kavanagh, Edel, et al.. (2012). p57KIP2 control of actin cytoskeleton dynamics is responsible for its mitochondrial pro-apoptotic effect. Cell Death and Disease. 3(5). e311–e311. 18 indexed citations
14.
Nürnberger, Sylvia, Ingrid Miller, J. Catharina Duvigneau, et al.. (2012). Impairment of endoplasmic reticulum in liver as an early consequence of the systemic inflammatory response in rats. American Journal of Physiology-Gastrointestinal and Liver Physiology. 303(12). G1373–G1383. 15 indexed citations
15.
Burguillos, Miguel Ángel, Tomas Deierborg, Edel Kavanagh, et al.. (2011). Caspase signalling controls microglia activation and neurotoxicity. Nature. 472(7343). 319–324. 478 indexed citations
16.
Kavanagh, Edel & Bertrand Joseph. (2011). The hallmarks of CDKN1C (p57, KIP2) in cancer. Biochimica et Biophysica Acta (BBA) - Reviews on Cancer. 1816(1). 50–56. 80 indexed citations
17.
Füllgrabe, Jens, Edel Kavanagh, & Bertrand Joseph. (2011). Histone onco-modifications. Oncogene. 30(31). 3391–3403. 216 indexed citations
18.
Szegezdi, Éva, et al.. (2008). Nerve growth factor blocks thapsigargin‐induced apoptosis at the level of the mitochondrion viaregulation of Bim. Journal of Cellular and Molecular Medicine. 12(6a). 2482–2496. 36 indexed citations
19.
Kavanagh, Edel, et al.. (2006). Functionality of NGF-protected PC12 cells following exposure to 6-hydroxydopamine. Biochemical and Biophysical Research Communications. 351(4). 890–895. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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