E. Perel

972 total citations
20 papers, 829 citations indexed

About

E. Perel is a scholar working on Genetics, Health, Toxicology and Mutagenesis and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, E. Perel has authored 20 papers receiving a total of 829 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Genetics, 7 papers in Health, Toxicology and Mutagenesis and 4 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in E. Perel's work include Estrogen and related hormone effects (12 papers), Effects and risks of endocrine disrupting chemicals (7 papers) and Hormonal and reproductive studies (4 papers). E. Perel is often cited by papers focused on Estrogen and related hormone effects (12 papers), Effects and risks of endocrine disrupting chemicals (7 papers) and Hormonal and reproductive studies (4 papers). E. Perel collaborates with scholars based in Canada, Israel and United States. E. Perel's co-authors include D.W. Killinger, H.R. Lindner, Andrew Zeitlin, A. M. Friedlander, Diana G. Wilkins, Martin E. Blackstein, William R. Lindsay, Marco M. Altaras, R. Tepper and Dov Flex and has published in prestigious journals such as The Journal of Clinical Endocrinology & Metabolism, Annals of the New York Academy of Sciences and Journal of Clinical Pathology.

In The Last Decade

E. Perel

19 papers receiving 757 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
E. Perel Canada	16	466	233	184	134	97	20	829
Leonard J. Lerner Malaysia	15	567 1.2×	189 0.8×	232 1.3×	148 1.1×	166 1.7×	49	1.0k
George E. Swaneck United States	13	438 0.9×	182 0.8×	369 2.0×	110 0.8×	38 0.4×	18	784
Claude Labrie Canada	15	442 0.9×	395 1.7×	307 1.7×	127 0.9×	109 1.1×	22	887
Hilton A. Salhanick United States	19	316 0.7×	253 1.1×	262 1.4×	60 0.4×	77 0.8×	53	935
S. Nilsson Sweden	13	514 1.1×	193 0.8×	377 2.0×	214 1.6×	51 0.5×	17	1.1k
A.E. Colás United States	15	172 0.4×	186 0.8×	244 1.3×	93 0.7×	63 0.6×	45	742
E. Ferguson United States	10	426 0.9×	89 0.4×	204 1.1×	39 0.3×	98 1.0×	16	679
Diane M. Klotz United States	11	440 0.9×	154 0.7×	301 1.6×	152 1.1×	76 0.8×	20	839
B.‐L. Nguyen France	15	504 1.1×	160 0.7×	253 1.4×	82 0.6×	72 0.7×	31	768
H.J. Kloosterboer Netherlands	24	637 1.4×	500 2.1×	300 1.6×	136 1.0×	496 5.1×	54	1.4k

Countries citing papers authored by E. Perel

Since Specialization

Citations

This map shows the geographic impact of E. Perel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E. Perel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E. Perel more than expected).

Fields of papers citing papers by E. Perel

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E. Perel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E. Perel. The network helps show where E. Perel may publish in the future.

Co-authorship network of co-authors of E. Perel

This figure shows the co-authorship network connecting the top 25 collaborators of E. Perel. A scholar is included among the top collaborators of E. Perel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E. Perel. E. Perel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Cohen, I., E. Perel, Dov Flex, et al.. (1999). Endometrial pathology in postmenopausal tamoxifen treatment: comparison between gynaecologically symptomatic and asymptomatic breast cancer patients.. Journal of Clinical Pathology. 52(4). 278–282. 49 indexed citations

Killinger, D.W., et al.. (1990). Influence of Adipose Tissue Distribution on the Biological Activity of Androgens^a. Annals of the New York Academy of Sciences. 595(1). 199–211. 47 indexed citations

Perel, E., et al.. (1988). Steroid modulation of aromatase activity in human cultured breast carcinoma cells. Journal of Steroid Biochemistry. 29(4). 393–399. 58 indexed citations

Killinger, D.W., et al.. (1987). The relationship between aromatase activity and body fat distribution. Steroids. 50(1-3). 61–72. 48 indexed citations

Killinger, D.W., et al.. (1987). Aromatase activity in the breast and other peripheral tissues and its therapeutic regulation. Steroids. 50(4-6). 523–536. 37 indexed citations

Perel, E., et al.. (1986). The Formation of 5α-Reduced Androgens in Stromal Cells from Human Breast Adipose Tissue*. The Journal of Clinical Endocrinology & Metabolism. 62(2). 314–318. 22 indexed citations

Perel, E., et al.. (1985). The relationship between growth and androstenedione metabolism in four cell lines of human breast carcinoma cells in culture. Molecular and Cellular Endocrinology. 41(2-3). 197–203. 16 indexed citations

Perel, E., et al.. (1984). The Intracellular Control of Aromatase Activity by 5α- Reduced Androgens in Human Breast Carcinoma Cells in Culture*. The Journal of Clinical Endocrinology & Metabolism. 58(3). 467–472. 16 indexed citations

Perel, E. & D.W. Killinger. (1983). The metabolism of androstenedione and testosterone to C19 metabolites in normal breast, breast carcinoma and benign prostatic hypertrophy tissue. Journal of Steroid Biochemistry. 19(2). 1135–1139. 31 indexed citations

10.

Perel, E., Martin E. Blackstein, & D.W. Killinger. (1983). 191 Control of aromatase activity in cultured human breast carcinoma cells. Journal of Steroid Biochemistry. 19. 64–64.

11.

Perel, E., et al.. (1983). Androstenedione metabolism in epithelial cells derived from early-lactation human milk. Steroids. 42(4). 389–399. 4 indexed citations

12.

Perel, E., Martin E. Blackstein, & D.W. Killinger. (1982). 237. Studies of androstenedione metabolism on breast carcinoma cells in culture with and without aromatase inhibitors. Journal of Steroid Biochemistry. 17(3). lxxix–lxxix. 1 indexed citations

13.

Perel, E., et al.. (1982). Aromatase in human breast carcinoma.. PubMed. 42(8 Suppl). 3369s–3372s. 27 indexed citations

14.

Perel, E., et al.. (1981). Effects of 4-hydroxy-4-androstene-3,17-dione and 10-propargylestr-4-ene-3,17-dione on the metabolism of androstenedione in human breast carcinoma and breast adipose tissues. Steroids. 38(4). 397–405. 10 indexed citations

15.

Perel, E., et al.. (1981). Androgen metabolism in male and female breast tissue. Steroids. 37(3). 345–352. 21 indexed citations

16.

Perel, E., Diana G. Wilkins, & D.W. Killinger. (1980). The conversion of androstenedione to estrone, estradiol, and testosterone in breast tissue. Journal of Steroid Biochemistry. 13(1). 89–94. 109 indexed citations

17.

Perel, E. & D.W. Killinger. (1979). The interconversion and aromatization of androgens by human adipose tissue. Journal of Steroid Biochemistry. 10(6). 623–627. 129 indexed citations

18.

Lindner, H.R., E. Perel, A. M. Friedlander, & Andrew Zeitlin. (1972). Specificity of antibodies to ovarian hormones in relation to the site of attachment of the steroid hapten to the peptide carrier. Steroids. 19(3). 357–375. 166 indexed citations

19.

Perel, E. & H.R. Lindner. (1970). DISSOCIATION OF UTEROTROPHIC ACTION FROM IMPLANTATION-INDUCING ACTIVITY IN TWO NON-STEROIDAL OESTROGENS (COUMESTROL AND GENISTEIN). Reproduction. 21(1). 171–175. 18 indexed citations

20.

Bauminger, S., H.R. Lindner, E. Perel, & Ruth Arnon. (1969). ANTIBODIES TO A PHYTO-OESTROGEN: ANTIGENICITY OF GENISTEIN COUPLED TO A SYNTHETIC POLYPEPTIDE. Journal of Endocrinology. 44(4). 567–578. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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