Drew Nassal

554 total citations
25 papers, 405 citations indexed

About

Drew Nassal is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Physiology. According to data from OpenAlex, Drew Nassal has authored 25 papers receiving a total of 405 indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Cardiology and Cardiovascular Medicine, 15 papers in Molecular Biology and 4 papers in Physiology. Recurrent topics in Drew Nassal's work include Cardiac electrophysiology and arrhythmias (14 papers), Ion channel regulation and function (11 papers) and Cardiac Fibrosis and Remodeling (7 papers). Drew Nassal is often cited by papers focused on Cardiac electrophysiology and arrhythmias (14 papers), Ion channel regulation and function (11 papers) and Cardiac Fibrosis and Remodeling (7 papers). Drew Nassal collaborates with scholars based in United States, Russia and Germany. Drew Nassal's co-authors include Thomas J. Hund, Isabelle Deschênes, Daniel Gratz, Eckhard Ficker, Nehal Patel, Adrienne T. Dennis, Peter J. Mohler, Amara Greer-Short, Sathya D. Unudurthi and Xiaoping Wan and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and PLoS ONE.

In The Last Decade

Drew Nassal

24 papers receiving 403 citations

Peers

Drew Nassal

CCM

CMN

PHYSI

Anne‐Coline Laurent France

Chastity L. Healy United States

Simon N.S. Louis Australia

Jianhua Huo China

Benoît‐Gilles Kerfant France

Tetsuro Marunouchi Japan

Walter E. Knight United States

Toshihide Kashihara Japan

Ruwan K. Perera Germany

Audrey Varin France

Anne‐Coline Laurent France

Drew Nassal

135 ×0.5

CCM

338 ×1.4

66 ×1.1

CMN

22 ×0.6

48 ×1.4

PHYSI

Citations per year, relative to Drew Nassal Drew Nassal (= 1×) peers Anne‐Coline Laurent

Countries citing papers authored by Drew Nassal

Since Specialization

Citations

This map shows the geographic impact of Drew Nassal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Drew Nassal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Drew Nassal more than expected).

Fields of papers citing papers by Drew Nassal

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Drew Nassal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Drew Nassal. The network helps show where Drew Nassal may publish in the future.

Co-authorship network of co-authors of Drew Nassal

This figure shows the co-authorship network connecting the top 25 collaborators of Drew Nassal. A scholar is included among the top collaborators of Drew Nassal based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Drew Nassal. Drew Nassal is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Johnson, C M, Drew Nassal, Xianyao Xu, et al.. (2025). The two-pore K⁺ channel TREK-1 regulates pressure overload-induced cardiac remodeling. American Journal of Physiology-Heart and Circulatory Physiology. 329(1). H178–H190.

Cervantes, Daniel O., Drew Nassal, Carl I. Thompson, et al.. (2024). Phosphorylation of cardiac sodium channel at Ser571 anticipates manifestations of the aging myopathy. American Journal of Physiology-Heart and Circulatory Physiology. 326(6). H1424–H1445. 2 indexed citations

Pinckard, Kelsey M., Shanna Hamilton, Radmila Terentyeva, et al.. (2024). Maternal exercise preserves offspring cardiovascular health via oxidative regulation of the ryanodine receptor. Molecular Metabolism. 82. 101914–101914. 2 indexed citations

King, D. Ryan, Mustafa Demirtaş, Drew Nassal, et al.. (2024). Cardiac-Specific Deletion of Scn8a Mitigates Dravet Syndrome-Associated Sudden Death in Adults. JACC. Clinical electrophysiology. 10(5). 829–842. 3 indexed citations

Nassal, Drew, et al.. (2023). Spectrin-Based Regulation of Cardiac Fibroblast Cell-Cell Communication. Cells. 12(5). 748–748. 1 indexed citations

Nassal, Drew, et al.. (2021). Ca2+/calmodulin kinase II–dependent regulation of βIV-spectrin modulates cardiac fibroblast gene expression, proliferation, and contractility. Journal of Biological Chemistry. 297(1). 100893–100893. 7 indexed citations

Dewal, Revati S., Amara Greer-Short, Shinsuke Nirengi, et al.. (2021). Phospho-ablation of cardiac sodium channel Nav1.5 mitigates susceptibility to atrial fibrillation and improves glucose homeostasis under conditions of diet-induced obesity. International Journal of Obesity. 45(4). 795–807. 17 indexed citations

Nassal, Drew, Daniel Gratz, & Thomas J. Hund. (2020). Challenges and Opportunities for Therapeutic Targeting of Calmodulin Kinase II in Heart. Frontiers in Pharmacology. 11. 35–35. 46 indexed citations

Unudurthi, Sathya D., Drew Nassal, Nehal Patel, et al.. (2020). Fibroblast growth factor-inducible 14 mediates macrophage infiltration in heart to promote pressure overload-induced cardiac dysfunction. Life Sciences. 247. 117440–117440. 22 indexed citations

10.

Patel, Nehal, Drew Nassal, Daniel Gratz, & Thomas J. Hund. (2020). Emerging therapeutic targets for cardiac arrhythmias: role of STAT3 in regulating cardiac fibroblast function. Expert Opinion on Therapeutic Targets. 25(1). 63–73. 11 indexed citations

11.

Patel, Nehal, Drew Nassal, Amara Greer-Short, et al.. (2019). βIV-Spectrin/STAT3 complex regulates fibroblast phenotype, fibrosis, and cardiac function. JCI Insight. 4(20). 17 indexed citations

12.

Unudurthi, Sathya D., Drew Nassal, Amara Greer-Short, et al.. (2018). βIV-Spectrin regulates STAT3 targeting to tune cardiac response to pressure overload. Journal of Clinical Investigation. 128(12). 5561–5572. 35 indexed citations

13.

Nassal, Drew, Xiaoping Wan, Haiyan Liu, Kenneth R. Laurita, & Isabelle Deschênes. (2017). KChIP2 regulates the cardiac Ca2+ transient and myocyte contractility by targeting ryanodine receptor activity. PLoS ONE. 12(4). e0175221–e0175221. 10 indexed citations

14.

Nassal, Drew, Xiaoping Wan, Haiyan Liu, et al.. (2017). KChIP2 is a core transcriptional regulator of cardiac excitability. eLife. 6. 27 indexed citations

15.

Nassal, Drew, et al.. (2016). Mesenchymal stem cells suppress cardiac alternans by activation of PI3K mediated nitroso-redox pathway. Journal of Molecular and Cellular Cardiology. 98. 138–145. 10 indexed citations

16.

Nassal, Drew, Xiaoping Wan, Haiyan Liu, & Isabelle Deschênes. (2016). Myocardial KChIP2 Expression in Guinea Pig Resolves an Expanded Electrophysiologic Role. PLoS ONE. 11(1). e0146561–e0146561. 10 indexed citations

17.

Dennis, Adrienne T., Lu Wang, Hanlin Wan, et al.. (2012). Molecular Determinants of Pentamidine-Induced hERG Trafficking Inhibition. Biophysical Journal. 102(3). 679a–680a. 4 indexed citations

18.

Dennis, Adrienne T., Drew Nassal, Isabelle Deschênes, Dierk Thomas, & Eckhard Ficker. (2011). Antidepressant-induced Ubiquitination and Degradation of the Cardiac Potassium Channel hERG. Journal of Biological Chemistry. 286(39). 34413–34425. 38 indexed citations

19.

Dennis, Adrienne T., Lu Wang, Hanlin Wan, et al.. (2011). Molecular Determinants of Pentamidine-Induced hERG Trafficking Inhibition. Molecular Pharmacology. 81(2). 198–209. 52 indexed citations

20.

Mohr, Susanne, Jason Vincent, Vijay P. Sarthy, et al.. (2009). Hyperglycemia Leads to Müller Cell Death through Induction of Pyroptosis-Like Mechanisms. Investigative Ophthalmology & Visual Science. 50(13). 15–15. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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