Douglas S. Smoot

459 total citations
8 papers, 404 citations indexed

About

Douglas S. Smoot is a scholar working on Immunology, Genetics and Virology. According to data from OpenAlex, Douglas S. Smoot has authored 8 papers receiving a total of 404 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 1 paper in Genetics and 1 paper in Virology. Recurrent topics in Douglas S. Smoot's work include Immune Cell Function and Interaction (6 papers), T-cell and B-cell Immunology (5 papers) and Immunotherapy and Immune Responses (4 papers). Douglas S. Smoot is often cited by papers focused on Immune Cell Function and Interaction (6 papers), T-cell and B-cell Immunology (5 papers) and Immunotherapy and Immune Responses (4 papers). Douglas S. Smoot collaborates with scholars based in United States. Douglas S. Smoot's co-authors include Carl H. June, Ryo Abe, Nancy Craighead, Peter Vandenberghe, Daniel C. St. Louis, Patrick J. Blair, Mark A. Moorman, J J Mond, Hiroshi Yamaguchi and Clifford M. Snapper and has published in prestigious journals such as The Journal of Immunology, Transplantation and Immunology.

In The Last Decade

Douglas S. Smoot

8 papers receiving 401 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Douglas S. Smoot United States 8 320 85 60 32 28 8 404
Catherine Dalbiez‐Gauthier France 7 358 1.1× 71 0.8× 69 1.1× 57 1.8× 21 0.8× 12 470
Emma McKenzie United Kingdom 9 261 0.8× 173 2.0× 46 0.8× 41 1.3× 28 1.0× 12 404
K. Inaba Japan 7 617 1.9× 107 1.3× 105 1.8× 53 1.7× 15 0.5× 9 708
Atef Allam United States 11 237 0.7× 83 1.0× 52 0.9× 15 0.5× 16 0.6× 13 340
Philipp Kohl Germany 4 357 1.1× 125 1.5× 48 0.8× 47 1.5× 7 0.3× 5 434
Jeff Hooley United States 7 280 0.9× 124 1.5× 71 1.2× 26 0.8× 8 0.3× 15 455
Juliana B. Woodcock United States 5 167 0.5× 114 1.3× 60 1.0× 20 0.6× 6 0.2× 5 297
G. De Petrillo Italy 9 235 0.7× 131 1.5× 55 0.9× 32 1.0× 20 0.7× 14 443
Susan F. Grammer United States 12 407 1.3× 99 1.2× 65 1.1× 74 2.3× 32 1.1× 15 531
N Shinohara Japan 7 318 1.0× 111 1.3× 84 1.4× 8 0.3× 19 0.7× 7 427

Countries citing papers authored by Douglas S. Smoot

Since Specialization
Citations

This map shows the geographic impact of Douglas S. Smoot's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Douglas S. Smoot with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Douglas S. Smoot more than expected).

Fields of papers citing papers by Douglas S. Smoot

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Douglas S. Smoot. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Douglas S. Smoot. The network helps show where Douglas S. Smoot may publish in the future.

Co-authorship network of co-authors of Douglas S. Smoot

This figure shows the co-authorship network connecting the top 25 collaborators of Douglas S. Smoot. A scholar is included among the top collaborators of Douglas S. Smoot based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Douglas S. Smoot. Douglas S. Smoot is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Louis, Daniel C. St., Juliana B. Woodcock, Patrick J. Blair, et al.. (2004). Evidence for Distinct Intracellular Signaling Pathways in CD34+ Progenitor to Dendritic Cell Differentiation from a Human Cell Line Model. The Journal of Immunology. 173(10). 6490–6490. 67 indexed citations
2.
Akpınar, Edip, Nancy Craighead, Douglas S. Smoot, & Douglas A. Hale. (2004). Potent Skin Allograft Survival Prolongation Using a Committed Progenitor Fraction of Bone Marrow in Mice. Transplantation. 78(3). 383–391. 7 indexed citations
3.
Haynes, J. David, J. Kathleen Moch, & Douglas S. Smoot. (2003). Erythrocytic Malaria Growth or Invasion Inhibition Assays with Emphasis on Suspension Culture GIA. Humana Press eBooks. 72. 535–554. 21 indexed citations
4.
Louis, Daniel C. St., Juliana B. Woodcock, Patrick J. Blair, et al.. (1999). Evidence for Distinct Intracellular Signaling Pathways in CD34+ Progenitor to Dendritic Cell Differentiation from a Human Cell Line Model. The Journal of Immunology. 162(6). 3237–3248. 83 indexed citations
5.
Perrin, P J, et al.. (1997). Mitogenic stimulation of T cells reveals differing contributions for B7‐1 (CD80) and B7‐2 (CD86) costimulation. Immunology. 90(4). 534–542. 43 indexed citations
6.
Hickey, Theresa E., Patrick J. Blair, Stephen P. Perfetto, et al.. (1996). Intact antigen receptor-mediated calcium signals in patients with early stage HIV-1 infection. The Journal of Immunology. 156(10). 4012–4017. 7 indexed citations
7.
Abe, Ryo, et al.. (1995). Distinct signal transduction in mouse CD4+ and CD8+ splenic T cells after CD28 receptor ligation.. The Journal of Immunology. 154(3). 985–997. 117 indexed citations
8.
Snapper, Clifford M., et al.. (1993). Natural killer cells induce activated murine B cells to secrete Ig.. The Journal of Immunology. 151(10). 5251–5260. 59 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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