Douglas E. Vetter

5.4k total citations · 2 hit papers
55 papers, 4.3k citations indexed

About

Douglas E. Vetter is a scholar working on Sensory Systems, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, Douglas E. Vetter has authored 55 papers receiving a total of 4.3k indexed citations (citations by other indexed papers that have themselves been cited), including 35 papers in Sensory Systems, 28 papers in Molecular Biology and 14 papers in Cognitive Neuroscience. Recurrent topics in Douglas E. Vetter's work include Hearing, Cochlea, Tinnitus, Genetics (34 papers), Nicotinic Acetylcholine Receptors Study (23 papers) and Ion channel regulation and function (13 papers). Douglas E. Vetter is often cited by papers focused on Hearing, Cochlea, Tinnitus, Genetics (34 papers), Nicotinic Acetylcholine Receptors Study (23 papers) and Ion channel regulation and function (13 papers). Douglas E. Vetter collaborates with scholars based in United States, Argentina and Germany. Douglas E. Vetter's co-authors include Jim Boulter, Stephen F. Heinemann, Ana Belén Elgoyhen, Eleonora Katz, Enrico Mugnaini, Stefan H. Heinemann, David S. Johnson, Carla V. Rothlin, M. Charles Liberman and Jeffrey R. Mann and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Douglas E. Vetter

54 papers receiving 4.2k citations

Hit Papers

α9: An acetylcholine receptor with novel pharmacological ... 1994 2026 2004 2015 1994 2001 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Douglas E. Vetter United States 29 2.5k 2.0k 1.1k 894 631 55 4.3k
Ana Belén Elgoyhen Argentina 37 2.5k 1.0× 2.8k 1.4× 916 0.8× 1.6k 1.8× 1.3k 2.0× 111 5.0k
Marlies Knipper Germany 48 2.6k 1.0× 4.2k 2.1× 1.9k 1.8× 2.0k 2.2× 1.6k 2.5× 167 7.0k
Leonardo Belluscio United States 29 2.0k 0.8× 1.6k 0.8× 4.4k 4.1× 482 0.5× 430 0.7× 52 6.3k
Thomas N. Parks United States 35 1.1k 0.4× 1.4k 0.7× 1.6k 1.5× 849 0.9× 331 0.5× 60 3.5k
Elisabeth Glowatzki United States 34 2.0k 0.8× 2.9k 1.4× 1.4k 1.3× 1.4k 1.5× 688 1.1× 63 4.6k
Masahiro Yamaguchi Japan 34 2.1k 0.8× 1.0k 0.5× 2.7k 2.5× 617 0.7× 1.2k 1.9× 72 6.3k
Jean‐Didier Vincent France 42 1.8k 0.7× 869 0.4× 2.3k 2.2× 480 0.5× 393 0.6× 111 5.2k
Charles A. Greer United States 51 1.6k 0.7× 4.4k 2.2× 4.8k 4.4× 534 0.6× 922 1.5× 157 7.9k
Alexander T. Chesler United States 30 1.3k 0.5× 1.2k 0.6× 1.2k 1.1× 246 0.3× 166 0.3× 43 3.7k
Benjamin R. Arenkiel United States 35 1.5k 0.6× 416 0.2× 1.7k 1.5× 942 1.1× 524 0.8× 97 4.4k

Countries citing papers authored by Douglas E. Vetter

Since Specialization
Citations

This map shows the geographic impact of Douglas E. Vetter's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Douglas E. Vetter with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Douglas E. Vetter more than expected).

Fields of papers citing papers by Douglas E. Vetter

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Douglas E. Vetter. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Douglas E. Vetter. The network helps show where Douglas E. Vetter may publish in the future.

Co-authorship network of co-authors of Douglas E. Vetter

This figure shows the co-authorship network connecting the top 25 collaborators of Douglas E. Vetter. A scholar is included among the top collaborators of Douglas E. Vetter based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Douglas E. Vetter. Douglas E. Vetter is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Beebe, Nichole L., et al.. (2020). Generation of a ChAT mouse line without the early onset hearing loss typical of the C57BL/6J strain. Hearing Research. 388. 107896–107896. 3 indexed citations
3.
Baumann, Lisa, Lutz Dürselen, Gabriela Krasteva‐Christ, et al.. (2018). Deletion of nicotinic acetylcholine receptor alpha9 in mice resulted in altered bone structure. Bone. 120. 285–296. 15 indexed citations
6.
Graham, Christine, et al.. (2011). The Cochlear CRF Signaling Systems and their Mechanisms of Action in Modulating Cochlear Sensitivity and Protection Against Trauma. Molecular Neurobiology. 44(3). 383–406. 16 indexed citations
8.
Maison, Stéphane F., Douglas E. Vetter, Ruth Anne Eatock, et al.. (2010). Muscarinic Signaling in the Cochlea: Presynaptic and Postsynaptic Effects on Efferent Feedback and Afferent Excitability. Journal of Neuroscience. 30(19). 6751–6762. 30 indexed citations
9.
Graham, Christine, et al.. (2010). A corticotropin-releasing factor system expressed in the cochlea modulates hearing sensitivity and protects against noise-induced hearing loss. Neurobiology of Disease. 38(2). 246–258. 24 indexed citations
10.
Turcan, Şevin, Donna K. Slonim, & Douglas E. Vetter. (2010). Lack of nAChR Activity Depresses Cochlear Maturation and Up-Regulates GABA System Components: Temporal Profiling of Gene Expression in α9 Null Mice. PLoS ONE. 5(2). e9058–e9058. 17 indexed citations
11.
Turcan, Şevin, et al.. (2010). Corticotropin‐releasing factor‐2 activation prevents gentamicin‐induced oxidative stress in cells derived from the inner ear. Journal of Neuroscience Research. 88(13). 2976–2990. 13 indexed citations
12.
Turcan, Şevin, et al.. (2010). MINING FUNCTIONALLY RELEVANT GENE SETS FOR ANALYZING PHYSIOLOGICALLY NOVEL CLINICAL EXPRESSION DATA. WORLD SCIENTIFIC eBooks. 50–61. 3 indexed citations
13.
Brown, M. Christian & Douglas E. Vetter. (2008). Olivocochlear Neuron Central Anatomy Is Normal in α9 Knockout Mice. Journal of the Association for Research in Otolaryngology. 10(1). 64–75. 14 indexed citations
14.
He, David Z. Z., et al.. (2003). Mouse outer hair cells lacking the α9 ACh receptor are motile. Developmental Brain Research. 148(1). 19–25. 8 indexed citations
15.
May, Bradford J., Cynthia A. Prosen, Donna Weiss, & Douglas E. Vetter. (2002). Behavioral investigation of some possible effects of the central olivocochlear pathways in transgenic mice. Hearing Research. 171(1-2). 142–157. 31 indexed citations
16.
Prosen, Cynthia A., Kevin G. Bath, Douglas E. Vetter, & Bradford J. May. (2000). Behavioral assessments of auditory sensitivity in transgenic mice. Journal of Neuroscience Methods. 97(1). 59–67. 17 indexed citations
17.
Vetter, Douglas E., M. Charles Liberman, Jeffrey R. Mann, et al.. (1999). Role of α9 Nicotinic ACh Receptor Subunits in the Development and Function of Cochlear Efferent Innervation. Neuron. 23(1). 93–103. 230 indexed citations
18.
Vetter, Douglas E., Jeffrey R. Mann, Philine Wangemann, et al.. (1996). Inner Ear Defects Induced by Null Mutationof the isk Gene. Neuron. 17(6). 1251–1264. 302 indexed citations
19.
Johnson, David S., et al.. (1994). α9: An acetylcholine receptor with novel pharmacological properties expressed in rat cochlear hair cells. Cell. 79(4). 705–715. 725 indexed citations breakdown →
20.
Vetter, Douglas E., Joe C. Adams, & Enrico Mugnaini. (1991). Chemically distinct rat olivocochlear neurons. Synapse. 7(1). 21–43. 152 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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