Doris Taha

1.3k total citations
27 papers, 655 citations indexed

About

Doris Taha is a scholar working on Genetics, Endocrinology, Diabetes and Metabolism and Surgery. According to data from OpenAlex, Doris Taha has authored 27 papers receiving a total of 655 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Genetics, 12 papers in Endocrinology, Diabetes and Metabolism and 11 papers in Surgery. Recurrent topics in Doris Taha's work include Pancreatic function and diabetes (9 papers), Diabetes and associated disorders (5 papers) and Neonatal Health and Biochemistry (4 papers). Doris Taha is often cited by papers focused on Pancreatic function and diabetes (9 papers), Diabetes and associated disorders (5 papers) and Neonatal Health and Biochemistry (4 papers). Doris Taha collaborates with scholars based in United States, Saudi Arabia and Türkiye. Doris Taha's co-authors include Céline Charon, Pascal Boileau, Hervé Blanc, Pierre Bougnères, Daorong Feng, Sabine Duchatelet, Jack‐Christophe Cossec, Claude Chelala, Douglas R. Cavener and Cécile Julier and has published in prestigious journals such as Nature Genetics, The Journal of Clinical Endocrinology & Metabolism and Molecular Genetics and Metabolism.

In The Last Decade

Doris Taha

24 papers receiving 642 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Doris Taha United States 13 378 331 266 238 58 27 655
Sameer Kassem Israel 8 227 0.6× 321 1.0× 291 1.1× 156 0.7× 52 0.9× 33 560
Petra Dušátková Czechia 17 509 1.3× 430 1.3× 384 1.4× 314 1.3× 41 0.7× 60 817
Abdelhadi Habeb Saudi Arabia 16 371 1.0× 426 1.3× 355 1.3× 199 0.8× 16 0.3× 40 721
Agnieszka Gach Poland 11 152 0.4× 210 0.6× 147 0.6× 202 0.8× 38 0.7× 52 570
Anne Lienhardt France 12 177 0.5× 161 0.5× 175 0.7× 173 0.7× 36 0.6× 26 636
Jürgen Herwig Germany 9 379 1.0× 100 0.3× 192 0.7× 157 0.7× 158 2.7× 12 705
Dalong Zhu China 12 332 0.9× 393 1.2× 378 1.4× 138 0.6× 24 0.4× 19 649
Nóra Hosszúfalusi Hungary 14 500 1.3× 415 1.3× 421 1.6× 158 0.7× 11 0.2× 44 768
Ru Gao China 13 176 0.5× 305 0.9× 154 0.6× 223 0.9× 10 0.2× 35 606
Michael Hannemann United Kingdom 10 163 0.4× 217 0.7× 160 0.6× 181 0.8× 11 0.2× 13 589

Countries citing papers authored by Doris Taha

Since Specialization
Citations

This map shows the geographic impact of Doris Taha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Doris Taha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Doris Taha more than expected).

Fields of papers citing papers by Doris Taha

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Doris Taha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Doris Taha. The network helps show where Doris Taha may publish in the future.

Co-authorship network of co-authors of Doris Taha

This figure shows the co-authorship network connecting the top 25 collaborators of Doris Taha. A scholar is included among the top collaborators of Doris Taha based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Doris Taha. Doris Taha is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Taha, Doris, et al.. (2023). THU230 Consumptive Hypothyroidism Secondary To Hemangioma. Journal of the Endocrine Society. 7(Supplement_1).
2.
Taha, Doris, et al.. (2020). Familial neonatal nonautoimmune hyperthyroidism due to a gain-of-function (D619G) thyrotropin-receptor mutation. Journal of Pediatric Endocrinology and Metabolism. 34(2). 267–271. 4 indexed citations
3.
Taha, Doris, et al.. (2019). SAT-297 Mutation in SRY Gene Presenting as Syndromic 46XY Disorder of Sexual Differentiation (DSD). Journal of the Endocrine Society. 3(Supplement_1).
4.
Dimitri, Paul, Elisa De Franco, Abdelhadi Habeb, et al.. (2016). An emerging, recognizable facial phenotype in association with mutations in GLI‐similar 3 (GLIS3). American Journal of Medical Genetics Part A. 170(7). 1918–1923. 16 indexed citations
5.
Dimitri, Paul, Abdelhadi Habeb, Ann Millward, et al.. (2015). Expanding the Clinical Spectrum Associated WithGLIS3Mutations. The Journal of Clinical Endocrinology & Metabolism. 100(10). E1362–E1369. 54 indexed citations
6.
Malloy, Peter J., Velibor Tasić, Doris Taha, et al.. (2013). Vitamin D receptor mutations in patients with hereditary 1,25-dihydroxyvitamin D-resistant rickets. Molecular Genetics and Metabolism. 111(1). 33–40. 39 indexed citations
7.
8.
Taha, Doris, Amélie Bonnefond, Marion Marchand, et al.. (2008). Neonatal diabetes mellitus because of pancreatic agenesis with dysmorphic features and recurrent bacterial infections. Pediatric Diabetes. 9(3pt1). 240–244. 6 indexed citations
9.
Taha, Doris, et al.. (2007). A novel VPS33B mutation in an ARC syndrome patient presenting with osteopenia and fractures at birth. American Journal of Medical Genetics Part A. 143A(23). 2835–2837. 16 indexed citations
10.
Senée, Valérie, Claude Chelala, Sabine Duchatelet, et al.. (2006). Mutations in GLIS3 are responsible for a rare syndrome with neonatal diabetes mellitus and congenital hypothyroidism. Nature Genetics. 38(6). 682–687. 270 indexed citations
11.
Taha, Doris, et al.. (2006). Type 2 Diabetes Mellitus in African-American Adolescents: Impaired Beta-Cell Function in the Face of Severe Insulin Resistance. Journal of Pediatric Endocrinology and Metabolism. 19(2). 135–142. 18 indexed citations
12.
El‐Naggar, Walid, et al.. (2005). Nephrocalcinosis in glucose-galactose malabsorption, association with renal tubular acidosis. Pediatric Nephrology. 20(9). 1336–1339. 9 indexed citations
13.
Taha, Doris, Primus-E. Mullis, Lourdes Ibáñez, & Francis de Zegher. (2005). Absent or Delayed Adrenarche in Pit-1/POU1F1 Deficiency. Hormone Research in Paediatrics. 64(4). 175–179. 24 indexed citations
14.
Taha, Doris, et al.. (2005). Arthrogryposis, renal tubular acidosis and cholestasis (ARC) syndrome: two new cases and review. Clinical Dysmorphology. 14(4). 191–196. 21 indexed citations
15.
Taha, Doris, et al.. (2004). Fatal lymphoproliferative disorder in a child with Schimke immuno‐osseous dysplasia. American Journal of Medical Genetics Part A. 131A(2). 194–199. 9 indexed citations
16.
AvRuskin, Theodore W., et al.. (2003). Monozygotic Twins with Congenital Adrenal Hyperplasia: Long-term Endocrine Evaluation and Gene Analysis. Journal of Pediatric Endocrinology and Metabolism. 16(4). 565–70. 2 indexed citations
17.
Taha, Doris, et al.. (2003). Neonatal diabetes mellitus, congenital hypothyroidism, hepatic fibrosis, polycystic kidneys, and congenital glaucoma: A new autosomal recessive syndrome?. American Journal of Medical Genetics Part A. 122A(3). 269–273. 56 indexed citations
18.
Taha, Doris, et al.. (2002). Beta-Cell Response to Intravenous Glucagon in African-American and Hispanic Children with Type 2 Diabetes Mellitus. Journal of Pediatric Endocrinology and Metabolism. 15(1). 59–67. 12 indexed citations
19.
Umpaichitra, Vatcharapan, et al.. (2001). C-peptide and Glucagon Profiles in Minority Children with Type 2 Diabetes Mellitus. The Journal of Clinical Endocrinology & Metabolism. 86(4). 1605–1609. 27 indexed citations
20.
Taha, Doris, et al.. (2001). Growth Hormone Replacement Therapy in Children with Leukemia in Remission. Clinical Pediatrics. 40(8). 441–445. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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