Dora Cavallo‐Medved
- Molecular Biology
- Cancer Research top 5%
- Oncology top 10%
- Cell Biology top 5%
- Immunology and Allergy top 5%
- Co-authors
- Bonnie F. SloaneMansoureh SameniMona Mostafa MohamedJulie DosescuKamiar MoinArulselvi AnbalaganMohamed El‐ShinawiMohamed A. Nouh
- Topics
- Protease and Inhibitor Mechanisms (14 papers)Cell Adhesion Molecules Research (14 papers)Caveolin-1 and cellular processes (8 papers)
- Journals
- Journal of Clinical OncologySHILAP Revista de lepidopterologíaPLoS ONE
- Partner nations
- CanadaUnited StatesEgypt
In The Last Decade
Dora Cavallo‐Medved
36 papers receiving 1.3k citations
Peers
Comparison fields: 5 of 115
- Molecular Biology 601
- Cancer Research 564
- Oncology 437
- Cell Biology 230
- Immunology and Allergy 200
Countries citing papers authored by Dora Cavallo‐Medved
This map shows the geographic impact of Dora Cavallo‐Medved's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dora Cavallo‐Medved with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dora Cavallo‐Medved more than expected).
Fields of papers citing papers by Dora Cavallo‐Medved
This network shows the impact of papers produced by Dora Cavallo‐Medved. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dora Cavallo‐Medved. The network helps show where Dora Cavallo‐Medved may publish in the future.
Co-authorship network of co-authors of Dora Cavallo‐Medved
This figure shows the co-authorship network connecting the top 25 collaborators of Dora Cavallo‐Medved. A scholar is included among the top collaborators of Dora Cavallo‐Medved based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dora Cavallo‐Medved. Dora Cavallo‐Medved is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 1 | |
| 3 | 1 | |
| 4 | 3 | |
| 5 | 7 | |
| 6 | 22 | |
| 7 | 25 | |
| 8 | 3 | |
| 9 | 3 | |
| 10 | 183 | |
| 11 | 95 | |
| 12 | 50 | |
| 13 | 96 | |
| 14 | 92 | |
| 15 | 37 | |
| 16 | 44 | |
| 17 | 36 | |
| 18 | 141 | |
| 19 | 49 | |
| 20 | 74 |
About Dora Cavallo‐Medved
Dora Cavallo‐Medved is a scholar working on Immunology and Allergy, Cancer Research and Cell Biology, having authored 37 papers that have together received 1.3k indexed citations. Recurring topics across this work include Protease and Inhibitor Mechanisms (14 papers), Cell Adhesion Molecules Research (14 papers) and Caveolin-1 and cellular processes (8 papers). The work is most often cited by research in Cancer Research (564 citations), Immunology and Allergy (200 citations) and Oncology (437 citations). Dora Cavallo‐Medved has collaborated with scholars based in Canada, United States and Egypt. Frequent co-authors include Bonnie F. Sloane, Mansoureh Sameni, Mona Mostafa Mohamed, Julie Dosescu, Kamiar Moin, Arulselvi Anbalagan, Mohamed El‐Shinawi, Mohamed A. Nouh, Hussein Khaled and Bruce E. Linebaugh. Their work appears in journals such as Journal of Clinical Oncology, SHILAP Revista de lepidopterología and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.