Donna P. Frazier

988 total citations
19 papers, 854 citations indexed

About

Donna P. Frazier is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Donna P. Frazier has authored 19 papers receiving a total of 854 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 10 papers in Oncology and 4 papers in Cancer Research. Recurrent topics in Donna P. Frazier's work include Cancer-related Molecular Pathways (9 papers), Ubiquitin and proteasome pathways (7 papers) and RNA modifications and cancer (4 papers). Donna P. Frazier is often cited by papers focused on Cancer-related Molecular Pathways (9 papers), Ubiquitin and proteasome pathways (7 papers) and RNA modifications and cancer (4 papers). Donna P. Frazier collaborates with scholars based in United States. Donna P. Frazier's co-authors include Kazushi Inoue, Nanette H. Bishopric, Keith A. Webster, Ali Mallakin, Huifang Li, Takayuki Sugiyama, Elizabeth A. Fry, John W. Thompson, Regina M. Graham and Pankaj Taneja and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Cancer Cell.

In The Last Decade

Donna P. Frazier

19 papers receiving 842 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Donna P. Frazier United States 17 562 294 156 123 81 19 854
Kanika A. Bowen United States 12 647 1.2× 288 1.0× 221 1.4× 97 0.8× 93 1.1× 15 943
Yvette Chin United States 14 685 1.2× 226 0.8× 145 0.9× 63 0.5× 74 0.9× 18 938
Wilma Jogunoori United States 13 498 0.9× 390 1.3× 190 1.2× 100 0.8× 140 1.7× 22 871
Pu Zhang China 13 450 0.8× 199 0.7× 112 0.7× 83 0.7× 53 0.7× 19 876
Pavel Pitule Czechia 16 429 0.8× 264 0.9× 285 1.8× 133 1.1× 81 1.0× 29 884
Zarir E. Karanjawala United States 14 708 1.3× 330 1.1× 222 1.4× 65 0.5× 44 0.5× 23 1.1k
Gang Hu China 14 448 0.8× 245 0.8× 143 0.9× 74 0.6× 58 0.7× 40 851
Laura Antonucci Italy 14 446 0.8× 209 0.7× 120 0.8× 118 1.0× 36 0.4× 21 746
Yan-Gao Man United States 15 525 0.9× 423 1.4× 278 1.8× 149 1.2× 99 1.2× 22 1.1k
Mark J. Cooper Australia 18 446 0.8× 232 0.8× 135 0.9× 107 0.9× 64 0.8× 37 1.6k

Countries citing papers authored by Donna P. Frazier

Since Specialization
Citations

This map shows the geographic impact of Donna P. Frazier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Donna P. Frazier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Donna P. Frazier more than expected).

Fields of papers citing papers by Donna P. Frazier

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Donna P. Frazier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Donna P. Frazier. The network helps show where Donna P. Frazier may publish in the future.

Co-authorship network of co-authors of Donna P. Frazier

This figure shows the co-authorship network connecting the top 25 collaborators of Donna P. Frazier. A scholar is included among the top collaborators of Donna P. Frazier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Donna P. Frazier. Donna P. Frazier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Inoue, Kazushi, Elizabeth A. Fry, & Donna P. Frazier. (2015). Transcription factors that interact with p53 and Mdm2. International Journal of Cancer. 138(7). 1577–1585. 88 indexed citations
2.
Frazier, Donna P., Robert D. Kendig, Fumitake Kai, et al.. (2012). Dmp1 Physically Interacts with p53 and Positively Regulates p53′s Stability, Nuclear Localization, and Function. Cancer Research. 72(7). 1740–1750. 34 indexed citations
3.
Tang, Shuai, Meenu Mishra, Donna P. Frazier, et al.. (2012). Positive and Negative Regulation of Prostate Stem Cell Antigen Expression by Yin Yang 1 in Prostate Epithelial Cell Lines. PLoS ONE. 7(4). e35570–e35570. 9 indexed citations
4.
Taneja, Pankaj, Dejan Maglic, Fumitake Kai, et al.. (2010). Critical Roles of DMP1 in Human Epidermal Growth Factor Receptor 2/neu-Arf-p53 Signaling and Breast Cancer Development. Cancer Research. 70(22). 9084–9094. 33 indexed citations
5.
Taneja, Pankaj, Donna P. Frazier, Robert D. Kendig, et al.. (2009). MMTV mouse models and the diagnostic values of MMTV-like sequences in human breast cancer. Expert Review of Molecular Diagnostics. 9(5). 423–440. 56 indexed citations
6.
Mallakin, Ali, Takayuki Sugiyama, Fumitake Kai, et al.. (2009). The Arf‐inducing transcription factor Dmp1 encodes a transcriptional activator of amphiregulin, thrombospondin‐1, JunB and Egr1. International Journal of Cancer. 126(6). 1403–1416. 33 indexed citations
7.
Sugiyama, Takayuki, Donna P. Frazier, Pankaj Taneja, et al.. (2008). Role of DMP1 and its future in lung cancer diagnostics. Expert Review of Molecular Diagnostics. 8(4). 435–447. 22 indexed citations
8.
Sugiyama, Takayuki, Donna P. Frazier, Pankaj Taneja, et al.. (2008). Signal Transduction Involving the Dmp1 Transcription Factor and its Alteration in Human Cancer. SHILAP Revista de lepidopterología. 2. 209–19. 12 indexed citations
9.
Inoue, Kazushi, Takayuki Sugiyama, Pankaj Taneja, Rachel L. Morgan, & Donna P. Frazier. (2008). Emerging Roles of DMP1 in Lung Cancer. Cancer Research. 68(12). 4487–4490. 17 indexed citations
10.
Inoue, Kazushi, Ali Mallakin, & Donna P. Frazier. (2007). Dmp1 and tumor suppression. Oncogene. 26(30). 4329–4335. 57 indexed citations
11.
Frazier, Donna P., Amber Wilson, Christopher J. Dougherty, et al.. (2007). PKC-α and TAK-1 are intermediates in the activation ofc-Jun NH2-terminal kinase by hypoxia-reoxygenation. American Journal of Physiology-Heart and Circulatory Physiology. 292(4). H1675–H1684. 27 indexed citations
12.
Mallakin, Ali, Takayuki Sugiyama, Pankaj Taneja, et al.. (2007). Mutually Exclusive Inactivation of DMP1 and ARF/p53 in Lung Cancer. Cancer Cell. 12(4). 381–394. 62 indexed citations
13.
Taneja, Pankaj, et al.. (2007). Repression of Dmp1 and Arf transcription by anthracyclins: critical roles of the NF-κB subunit p65. Oncogene. 26(53). 7457–7466. 32 indexed citations
14.
Webster, Keith A., Regina M. Graham, John W. Thompson, et al.. (2006). Redox Stress and the Contributions of BH3-Only Proteins to Infarction. Antioxidants and Redox Signaling. 8(9-10). 1667–1676. 55 indexed citations
15.
Frazier, Donna P., Amber Wilson, Regina M. Graham, et al.. (2006). Acidosis Regulates the Stability, Hydrophobicity, and Activity of the BH3-Only Protein Bnip3. Antioxidants and Redox Signaling. 8(9-10). 1625–1634. 34 indexed citations
16.
Dougherty, Christopher J., et al.. (2004). Mitochondrial signals initiate the activation of c‐Jun N ‐terminal kinase (JNK) by hypoxia‐reoxygenation. The FASEB Journal. 18(10). 1060–1070. 84 indexed citations
17.
Graham, Regina M., Donna P. Frazier, John W. Thompson, et al.. (2004). A unique pathway of cardiac myocyte death caused by hypoxia–acidosis. Journal of Experimental Biology. 207(18). 3189–3200. 159 indexed citations
18.
19.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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