Dong‐In Koh

411 total citations
25 papers, 290 citations indexed

About

Dong‐In Koh is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, Dong‐In Koh has authored 25 papers receiving a total of 290 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 15 papers in Oncology and 4 papers in Pathology and Forensic Medicine. Recurrent topics in Dong‐In Koh's work include Cancer-related Molecular Pathways (9 papers), DNA Repair Mechanisms (4 papers) and Cancer-related gene regulation (4 papers). Dong‐In Koh is often cited by papers focused on Cancer-related Molecular Pathways (9 papers), DNA Repair Mechanisms (4 papers) and Cancer-related gene regulation (4 papers). Dong‐In Koh collaborates with scholars based in South Korea, Japan and United States. Dong‐In Koh's co-authors include Man‐Wook Hur, Bu‐Nam Jeon, Won‐Il Choi, Kyung‐Sup Kim, Minyoung Kim, Seo‐Hyun Choi, Chae‐Ok Yun, S. Ahn, Jaeku Kang and Jaechun Lee and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Oncogene.

In The Last Decade

Dong‐In Koh

24 papers receiving 288 citations

Peers

Dong‐In Koh

ONCOL

GENET

IMMUN

Yuxia Yang China

Majdina Isovic Canada

Pavana Anur United States

Asuka Hira Japan

Nicole Kiweler Germany

Jia Bian China

Ahlima Roumane United Kingdom

Peter J. Mullen Canada

Katie Holmes United States

Yuxia Yang China

Dong‐In Koh

225 ×1.0

102 ×1.6

37 ×0.8

ONCOL

31 ×1.1

GENET

28 ×1.2

IMMUN

Citations per year, relative to Dong‐In Koh Dong‐In Koh (= 1×) peers Yuxia Yang

Countries citing papers authored by Dong‐In Koh

Since Specialization

Citations

This map shows the geographic impact of Dong‐In Koh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dong‐In Koh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dong‐In Koh more than expected).

Fields of papers citing papers by Dong‐In Koh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dong‐In Koh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dong‐In Koh. The network helps show where Dong‐In Koh may publish in the future.

Co-authorship network of co-authors of Dong‐In Koh

This figure shows the co-authorship network connecting the top 25 collaborators of Dong‐In Koh. A scholar is included among the top collaborators of Dong‐In Koh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dong‐In Koh. Dong‐In Koh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kim, Do Yeon, et al.. (2025). TP53 and DNA-PK as potential biomarkers for enhanced efficacy of Olaparib in colorectal cancer. Investigational New Drugs. 43(3). 656–668. 1 indexed citations

Koh, Dong‐In, Minki Lee, Joseph Kim, et al.. (2024). The Immune Suppressor IGSF1 as a Potential Target for Cancer Immunotherapy. Cancer Immunology Research. 12(4). 491–507. 2 indexed citations

Kim, Do Yeon, et al.. (2024). Degradation of AZGP1 suppresses apoptosis and facilitates cholangiocarcinoma tumorigenesis via TRIM25. Journal of Cellular and Molecular Medicine. 28(3). e18104–e18104. 5 indexed citations

Ryu, Yea Seong, Yu Jin Shin, Jeong Hee Kim, et al.. (2023). Mutant PIK3CA as a negative predictive biomarker for treatment with a highly selective PIM1 inhibitor in human colon cancer. Cancer Biology & Therapy. 24(1). 2246208–2246208. 3 indexed citations

Hong, Jun Ki, et al.. (2023). TCOF1 promotes the colorectal cancer progression by stabilizing β-catenin. Medical Oncology. 40(12). 1 indexed citations

Kim, Do Yeon, Yea Seong Ryu, Eunsil Lee, et al.. (2022). DGG-300273, a novel WNT/β-catenin inhibitor, induces apoptotic cell death by activating ROS-BIM signaling in a Wnt-dependent manner in colon cancer cells. Investigational New Drugs. 41(1). 105–114. 4 indexed citations

Park, Sang Soo, Yea Seong Ryu, Dong‐In Koh, et al.. (2021). Mutation SVCT2 promotes cell proliferation, invasion and migration in colorectal cancer. Journal of Cancer. 12(18). 5385–5393. 2 indexed citations

Kim, Eun Ho, Dong‐In Koh, Yea Seong Ryu, et al.. (2021). Role of p53 in transcriptional repression of SVCT2. Molecular Biology Reports. 48(2). 1651–1658. 4 indexed citations

Choi, Seo‐Hyun, et al.. (2019). Hypoxia-induced RelA/p65 derepresses SLC16A3 (MCT4) by downregulating ZBTB7A. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1862(8). 771–785. 30 indexed citations

10.

Choi, Won‐Il, Bu‐Nam Jeon, Dong‐In Koh, et al.. (2019). Zbtb7c is a critical gluconeogenic transcription factor that induces glucose-6-phosphatase and phosphoenylpyruvate carboxykinase 1 genes expression during mice fasting. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1862(6). 643–656. 14 indexed citations

11.

Koh, Dong‐In, Jin Young Kim, Masahiko Hatano, et al.. (2018). Reciprocal negative regulation between the tumor suppressor protein p53 and B cell CLL/lymphoma 6 (BCL6) via control of caspase-1 expression. Journal of Biological Chemistry. 294(1). 299–313. 12 indexed citations

12.

Koh, Dong‐In, et al.. (2016). Role of MIZ-1 in AMELX gene expression. Biochemistry and Biophysics Reports. 8. 340–345. 2 indexed citations

13.

Jeon, Bu‐Nam, Min-Kyeong Kim, Won‐Il Choi, et al.. (2016). Zbtb7c is a molecular ‘off’ and ‘on’ switch of Mmp gene transcription. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1859(11). 1429–1439. 9 indexed citations

14.

Koh, Dong‐In, et al.. (2015). Transcriptional activation of APAF1 by KAISO (ZBTB33) and p53 is attenuated by RelA/p65. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1849(9). 1170–1178. 13 indexed citations

15.

Jeon, Bu‐Nam, Minkyeong Kim, Min‐Young Kim, et al.. (2014). Two ZNF509 (ZBTB49) isoforms induce cell-cycle arrest by activating transcription of p21/CDKN1A and RB upon exposure to genotoxic stress. Nucleic Acids Research. 42(18). 11447–11461. 18 indexed citations

16.

Choi, Won‐Il, Bu‐Nam Jeon, Dong‐In Koh, et al.. (2014). Human Krüppel-related 3 (HKR3) Is a Novel Transcription Activator of Alternate Reading Frame (ARF) Gene. Journal of Biological Chemistry. 289(7). 4018–4031. 8 indexed citations

17.

Koh, Dong‐In, et al.. (2013). Kaiso is a key regulator of spleen germinal center formation by repressing Bcl6 expression in splenocytes. Biochemical and Biophysical Research Communications. 442(3-4). 177–182. 7 indexed citations

18.

Jeon, Bu‐Nam, et al.. (2013). Regulation of the Cyclin-dependent Kinase Inhibitor 1A Gene (CDKN1A) by the Repressor BOZF1 through Inhibition of p53 Acetylation and Transcription Factor Sp1 Binding. Journal of Biological Chemistry. 288(10). 7053–7064. 14 indexed citations

19.

Jeon, Bu‐Nam, Kim Ys, Won‐Il Choi, et al.. (2012). Kr-pok increases FASN expression by modulating the DNA binding of SREBP-1c and Sp1 at the proximal promoter. Journal of Lipid Research. 53(4). 755–766. 29 indexed citations

20.

Koh, Dong‐In, Won‐Il Choi, Bu‐Nam Jeon, et al.. (2009). A Novel POK Family Transcription Factor, ZBTB5, Represses Transcription of p21CIP1 Gene. Journal of Biological Chemistry. 284(30). 19856–19866. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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