Dong Kuk Ahn

2.6k total citations
109 papers, 2.2k citations indexed

About

Dong Kuk Ahn is a scholar working on Physiology, Cellular and Molecular Neuroscience and Sensory Systems. According to data from OpenAlex, Dong Kuk Ahn has authored 109 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 79 papers in Physiology, 65 papers in Cellular and Molecular Neuroscience and 22 papers in Sensory Systems. Recurrent topics in Dong Kuk Ahn's work include Pain Mechanisms and Treatments (74 papers), Neuropeptides and Animal Physiology (34 papers) and Neuroscience and Neuropharmacology Research (23 papers). Dong Kuk Ahn is often cited by papers focused on Pain Mechanisms and Treatments (74 papers), Neuropeptides and Animal Physiology (34 papers) and Neuroscience and Neuropharmacology Research (23 papers). Dong Kuk Ahn collaborates with scholars based in South Korea, Japan and United States. Dong Kuk Ahn's co-authors include Yong Chul Bae, Jin Sook Ju, Seung Ro Han, Sang Kyoo Paik, Yun‐Sook Kim, Seog Bae Oh, Sung Jun Jung, Hyo Sun Choi, Yong Chul Bae and Jin Young Bae and has published in prestigious journals such as PLoS ONE, The Journal of Comparative Neurology and Scientific Reports.

In The Last Decade

Dong Kuk Ahn

106 papers receiving 2.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Dong Kuk Ahn South Korea 30 1.3k 893 516 408 232 109 2.2k
Andrew J. Mannes United States 32 1.5k 1.2× 770 0.9× 618 1.2× 650 1.6× 200 0.9× 82 2.8k
Sachia G. Khasar United States 29 1.7k 1.3× 879 1.0× 822 1.6× 447 1.1× 189 0.8× 41 2.7k
Cláudia Herrera Tambeli Brazil 30 1.6k 1.3× 984 1.1× 477 0.9× 244 0.6× 269 1.2× 103 2.7k
Robert M. Caudle United States 33 1.5k 1.2× 1.2k 1.3× 811 1.6× 483 1.2× 167 0.7× 89 2.8k
Heung Sik Na South Korea 26 1.4k 1.1× 726 0.8× 686 1.3× 294 0.7× 258 1.1× 60 2.4k
Niels Eijkelkamp Netherlands 28 1.4k 1.1× 800 0.9× 1.1k 2.2× 299 0.7× 158 0.7× 80 2.9k
Kata Bölcskei Hungary 28 915 0.7× 567 0.6× 430 0.8× 694 1.7× 212 0.9× 64 2.0k
Alexander M. Binshtok Israel 24 1.3k 1.0× 829 0.9× 846 1.6× 620 1.5× 124 0.5× 44 2.8k
Olayinka A. Dina United States 29 1.8k 1.4× 892 1.0× 740 1.4× 881 2.2× 180 0.8× 32 2.9k

Countries citing papers authored by Dong Kuk Ahn

Since Specialization
Citations

This map shows the geographic impact of Dong Kuk Ahn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dong Kuk Ahn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dong Kuk Ahn more than expected).

Fields of papers citing papers by Dong Kuk Ahn

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dong Kuk Ahn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dong Kuk Ahn. The network helps show where Dong Kuk Ahn may publish in the future.

Co-authorship network of co-authors of Dong Kuk Ahn

This figure shows the co-authorship network connecting the top 25 collaborators of Dong Kuk Ahn. A scholar is included among the top collaborators of Dong Kuk Ahn based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dong Kuk Ahn. Dong Kuk Ahn is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Jung, Sung Jun, Yu‐Mi Kim, Minjeong Jo, et al.. (2025). Can Botulinum Toxin Type E Serve as a Novel Therapeutic Target for Managing Chronic Orofacial Pain?. Toxins. 17(3). 130–130. 1 indexed citations
3.
Jo, Minjeong, et al.. (2024). Blockade of Piezo2 Pathway Attenuates Inflammatory and Neuropathic Pain in the Orofacial Area. Pain Research and Management. 2024(1). 9179928–9179928. 3 indexed citations
4.
5.
Park, Jae‐Hyung, et al.. (2020). Mannitol Enhances the Antinociceptive Effects of Diphenhydramine as an Alternative Local Anesthetic. Pain Research and Management. 2020. 1–6. 4 indexed citations
6.
7.
Yang, Kun, et al.. (2016). Antinociceptive Effects of Botulinum Toxin Type A on Trigeminal Neuropathic Pain. Journal of Dental Research. 95(10). 1183–1190. 36 indexed citations
8.
Han, Seung Ro, et al.. (2012). A novel trigeminal neuropathic pain model: Compression of the trigeminal nerve root produces prolonged nociception in rats. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 38(2). 149–158. 36 indexed citations
9.
Ahn, Dong Kuk, et al.. (2011). Participation of Peripheral P2X Receptors in Orofacial Inflammatory Nociception in Rats. 36(3). 143–148. 2 indexed citations
10.
Park, Sang H., et al.. (2011). Intracisternal Administration of Voltage Dependent Calcium Channel Blockers Attenuates Orofacial Inflammatory Nociceptive Behavior in Rats. 36(2). 43–50. 1 indexed citations
11.
Park, Yoon-Yub, et al.. (2010). The Intracisternal Administration of MEK Inhibitor Attenuates Mechanical and Cold Allodynia in a Rat Model of Compression of the Trigeminal Ganglion. 35(3). 75–81.
12.
Cho, Yi Sul, Sang Kyoo Paik, Atsushi Yoshida, et al.. (2010). Ultrastructural analysis of the synaptic connectivity of TRPV1‐expressing primary afferent terminals in the rat trigeminal caudal nucleus. The Journal of Comparative Neurology. 518(20). 4134–4146. 22 indexed citations
13.
Han, Seong Kyu, et al.. (2009). Functional type I vanilloid receptor expression by substantia gelatinosa neurons of trigeminal subnucleus caudalis in mice. Neuroscience Letters. 452(3). 228–231. 5 indexed citations
14.
Ahn, Dong Kuk, Sang Yup Lee, Seung Ro Han, et al.. (2009). Intratrigeminal ganglionic injection of LPA causes neuropathic pain-like behavior and demyelination in rats. Pain. 146(1). 114–120. 59 indexed citations
15.
Moon, Yong Suk, Sang Kyoo Paik, Masayuki Moritani, et al.. (2007). GABA- and glycine-like immunoreactivity in axonal endings presynaptic to the vibrissa afferents in the cat trigeminal interpolar nucleus. Neuroscience. 152(1). 138–145. 14 indexed citations
16.
Choi, Hyo Sun, Jin Sook Ju, Hyo‐Sang Park, et al.. (2006). Central Metabotropic Glutamate Receptors Differentially Participate in Interleukin-1β–Induced Mechanical Allodynia in the Orofacial Area of Conscious Rats. Journal of Pain. 7(10). 747–756. 19 indexed citations
17.
Jung, Sung‐Chul, et al.. (2005). Intra-articular Injection of $IL-1{\beta}$ Facilitated Formalin-induced Temporomandibular Joint Pain in Freely Moving Rats. Korean Journal of Physiology and Pharmacology. 9(1). 23–27. 5 indexed citations
18.
Lee, Ho Jeong, et al.. (2004). Intracisternal NMDA produces analgesia in the orofacial formalin test of freely moving rats. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 28(3). 497–503. 7 indexed citations
19.
Ahn, Dong Kuk, Yun‐Sook Kim, & Jaesik Park. (1998). Microinjection of Glutamate into the Amygdala Modulates Nociceptive and Cardiovascular Response in Freely Moving Rats. Korean Journal of Physiology and Pharmacology. 2(6). 687–693. 1 indexed citations
20.
Ahn, Dong Kuk, et al.. (1997). Cardiovascular Effects of Endogenous GABA in the Nucleus Tractus Solitarius. Sunhwan'gi. 27(1). 94–94. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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