Donata Simińska
- Molecular Biology
- Immunology top 10%
- Oncology top 10%
- Cancer Research top 10%
- Genetics top 5%
- Co-authors
- Irena Baranowska‐BosiackaDariusz ChlubekJan KorbeckiKlaudyna KojderIzabela GutowskaSzymon GrochansAnna Maria CybulskaRomuald Bohatyrewicz
- Topics
- Cancer, Hypoxia, and Metabolism (6 papers)Glioma Diagnosis and Treatment (6 papers)Chemokine receptors and signaling (5 papers)
- Cited by
- GeneticsCancer ResearchImmunology
- Partner nations
- Poland
In The Last Decade
Donata Simińska
18 papers receiving 1.2k citations
Hit Papers
Peers
Comparison fields: 5 of 113
- Molecular Biology 439
- Immunology 385
- Oncology 345
- Cancer Research 281
- Genetics 220
Countries citing papers authored by Donata Simińska
This map shows the geographic impact of Donata Simińska's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Donata Simińska with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Donata Simińska more than expected).
Fields of papers citing papers by Donata Simińska
This network shows the impact of papers produced by Donata Simińska. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Donata Simińska. The network helps show where Donata Simińska may publish in the future.
Co-authorship network of co-authors of Donata Simińska
This figure shows the co-authorship network connecting the top 25 collaborators of Donata Simińska. A scholar is included among the top collaborators of Donata Simińska based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Donata Simińska. Donata Simińska is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 3 | |
| 4 | 3 | |
| 5 | Epidemiology of Glioblastoma Multiforme–Literature Reviewbreakdown → | 329 |
| 6 | 8 | |
| 7 | 7 | |
| 8 | 4 | |
| 9 | Chronic and Cycling Hypoxia: Drivers of Cancer Chronic Inflammation through HIF-1 and NF-κB Activation: A Review of the Molecular Mechanismsbreakdown → | 251 |
| 10 | 18 | |
| 11 | 107 | |
| 12 | 295 | |
| 13 | 16 | |
| 14 | 68 | |
| 15 | 2 | |
| 16 | 13 | |
| 17 | 79 | |
| 18 | 28 | |
| 19 | 13 |
About Donata Simińska
Donata Simińska is a scholar working on Genetics, Cancer Research and Immunology, having authored 19 papers that have together received 1.2k indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (6 papers), Glioma Diagnosis and Treatment (6 papers) and Chemokine receptors and signaling (5 papers). The work is most often cited by research in Genetics (220 citations), Cancer Research (281 citations) and Immunology (385 citations). Donata Simińska has collaborated with scholars based in Poland. Frequent co-authors include Irena Baranowska‐Bosiacka, Dariusz Chlubek, Jan Korbecki, Klaudyna Kojder, Izabela Gutowska, Szymon Grochans, Anna Maria Cybulska, Romuald Bohatyrewicz, Joanna Listos and Magdalena Gąssowska-Dobrowolska. Their work appears in journals such as International Journal of Molecular Sciences, Nutrients and Cancers.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.