Dipankar Bhandari

592 total citations
11 papers, 401 citations indexed

About

Dipankar Bhandari is a scholar working on Molecular Biology, Immunology and Epidemiology. According to data from OpenAlex, Dipankar Bhandari has authored 11 papers receiving a total of 401 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Immunology and 3 papers in Epidemiology. Recurrent topics in Dipankar Bhandari's work include RNA and protein synthesis mechanisms (5 papers), RNA Research and Splicing (5 papers) and Research on Leishmaniasis Studies (3 papers). Dipankar Bhandari is often cited by papers focused on RNA and protein synthesis mechanisms (5 papers), RNA Research and Splicing (5 papers) and Research on Leishmaniasis Studies (3 papers). Dipankar Bhandari collaborates with scholars based in Germany, India and United States. Dipankar Bhandari's co-authors include Elisa Izaurralde, Oliver Weichenrieder, Tobias Raisch, Stefanie Jonas, Duygu Kuzuoğlu‐Öztürk, Sigrun Helms, Eric Huntzinger, Ying Chen, Eugene Valkov and Cátia Igreja and has published in prestigious journals such as Nucleic Acids Research, Nature Communications and Genes & Development.

In The Last Decade

Dipankar Bhandari

11 papers receiving 401 citations

Peers

Countries citing papers authored by Dipankar Bhandari

Since Specialization

Citations

This map shows the geographic impact of Dipankar Bhandari's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dipankar Bhandari with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dipankar Bhandari more than expected).

Fields of papers citing papers by Dipankar Bhandari

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dipankar Bhandari. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dipankar Bhandari. The network helps show where Dipankar Bhandari may publish in the future.

Co-authorship network of co-authors of Dipankar Bhandari

This figure shows the co-authorship network connecting the top 25 collaborators of Dipankar Bhandari. A scholar is included among the top collaborators of Dipankar Bhandari based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dipankar Bhandari. Dipankar Bhandari is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

#	Work	Indexed citations
1	HELZ directly interacts with CCR4–NOT and causes decay of bound mRNAs 2019 ·Life Science Alliance ·(unknown), (unknown), Ramona Weber, (unknown), (unknown), Tobias Raisch, Duygu Kuzuoğlu‐Öztürk, Chung-Te Chang, Dipankar Bhandari, Cátia Igreja, Lara Wohlbold	15
2	A conserved CAF40-binding motif in metazoan NOT4 mediates association with the CCR4–NOT complex 2019 ·Genes & Development ·(unknown), Tobias Raisch, (unknown), Cátia Igreja, Dipankar Bhandari, Oliver Weichenrieder, Elisa Izaurralde	30
3	A low-complexity region in human XRN1 directly recruits deadenylation and decapping factors in 5′–3′ messenger RNA decay 2019 ·Nucleic Acids Research ·Chung-Te Chang, Sowndarya Muthukumar, Ramona Weber, Yevgen Levdansky, Ying Chen, Dipankar Bhandari, Cátia Igreja, Lara Wohlbold, Eugene Valkov, Elisa Izaurralde	32
4	A CAF40-binding motif facilitates recruitment of the CCR4-NOT complex to mRNAs targeted by Drosophila Roquin 2017 ·Nature Communications ·(unknown), Tobias Raisch, Praveen Bawankar, Dipankar Bhandari, Ying Chen, Duygu Kuzuoğlu‐Öztürk, Oliver Weichenrieder, Elisa Izaurralde	65
5	Distinct modes of recruitment of the CCR 4– NOT complex by Drosophila and vertebrate Nanos 2016 ·The EMBO Journal ·Tobias Raisch, Dipankar Bhandari, (unknown), Sigrun Helms, Eugene Valkov, Oliver Weichenrieder, Elisa Izaurralde	49
6	mi RISC and the CCR 4– NOT complex silence mRNA targets independently of 43S ribosomal scanning 2016 ·The EMBO Journal ·Duygu Kuzuoğlu‐Öztürk, Dipankar Bhandari, Eric Huntzinger, (unknown), Sigrun Helms, Elisa Izaurralde	62
7	An asymmetric PAN3 dimer recruits a single PAN2 exonuclease to mediate mRNA deadenylation and decay 2014 ·Nature Structural & Molecular Biology ·Stefanie Jonas, Mary Christie, Daniel Peter, Dipankar Bhandari, Belinda Loh, Eric Huntzinger, Oliver Weichenrieder, Elisa Izaurralde	38
8	Ubiquitination-mediated interaction among domains is responsible for inhibition of RNA endonuclease activity of mRNA cycling sequence binding protein from L. donovani (LdCSBP) 2014 ·Parasitology Research ·(unknown), Dipankar Bhandari, (unknown), Partha Saha	1
9	Structural basis for the Nanos-mediated recruitment of the CCR4–NOT complex and translational repression 2014 ·Genes & Development ·Dipankar Bhandari, Tobias Raisch, Oliver Weichenrieder, Stefanie Jonas, Elisa Izaurralde	89
10	Ubiquitination of mRNA cycling sequence binding protein from Leishmania donovani (LdCSBP) modulates the RNA endonuclease activity of its Smr domain 2011 ·FEBS Letters ·Dipankar Bhandari, (unknown), (unknown), Partha Saha	15
11	mRNA cycling sequence binding protein fromLeishmania donovani(LdCSBP) is covalently modified by ubiquitination 2007 ·FEMS Microbiology Letters ·Dipankar Bhandari, Partha Saha	5

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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