Dinko Berkovic
- Immunology top 10%
- Molecular Biology top 10%
- Protein Kinase Regulation and GTPase Signaling 6
- Sphingolipid Metabolism and Signaling 4
- PI3K/AKT/mTOR signaling in cancer 3
- Retinoids in leukemia and cellular processes 3
- Lipid Membrane Structure and Behavior 2
- Cancer Research top 10%
- Physiology top 10%
- Cell Biology top 10%
- Cellular transport and secretion 2
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- Drug Transport and Resistance Mechanisms 5
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- Organophosphorus compounds synthesis 4
Dinko Berkovic
27 papers receiving 1.6k citations
Hit Papers
Peers
Comparison fields: 5 of 99
- Immunology 397
- Molecular Biology 1.0k
- Cancer Research 223
- Physiology 50
- Cell Biology 146
Countries citing papers authored by Dinko Berkovic
This map shows the geographic impact of Dinko Berkovic's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dinko Berkovic with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dinko Berkovic more than expected).
Fields of papers citing papers by Dinko Berkovic
This network shows the impact of papers produced by Dinko Berkovic. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dinko Berkovic. The network helps show where Dinko Berkovic may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Dinko Berkovic, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 0 | |
| 2 | 2015 | 3 | |
| 3 | 2006 | 40 | |
| 4 | 2004 | 1 | |
| 5 | 2003 | 13 | |
| 6 | 2003 | 38 | |
| 7 | 2002 | 8 | |
| 8 | 2002 | 7 | |
| 9 | 2001 | 8 | |
| 10 | 1998 | 54 | |
| 11 | 1997 | 2 | |
| 12 | 1997 | 13 | |
| 13 | 1997 | 25 | |
| 14 | Induction of resistance in the human leukemia cell line HL60 towards hexadecylphosphocholine and other ether phospholipid analogues. | 1996 | 11 |
| 15 | 1996 | 16 | |
| 16 | 1995 | 24 | |
| 17 | 1995 | 34 | |
| 18 | TNF activates NF-κB by phosphatidylcholine-specific phospholipase C-induced “Acidic” sphingomyelin breakdownbreakdown → | 1992 | 949 |
| 19 | 1991 | 169 | |
| 20 | 1990 | 41 |
About Dinko Berkovic
Dinko Berkovic is a scholar working on Oncology, Toxicology and Biochemistry, having authored 28 papers that have together received 1.6k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (6 papers), Drug Transport and Resistance Mechanisms (5 papers), Organophosphorus compounds synthesis (4 papers), Sphingolipid Metabolism and Signaling (4 papers), PI3K/AKT/mTOR signaling in cancer (3 papers), Retinoids in leukemia and cellular processes (3 papers), Lipid Membrane Structure and Behavior (2 papers) and Cellular transport and secretion (2 papers). The work is most often cited by research in Immunology (397 citations), Molecular Biology (1.0k citations) and Cancer Research (223 citations). Dinko Berkovic has collaborated with scholars based in Germany and Estonia. Frequent co-authors include Stefan Schütze, Martin Krönke, Karin Potthoff, Katja Wiegmann, Thomas Machleidt, Clemens Unger, E. A. M. Fleer, Claudia R. Binder, Lutz Binder and Lorenz Trümper. Their work appears in journals such as Cell, The Journal of Experimental Medicine and Clinical Infectious Diseases.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.