Ding Cao

472 total citations
32 papers, 368 citations indexed

About

Ding Cao is a scholar working on Immunology, Surgery and Molecular Biology. According to data from OpenAlex, Ding Cao has authored 32 papers receiving a total of 368 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Immunology, 9 papers in Surgery and 8 papers in Molecular Biology. Recurrent topics in Ding Cao's work include Immune Response and Inflammation (11 papers), Organ Transplantation Techniques and Outcomes (8 papers) and Antimicrobial Peptides and Activities (4 papers). Ding Cao is often cited by papers focused on Immune Response and Inflammation (11 papers), Organ Transplantation Techniques and Outcomes (8 papers) and Antimicrobial Peptides and Activities (4 papers). Ding Cao collaborates with scholars based in China, Germany and Australia. Ding Cao's co-authors include Jianping Gong, Jinzheng Li, Linghua Zhang, Menghao Wang, Yang Xu, Xiumei Liang, Huazhou Li, Junhua Gong, Bing Tu and Yong Chen and has published in prestigious journals such as Annals of Oncology, Applied Microbiology and Biotechnology and Biochemical Pharmacology.

In The Last Decade

Ding Cao

31 papers receiving 366 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ding Cao China 13 127 115 56 56 43 32 368
Yuhua Zhao China 12 137 1.1× 92 0.8× 60 1.1× 69 1.2× 60 1.4× 17 410
Fang Cui China 16 174 1.4× 75 0.7× 42 0.8× 173 3.1× 93 2.2× 44 626
Bing Thio Netherlands 11 89 0.7× 174 1.5× 29 0.5× 87 1.6× 15 0.3× 24 506
Dani Nazzal France 10 98 0.8× 136 1.2× 24 0.4× 63 1.1× 14 0.3× 11 433
Maísa Mota Antunes Brazil 14 130 1.0× 139 1.2× 38 0.7× 86 1.5× 26 0.6× 31 479
Jiaxin Zhang China 11 193 1.5× 61 0.5× 22 0.4× 103 1.8× 33 0.8× 40 461
Hyo Jung Nam South Korea 12 190 1.5× 118 1.0× 29 0.5× 63 1.1× 39 0.9× 22 488
Caifeng Chen China 11 149 1.2× 113 1.0× 16 0.3× 46 0.8× 41 1.0× 24 328
Shamsher S. Saini United States 14 111 0.9× 62 0.5× 27 0.5× 25 0.4× 23 0.5× 25 453

Countries citing papers authored by Ding Cao

Since Specialization
Citations

This map shows the geographic impact of Ding Cao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ding Cao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ding Cao more than expected).

Fields of papers citing papers by Ding Cao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ding Cao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ding Cao. The network helps show where Ding Cao may publish in the future.

Co-authorship network of co-authors of Ding Cao

This figure shows the co-authorship network connecting the top 25 collaborators of Ding Cao. A scholar is included among the top collaborators of Ding Cao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ding Cao. Ding Cao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cao, Ding, et al.. (2025). Early prediction and risk stratification of ovarian cancer based on clinical data using machine learning approaches. Journal of Gynecologic Oncology. 36(4). e53–e53. 1 indexed citations
2.
Ma, Junjie, Ding Cao, Qianhua Zhao, et al.. (2025). 2113P Phase I/II study of claudin 18.2 ADC ATG-022 in patients with advanced gastric/gastroesophageal junction cancer (CLINCH). Annals of Oncology. 36. S1111–S1112.
3.
Cao, Ding, Ekkehard Grünig, Yu.M. Sіrenko, et al.. (2024). SMAD5 as a novel gene for familial pulmonary arterial hypertension. Clinical Science. 139(1). 15–27. 2 indexed citations
4.
Zhao, Zitong, Qianyi Liang, Zhiyang Chen, et al.. (2021). Overexpression of pEGF improved the gut protective function of Clostridium butyricum partly through STAT3 signal pathway. Applied Microbiology and Biotechnology. 105(14-15). 5973–5991. 14 indexed citations
5.
Wu, Hao, et al.. (2020). IL‐4 Alleviates Ischaemia‐Reperfusion Injury by Inducing Kupffer Cells M2 Polarization via STAT6‐JMJD3 Pathway after Rat Liver Transplantation. BioMed Research International. 2020(1). 2953068–2953068. 12 indexed citations
6.
Zhang, Weikang, et al.. (2020). XBP1s repression regulates Kupffer cell polarization leading to immune suppressive effects protecting liver allograft in rats. International Immunopharmacology. 91. 107294–107294. 9 indexed citations
7.
Li, Jiayi, Ding Cao, Min Zeng, et al.. (2020). Protective effects of Clostridium butyricum against oxidative stress induced by food processing and lipid-derived aldehydes in Caco-2 cells. Applied Microbiology and Biotechnology. 104(21). 9343–9361. 13 indexed citations
8.
Cao, Ding, et al.. (2019). α-ketoglutarate attenuates ischemia-reperfusion injury of liver graft in rats. Biomedicine & Pharmacotherapy. 111. 1141–1146. 24 indexed citations
9.
Li, Jinzheng, et al.. (2019). VEGF-C attenuates ischemia reperfusion injury of liver graft in rats. Transplant Immunology. 54. 59–64. 8 indexed citations
10.
Xu, Xuesong, Menghao Wang, Jinzheng Li, et al.. (2018). Tauroursodeoxycholic acid alleviates hepatic ischemia reperfusion injury by suppressing the function of Kupffer cells in mice. Biomedicine & Pharmacotherapy. 106. 1271–1281. 14 indexed citations
11.
Cao, Ding, Zuojin Liu, Jin‐Heng Li, et al.. (2018). Dysfunctional immunoregulation in human liver allograft rejection associated with compromised galectin-1/CD7 pathway function. Cell Death and Disease. 9(3). 293–293. 6 indexed citations
12.
Zhang, Wenfeng, et al.. (2017). Nobiletin ameliorates ischemia–reperfusion injury by suppressing the function of Kupffer cells after liver transplantation in rats. Biomedicine & Pharmacotherapy. 89. 732–741. 25 indexed citations
13.
Gong, Junhua, Ding Cao, Yong Chen, et al.. (2017). Role of programmed death ligand 1 and Kupffer cell in immune regulation after orthotopic liver transplantation in rats. International Immunopharmacology. 48. 8–16. 14 indexed citations
14.
15.
Cao, Ding, et al.. (2016). Isoflurane: An Ideal Anesthetic for Rodent Orthotopic Liver Transplantation Surgery?. Transplantation Proceedings. 48(8). 2815–2820. 1 indexed citations
16.
Jiang, Xiaowei, Kunlun He, Peizhi Li, et al.. (2016). Bone marrow stromal cells attenuate LPS-induced mouse acute liver injury via the prostaglandin E 2-dependent repression of the NLRP3 inflammasome in Kupffer cells. Immunology Letters. 179. 102–113. 31 indexed citations
17.
Yang, Jun, Shuiqing Zhang, Huazhou Li, et al.. (2012). Innate defense regulator peptide synergizes with CpG ODN for enhanced innate intestinal immune responses in neonate piglets. International Immunopharmacology. 12(2). 415–424. 12 indexed citations
18.
19.
Cao, Ding, et al.. (2010). Synthetic innate defence regulator peptide enhances in vivo immunostimulatory effects of CpG-ODN in newborn piglets. Vaccine. 28(37). 6006–6013. 20 indexed citations
20.
Cheng, Qing, et al.. (2010). CpG oligodeoxynucleotide promotes protective immunity in the enteric mucosa and suppresses enterotoxigenic E. coli in the weaning piglets. International Immunopharmacology. 10(10). 1249–1260. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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