Dietbert Neumann

10.4k total citations · 1 hit paper
83 papers, 6.0k citations indexed

About

Dietbert Neumann is a scholar working on Molecular Biology, Surgery and Physiology. According to data from OpenAlex, Dietbert Neumann has authored 83 papers receiving a total of 6.0k indexed citations (citations by other indexed papers that have themselves been cited), including 74 papers in Molecular Biology, 41 papers in Surgery and 7 papers in Physiology. Recurrent topics in Dietbert Neumann's work include Metabolism, Diabetes, and Cancer (51 papers), Pancreatic function and diabetes (41 papers) and ATP Synthase and ATPases Research (12 papers). Dietbert Neumann is often cited by papers focused on Metabolism, Diabetes, and Cancer (51 papers), Pancreatic function and diabetes (41 papers) and ATP Synthase and ATPases Research (12 papers). Dietbert Neumann collaborates with scholars based in Netherlands, Switzerland and United States. Dietbert Neumann's co-authors include Uwe Schlattner, Theo Wallimann, David Carling, Angela Woods, Stephen R. Johnstone, Lee G.D. Fryer, Marian Carlson, Fiona C. Leiper, Roland Tuerk and Roland W. Scholz and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Nature Communications.

In The Last Decade

Dietbert Neumann

82 papers receiving 5.9k citations

Hit Papers

LKB1 Is the Upstream Kinase in the AMP-Activated Protein ... 2003 2026 2010 2018 2003 400 800 1.2k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. LKB1 Is the Upstream Kinase in the AMP-Activated Protein Kinase Cascade
    2003 1.4k citations Dietbert Neumann et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Dietbert Neumann Netherlands 34 4.8k 2.0k 1.1k 816 763 83 6.0k
Fiona A. Ross United Kingdom 22 5.0k 1.0× 1.9k 1.0× 1.4k 1.3× 1.4k 1.7× 924 1.2× 30 6.9k
Kirsty J. Mustard United Kingdom 20 3.9k 0.8× 1.7k 0.8× 1.3k 1.3× 826 1.0× 605 0.8× 22 5.0k
Mark H. Rider Belgium 51 5.6k 1.2× 1.6k 0.8× 1.3k 1.2× 860 1.1× 788 1.0× 138 7.7k
Belinda J. Michell Australia 27 4.4k 0.9× 1.7k 0.9× 2.7k 2.5× 648 0.8× 832 1.1× 31 6.5k
Kouichi Inukai Japan 41 3.1k 0.7× 1.2k 0.6× 1.2k 1.1× 995 1.2× 974 1.3× 99 5.6k
Yoshimi Nakagawa Japan 36 2.7k 0.6× 1.2k 0.6× 852 0.8× 1.1k 1.4× 616 0.8× 94 4.8k
Motonobu Anai Japan 44 3.0k 0.6× 1.4k 0.7× 1.2k 1.1× 891 1.1× 1.3k 1.6× 77 5.2k
Tadahiro Kitamura Japan 48 6.5k 1.4× 3.4k 1.7× 2.5k 2.3× 1.1k 1.3× 1.7k 2.3× 130 9.9k
Yukari Kitamura Japan 20 3.8k 0.8× 1.7k 0.8× 1.5k 1.4× 628 0.8× 669 0.9× 30 5.5k
H. Henry Dong United States 39 3.5k 0.7× 1.6k 0.8× 1.3k 1.2× 1.0k 1.2× 1.3k 1.6× 71 5.9k

Countries citing papers authored by Dietbert Neumann

Since Specialization
Citations

This map shows the geographic impact of Dietbert Neumann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dietbert Neumann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dietbert Neumann more than expected).

Fields of papers citing papers by Dietbert Neumann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dietbert Neumann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dietbert Neumann. The network helps show where Dietbert Neumann may publish in the future.

Co-authorship network of co-authors of Dietbert Neumann

This figure shows the co-authorship network connecting the top 25 collaborators of Dietbert Neumann. A scholar is included among the top collaborators of Dietbert Neumann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dietbert Neumann. Dietbert Neumann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Neumann, Dietbert, et al.. (2024). Specific Compounds Derived from Traditional Chinese Medicine Ameliorate Lipid-Induced Contractile Dysfunction in Cardiomyocytes. International Journal of Molecular Sciences. 25(15). 8131–8131.
2.
Wang, Shujin, Mengqian Hou, Dietbert Neumann, et al.. (2024). Glycolysis-Mediated Activation of v-ATPase by Nicotinamide Mononucleotide Ameliorates Lipid-Induced Cardiomyopathy by Repressing the CD36-TLR4 Axis. Circulation Research. 134(5). 505–525. 16 indexed citations
3.
Wang, Shujin, Dietbert Neumann, B. Daan Westenbrink, et al.. (2022). Ketone Body Exposure of Cardiomyocytes Impairs Insulin Sensitivity and Contractile Function through Vacuolar-Type H+-ATPase Disassembly—Rescue by Specific Amino Acid Supplementation. International Journal of Molecular Sciences. 23(21). 12909–12909. 4 indexed citations
4.
Wang, Shujin, et al.. (2022). Endosomal v-ATPase as a Sensor Determining Myocardial Substrate Preference. Metabolites. 12(7). 579–579. 7 indexed citations
5.
Wang, Shujin, Dietbert Neumann, Yilin Liu, et al.. (2020). Augmenting Vacuolar H+-ATPase Function Prevents Cardiomyocytes from Lipid-Overload Induced Dysfunction. International Journal of Molecular Sciences. 21(4). 1520–1520. 19 indexed citations
6.
Dohmen, Marc, Sarah Krieg, Xiaoqing Zhu, et al.. (2020). AMPK-dependent activation of the Cyclin Y/CDK16 complex controls autophagy. Nature Communications. 11(1). 1032–1032. 30 indexed citations
7.
Glatz, Jan F. C., et al.. (2020). Putative Role of Protein Palmitoylation in Cardiac Lipid-Induced Insulin Resistance. International Journal of Molecular Sciences. 21(24). 9438–9438. 16 indexed citations
8.
Luiken, Joost J.F.P., Miranda Nabben, Dietbert Neumann, & Jan F. C. Glatz. (2020). Understanding the distinct subcellular trafficking of CD36 and GLUT4 during the development of myocardial insulin resistance. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1866(7). 165775–165775. 30 indexed citations
9.
Liu, Yilin, Ricardo Rodríguez‐Calvo, Shujin Wang, et al.. (2019). Fluorescent labelling of membrane fatty acid transporter CD36 (SR-B2) in the extracellular loop. PLoS ONE. 14(1). e0210704–e0210704. 5 indexed citations
10.
Chanda, Dipanjan, Dietbert Neumann, & Jan F. C. Glatz. (2018). The endocannabinoid system: Overview of an emerging multi-faceted therapeutic target. Prostaglandins Leukotrienes and Essential Fatty Acids. 140. 51–56. 59 indexed citations
11.
Neumann, Dietbert. (2018). Is TAK1 a Direct Upstream Kinase of AMPK?. International Journal of Molecular Sciences. 19(8). 2412–2412. 71 indexed citations
12.
Ho, Pei‐Yin, Hui Li, Tengis S. Pavlov, et al.. (2018). β1Pix exchange factor stabilizes the ubiquitin ligase Nedd4-2 and plays a critical role in ENaC regulation by AMPK in kidney epithelial cells. Journal of Biological Chemistry. 293(29). 11612–11624. 18 indexed citations
13.
Chanda, Dipanjan, Yvonne Oligschlaeger, Xiaoqing Zhu, et al.. (2017). 2-Arachidonoylglycerol ameliorates inflammatory stress-induced insulin resistance in cardiomyocytes. Journal of Biological Chemistry. 292(17). 7105–7114. 31 indexed citations
14.
Rodríguez‐Calvo, Ricardo, Dipanjan Chanda, Yvonne Oligschlaeger, et al.. (2017). Small heterodimer partner (SHP) contributes to insulin resistance in cardiomyocytes. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1862(5). 541–551. 9 indexed citations
15.
Chanda, Dipanjan, Florence H. J. van Tienen, Arthur van den Wijngaard, et al.. (2017). Human embryonic stem cell-derived cardiomyocytes as an in vitro model to study cardiac insulin resistance. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1864(5). 1960–1967. 16 indexed citations
16.
Zhu, Xiaoqing, Vivian E.H. Dahlmans, Ramon F. Thali, et al.. (2016). AMP-activated Protein Kinase Up-regulates Mitogen-activated Protein (MAP) Kinase-interacting Serine/Threonine Kinase 1a-dependent Phosphorylation of Eukaryotic Translation Initiation Factor 4E. Journal of Biological Chemistry. 291(33). 17020–17027. 10 indexed citations
17.
Chanda, Dipanjan, Jieyi Li, Yvonne Oligschlaeger, et al.. (2016). MSP is a negative regulator of inflammation and lipogenesis in ex vivo models of non-alcoholic steatohepatitis. Experimental & Molecular Medicine. 48(9). e258–e258. 16 indexed citations
18.
King, Jennifer, Dietbert Neumann, Sheng Xiong, et al.. (2012). Role of Binding and Nucleoside Diphosphate Kinase A in the Regulation of the Cystic Fibrosis Transmembrane Conductance Regulator by AMP-activated Protein Kinase. Journal of Biological Chemistry. 287(40). 33389–33400. 30 indexed citations
19.
Bar‐Tana, Jacob, Bella Kalderon, Marianne Suter, et al.. (2008). AMPK activation by long chain fatty acyl analogs. Biochemical Pharmacology. 76(10). 1263–1275. 32 indexed citations
20.
Hartmann, Hanna, Holger K. Eltzschig, Klaus Hantke, et al.. (2006). Enterobacteriaceae and enterobacterial siderophores induce a hypoxia inducible factor-1-dependent host cell response in vitro and in vivo. International Journal of Medical Microbiology. 296. 172–172. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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