Diego Acosta‐Alvear

5.0k citations
29 papers · 3.4k indexed · 3 hit papers · h-index 20
  • Cell Biology top 0.5%
    • Endoplasmic Reticulum Stress and Disease 16
  • Aging top 2%
    • RNA regulation and disease 7
    • Ubiquitin and proteasome pathways 5
    • Photosynthetic Processes and Mechanisms 3
    • CRISPR and Genetic Engineering 3
    • Autophagy in Disease and Therapy 3
  • Immunology top 10%
    • Transgenic Plants and Applications 4
    • Pancreatic function and diabetes 4

Diego Acosta‐Alvear

28 papers receiving 3.3k citations

Hit Papers

Opposing unfolded-protein-response signals converge on...4472007202620132019200400600

Peers

Diego Acosta‐Alvear
Comparison fields: 5 of 105
  • Cell Biology 1.8k
  • Aging 99
  • Molecular Biology 2.1k
  • Epidemiology 922
  • Immunology 348
Replace Thore Hettmann with:
Thore Hettmann United States
Katarzyna Mnich Ireland
Yanjun Ma United States
Olivier Pluquet France
Hélène Zinszner United States
Diego Rojas‐Rivera Chile
Sonia M. Scaria United States
Kyosuke Haze Japan
Jenna L. Jewell United States
Kaoru Saegusa Japan
Diego Acosta‐Alvear relative to Thore Hettmann United States Thore Hettmann's profile →
Citations per field
00.5×1.5×1.8×
Thore Hettmann · 1×
Citations per year

Countries citing papers authored by Diego Acosta‐Alvear

Since Specialization
Citations

This map shows the geographic impact of Diego Acosta‐Alvear's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Diego Acosta‐Alvear with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Diego Acosta‐Alvear more than expected).

Fields of papers citing papers by Diego Acosta‐Alvear

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Diego Acosta‐Alvear. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Diego Acosta‐Alvear. The network helps show where Diego Acosta‐Alvear may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Diego Acosta‐Alvear, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Diego Acosta‐Alvear Line = papers co-authored together Diego Acosta‐Alvear links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20253
2 202429
3 20243
4 202311
5 202228
6 20220
7 20211
8 202017
9 20191
10 2019208
11 201871
12 2017162
13 2017155
14 201683
15 2015101
16 201535
17
Opposing unfolded-protein-response signals converge on death receptor 5 to control apoptosisbreakdown →
2014447
18 200951
19
XBP1 Controls Diverse Cell Type- and Condition-Specific Transcriptional Regulatory Networksbreakdown →
2007665
20 2005362

About Diego Acosta‐Alvear

Diego Acosta‐Alvear is a scholar working on Cell Biology, Biotechnology and Aging, having authored 29 papers that have together received 3.4k indexed citations. Recurring topics across this work include Endoplasmic Reticulum Stress and Disease (16 papers), RNA regulation and disease (7 papers), Ubiquitin and proteasome pathways (5 papers), Transgenic Plants and Applications (4 papers), Pancreatic function and diabetes (4 papers), Photosynthetic Processes and Mechanisms (3 papers), Autophagy in Disease and Therapy (3 papers) and CRISPR and Genetic Engineering (3 papers). The work is most often cited by research in Cell Biology (1.8k citations), Aging (99 citations) and Molecular Biology (2.1k citations). Diego Acosta‐Alvear has collaborated with scholars based in United States, Germany and Canada. Frequent co-authors include Peter Walter, Brian David Dynlacht, Alexandre Blais, Mary Tsikitis, Yuval Kluger, G Elif Karagöz, Carolina Arias, Avi Ashkenazi, Nathan H. Lents and Yiming Zhou. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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