Diana Walluscheck

550 total citations
7 papers, 394 citations indexed

About

Diana Walluscheck is a scholar working on Molecular Biology, Cancer Research and Pathology and Forensic Medicine. According to data from OpenAlex, Diana Walluscheck has authored 7 papers receiving a total of 394 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 3 papers in Cancer Research and 1 paper in Pathology and Forensic Medicine. Recurrent topics in Diana Walluscheck's work include DNA Repair Mechanisms (4 papers), Cell death mechanisms and regulation (3 papers) and NF-κB Signaling Pathways (3 papers). Diana Walluscheck is often cited by papers focused on DNA Repair Mechanisms (4 papers), Cell death mechanisms and regulation (3 papers) and NF-κB Signaling Pathways (3 papers). Diana Walluscheck collaborates with scholars based in Germany, United Kingdom and United States. Diana Walluscheck's co-authors include Albert Roessner, Khuloud Bajbouj, Regine Schneider‐Stock, Peter Schönfeld, Hala Gali‐Muhtasib, Antje Diestel, Günter Reuter, Maria Cristina Onorati, Sameer Phalke and Roland Hartig and has published in prestigious journals such as Nature Genetics, Oxidative Medicine and Cellular Longevity and Journal of Cellular and Molecular Medicine.

In The Last Decade

Diana Walluscheck

7 papers receiving 382 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Diana Walluscheck Germany 7 215 146 114 62 53 7 394
Abdulkhaleg Ibrahim France 9 274 1.3× 42 0.3× 27 0.2× 16 0.3× 14 0.3× 14 345
Luquan Yang China 10 257 1.2× 49 0.3× 17 0.1× 16 0.3× 103 1.9× 12 433
Beibei Lv China 11 199 0.9× 15 0.1× 30 0.3× 19 0.3× 59 1.1× 30 333
Renate Schäfer Germany 11 242 1.1× 14 0.1× 13 0.1× 10 0.2× 59 1.1× 18 409
Xiaokang Liu China 10 112 0.5× 9 0.1× 12 0.1× 30 0.5× 97 1.8× 23 312
Stephany El‐Hayek Lebanon 10 301 1.4× 22 0.2× 12 0.1× 13 0.2× 6 0.1× 25 561
Meng Lin China 11 210 1.0× 9 0.1× 10 0.1× 20 0.3× 20 0.4× 16 428
Yuyu Guo China 8 210 1.0× 13 0.1× 5 0.0× 20 0.3× 117 2.2× 15 317
Dávid Drutovič Czechia 10 275 1.3× 5 0.0× 24 0.2× 12 0.2× 49 0.9× 16 385

Countries citing papers authored by Diana Walluscheck

Since Specialization
Citations

This map shows the geographic impact of Diana Walluscheck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Diana Walluscheck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Diana Walluscheck more than expected).

Fields of papers citing papers by Diana Walluscheck

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Diana Walluscheck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Diana Walluscheck. The network helps show where Diana Walluscheck may publish in the future.

Co-authorship network of co-authors of Diana Walluscheck

This figure shows the co-authorship network connecting the top 25 collaborators of Diana Walluscheck. A scholar is included among the top collaborators of Diana Walluscheck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Diana Walluscheck. Diana Walluscheck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Silver, Andrew, et al.. (2017). Chk1 Promotes DNA Damage Response Bypass following Oxidative Stress in a Model of Hydrogen Peroxide‐Associated Ulcerative Colitis through JNK Inactivation and Chromatin Binding. Oxidative Medicine and Cellular Longevity. 2017(1). 9303158–9303158. 6 indexed citations
2.
Walluscheck, Diana, Angela Poehlmann, Roland Hartig, et al.. (2013). ATF2 knockdown reinforces oxidative stress‐induced apoptosis in TE7 cancer cells. Journal of Cellular and Molecular Medicine. 17(8). 976–988. 21 indexed citations
3.
Poehlmann, Angela, Diana Walluscheck, Roland Hartig, et al.. (2013). Non‐apoptotic function of caspases in a cellular model of hydrogen peroxide‐associated colitis. Journal of Cellular and Molecular Medicine. 17(7). 901–913. 10 indexed citations
4.
Poehlmann, Angela, Peter Schönfeld, Diana Walluscheck, et al.. (2013). Repeated H2O2 exposure drives cell cycle progression in an in vitro model of ulcerative colitis. Journal of Cellular and Molecular Medicine. 17(12). 1619–1631. 15 indexed citations
5.
Poehlmann, Angela, Diana Walluscheck, Khuloud Bajbouj, et al.. (2010). Cutting edge: Chk1 directs senescence and mitotic catastrophe in recovery from G2 checkpoint arrest. Journal of Cellular and Molecular Medicine. 15(7). 1528–1541. 24 indexed citations
6.
Phalke, Sameer, et al.. (2009). Retrotransposon silencing and telomere integrity in somatic cells of Drosophila depends on the cytosine-5 methyltransferase DNMT2. Nature Genetics. 41(6). 696–702. 146 indexed citations
7.
Diestel, Antje, Khuloud Bajbouj, Diana Walluscheck, et al.. (2007). Lack of p53 augments thymoquinone-induced apoptosis and caspase activation in human osteosarcoma cells. Cancer Biology & Therapy. 6(2). 160–169. 172 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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