Dengjie Ouyang
- Molecular Biology
- Cancer Research
- Oncology
- Pulmonary and Respiratory Medicine
- Endocrinology, Diabetes and Metabolism
- Topics
- Thyroid Cancer Diagnosis and Treatment (9 papers)Cancer Immunotherapy and Biomarkers (4 papers)Cancer Diagnosis and Treatment (4 papers)
- Journals
- SHILAP Revista de lepidopterologíaScientific ReportsJournal of Neuroinflammation
- Partner nations
- China
In The Last Decade
Dengjie Ouyang
22 papers receiving 224 citations
Peers
Comparison fields: 5 of 52
- Molecular Biology 103
- Cancer Research 73
- Oncology 67
- Pulmonary and Respiratory Medicine 40
- Endocrinology, Diabetes and Metabolism 35
Countries citing papers authored by Dengjie Ouyang
This map shows the geographic impact of Dengjie Ouyang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dengjie Ouyang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dengjie Ouyang more than expected).
Fields of papers citing papers by Dengjie Ouyang
This network shows the impact of papers produced by Dengjie Ouyang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dengjie Ouyang. The network helps show where Dengjie Ouyang may publish in the future.
Co-authorship network of co-authors of Dengjie Ouyang
This figure shows the co-authorship network connecting the top 25 collaborators of Dengjie Ouyang. A scholar is included among the top collaborators of Dengjie Ouyang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dengjie Ouyang. Dengjie Ouyang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 4 | |
| 3 | 1 | |
| 4 | 3 | |
| 5 | 16 | |
| 6 | Insights into the Association Between QSER1 and M2 Macrophages and Remarkable Malignancy Characteristics in Hepatocellular Carcinoma | 2 |
| 7 | 5 | |
| 8 | 7 | |
| 9 | 8 | |
| 10 | 6 | |
| 11 | 5 | |
| 12 | 0 | |
| 13 | 6 | |
| 14 | 4 | |
| 15 | 8 | |
| 16 | 6 | |
| 17 | 3 | |
| 18 | 35 | |
| 19 | 1 | |
| 20 | Identification of lncRNAs via microarray analysis for predicting HER2-negative breast cancer response to neoadjuvant chemotherapy. | 14 |
About Dengjie Ouyang
Dengjie Ouyang is a scholar working on Endocrinology, Diabetes and Metabolism, Cancer Research and Oncology, having authored 24 papers that have together received 225 indexed citations. Recurring topics across this work include Thyroid Cancer Diagnosis and Treatment (9 papers), Cancer Immunotherapy and Biomarkers (4 papers) and Cancer Diagnosis and Treatment (4 papers). The work is most often cited by research in Cancer Research (73 citations), Oncology (67 citations) and Endocrinology, Diabetes and Metabolism (35 citations). Dengjie Ouyang has collaborated with scholars based in China. Frequent co-authors include Wenjun Yi, Qiongyan Zou, Qitong Chen, Peng Huang, Hongye He, Shi Chang, Lun Li, Qianying Li, Piao Zhao and Mian Liu. Their work appears in journals such as SHILAP Revista de lepidopterología, Scientific Reports and Journal of Neuroinflammation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.