David Wynick

5.3k total citations
96 papers, 4.2k citations indexed

About

David Wynick is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology. According to data from OpenAlex, David Wynick has authored 96 papers receiving a total of 4.2k indexed citations (citations by other indexed papers that have themselves been cited), including 65 papers in Cellular and Molecular Neuroscience, 45 papers in Molecular Biology and 23 papers in Physiology. Recurrent topics in David Wynick's work include Neuropeptides and Animal Physiology (60 papers), Receptor Mechanisms and Signaling (25 papers) and Pain Mechanisms and Treatments (22 papers). David Wynick is often cited by papers focused on Neuropeptides and Animal Physiology (60 papers), Receptor Mechanisms and Signaling (25 papers) and Pain Mechanisms and Treatments (22 papers). David Wynick collaborates with scholars based in United Kingdom, United States and Australia. David Wynick's co-authors include Fiona E. Holmes, Andrea Bacon, Niall C. H. Kerr, Robert J. P. Pope, Penny Vanderplank, Stephen R. Bloom, Robert A. Steiner, S.R. Bloom, Peter Hammond and Vassilis Pachnis and has published in prestigious journals such as Nature, New England Journal of Medicine and Proceedings of the National Academy of Sciences.

In The Last Decade

David Wynick

94 papers receiving 4.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David Wynick United Kingdom 39 2.8k 2.0k 834 817 540 96 4.2k
Andrea Tamás Hungary 39 3.9k 1.4× 2.4k 1.2× 574 0.7× 1.1k 1.4× 326 0.6× 196 5.3k
Gong Ju China 40 2.8k 1.0× 1.9k 0.9× 1.4k 1.7× 517 0.6× 872 1.6× 167 5.8k
M.A. Ghatei United Kingdom 34 3.5k 1.3× 2.0k 1.0× 1.4k 1.7× 1.3k 1.6× 810 1.5× 89 5.6k
Anne‐Marie O’Carroll United Kingdom 29 1.4k 0.5× 1.2k 0.6× 324 0.4× 951 1.2× 684 1.3× 50 4.5k
Manjula Mahata United States 36 1.8k 0.6× 2.4k 1.2× 472 0.6× 306 0.4× 323 0.6× 104 4.1k
Anders Nobin Sweden 33 2.3k 0.8× 1.1k 0.6× 655 0.8× 385 0.5× 691 1.3× 94 4.5k
Henri Doods Germany 35 3.4k 1.2× 2.4k 1.2× 1.2k 1.4× 413 0.5× 1.0k 1.9× 107 5.3k
F. Lembeck Austria 40 3.7k 1.3× 2.4k 1.2× 2.2k 2.6× 636 0.8× 399 0.7× 188 6.0k
V. Höllt Germany 39 3.2k 1.1× 2.3k 1.1× 1.3k 1.6× 264 0.3× 326 0.6× 95 4.5k
Tomoya Nakamachi Japan 33 1.8k 0.6× 1.3k 0.6× 406 0.5× 468 0.6× 822 1.5× 107 3.5k

Countries citing papers authored by David Wynick

Since Specialization
Citations

This map shows the geographic impact of David Wynick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Wynick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Wynick more than expected).

Fields of papers citing papers by David Wynick

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Wynick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Wynick. The network helps show where David Wynick may publish in the future.

Co-authorship network of co-authors of David Wynick

This figure shows the co-authorship network connecting the top 25 collaborators of David Wynick. A scholar is included among the top collaborators of David Wynick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Wynick. David Wynick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Holmes, Fiona E., Niall C. H. Kerr, Ying‐Ju Chen, et al.. (2016). Targeted disruption of the orphan receptor Gpr151 does not alter pain-related behaviour despite a strong induction in dorsal root ganglion expression in a model of neuropathic pain. Molecular and Cellular Neuroscience. 78. 35–40. 17 indexed citations
2.
Kerr, Niall C. H., et al.. (2015). The generation of knock-in mice expressing fluorescently tagged galanin receptors 1 and 2. Molecular and Cellular Neuroscience. 68. 258–271. 12 indexed citations
3.
4.
Kerr, Niall C. H., Alexander Pintzas, Fiona E. Holmes, et al.. (2010). The expression of ELK transcription factors in adult DRG: Novel isoforms, antisense transcripts and upregulation by nerve damage. Molecular and Cellular Neuroscience. 44(2). 165–177. 18 indexed citations
5.
Hulse, Richard P., David Wynick, & Lucy F. Donaldson. (2009). Intact cutaneous C fibre afferent properties in mechanical and cold neuropathic allodynia. European Journal of Pain. 14(6). 565.e1–565.e10. 32 indexed citations
6.
Kerr, Niall C. H., Fiona E. Holmes, & David Wynick. (2008). Novel mRNA isoforms of the sodium channels Nav1.2, Nav1.3 and Nav1.7 encode predicted two-domain, truncated proteins. Neuroscience. 155(3). 797–808. 10 indexed citations
7.
Holmes, Fiona E., Penny Vanderplank, Niall C. H. Kerr, et al.. (2008). Intra‐neural administration of fractalkine attenuates neuropathic pain‐related behaviour. Journal of Neurochemistry. 106(2). 640–649. 31 indexed citations
8.
Bacon, Andrea, et al.. (2007). Characterization of an Enhancer Region of the Galanin Gene That Directs Expression to the Dorsal Root Ganglion and Confers Responsiveness to Axotomy. Journal of Neuroscience. 27(24). 6573–6580. 24 indexed citations
9.
Kerr, Niall C. H., et al.. (2007). Osteopontin expression and function within the dorsal root ganglion. Neuroreport. 18(2). 153–157. 21 indexed citations
10.
Hawes, Jessica J., et al.. (2007). Galanin Protects Against Behavioral and Neurochemical Correlates of Opiate Reward. Neuropsychopharmacology. 33(8). 1864–1873. 50 indexed citations
11.
Pope, Robert J. P., et al.. (2006). Activation of the galanin receptor 2 (GalR2) protects the hippocampus from neuronal damage. Journal of Neurochemistry. 100(3). 780–789. 96 indexed citations
12.
Holmes, Fiona E., et al.. (2006). Mice deficient for galanin receptor 2 have decreased neurite outgrowth from adult sensory neurons and impaired pain‐like behaviour. Journal of Neurochemistry. 99(3). 1000–1010. 80 indexed citations
13.
Holmes, Fiona E., et al.. (2005). Use of genetically engineered transgenic mice to investigate the role of galanin in the peripheral nervous system after injury. Neuropeptides. 39(3). 191–199. 48 indexed citations
14.
Hawes, Jessica J., Darlene H. Brunzell, David Wynick, Venetia Zachariou, & Marina R. Picciotto. (2005). GalR1, but not GalR2 or GalR3, levels are regulated by galanin signaling in the locus coeruleus through a cyclic AMP‐dependent mechanism. Journal of Neurochemistry. 93(5). 1168–1176. 28 indexed citations
15.
Bacon, Andrea, Fiona E. Holmes, Caroline J. Small, et al.. (2002). Transgenic over-expression of galanin in injured primary sensory neurons. Neuroreport. 13(16). 2129–2132. 24 indexed citations
16.
Kehr, Ján, Takashi Yoshitake, David Wynick, et al.. (2001). Microdialysis in freely moving mice: determination of acetylcholine, serotonin and noradrenaline release in galanin transgenic mice. Journal of Neuroscience Methods. 109(1). 71–80. 59 indexed citations
17.
Kerr, Bradley J., et al.. (2001). Endogenous galanin potentiates spinal nociceptive processing following inflammation. Pain. 93(3). 267–277. 44 indexed citations
18.
Wynick, David, John V. Priestley, Stephen B. McMahon, et al.. (1999). Galanin is a growth factor to the central and peripheral nervous system. Brain Research. 848. 1 indexed citations
19.
Wynick, David & Stephen R. Bloom. (1990). Magnetic Bead Separation of Anterior Pituitary Cells. Neuroendocrinology. 52(6). 560–565. 9 indexed citations
20.
Wynick, David, J. V. Anderson, Susan Williams, & S.R. Bloom. (1989). RESISTANCE OF METASTATIC PANCREATIC ENDOCRINE TUMOURS AFTER LONG‐TERM TREATMENT WITH THE SOMATOSTATIN ANALOGUE OCTREOTIDE (SMS 201–995). Clinical Endocrinology. 30(4). 385–388. 44 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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