David T. Fritz
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- TGF-β signaling in diseases 8
- RNA Research and Splicing 7
- RNA modifications and cancer 5
- Bone Metabolism and Diseases 5
- RNA and protein synthesis mechanisms 4
- Developmental Biology and Gene Regulation 3
- Cancer-related gene regulation 2
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- Viral Infections and Immunology Research 3
- Co-authors
- Jeffrey WiluszLance P. FordMin GaoMelissa B. RogersShan JiangRein StrijkerArthur D. LevinsonDonglin Liu
- Journals
- Journal of Cellular Biochemistry (4 papers)Journal of Biological Chemistry (2 papers)Current Medicinal Chemistry (1 paper)
- Partner nations
- United StatesIrelandSaudi Arabia
In The Last Decade
David T. Fritz
20 papers receiving 494 citations
Peers
Comparison fields: 5 of 69
- Molecular Biology 422
- Cancer Research 42
- Genetics 53
- Cardiology and Cardiovascular Medicine 36
- Virology 7
Countries citing papers authored by David T. Fritz
This map shows the geographic impact of David T. Fritz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David T. Fritz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David T. Fritz more than expected).
Fields of papers citing papers by David T. Fritz
This network shows the impact of papers produced by David T. Fritz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David T. Fritz. The network helps show where David T. Fritz may publish in the future.
Co-authorship network
The 25 scholars most cited alongside David T. Fritz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 7 | |
| 2 | Bone morphogenetic protein-focused strategies to induce cytotoxicity in lung cancer cells. | 2014 | 24 |
| 3 | 2011 | 11 | |
| 4 | 2011 | 6 | |
| 5 | 2010 | 9 | |
| 6 | 2010 | 7 | |
| 7 | 2010 | 8 | |
| 8 | 2009 | 30 | |
| 9 | 2008 | 12 | |
| 10 | 2008 | 11 | |
| 11 | 2006 | 5 | |
| 12 | 2006 | 28 | |
| 13 | 2004 | 21 | |
| 14 | 2004 | 24 | |
| 15 | 2004 | 25 | |
| 16 | 2002 | 47 | |
| 17 | 2000 | 1 | |
| 18 | 2000 | 156 | |
| 19 | 2000 | 37 | |
| 20 | 1989 | 32 |
About David T. Fritz
David T. Fritz is a scholar working on Molecular Biology, Cancer Research and Cardiology and Cardiovascular Medicine, having authored 20 papers that have together received 501 indexed citations. Recurring topics across this work include TGF-β signaling in diseases (8 papers), RNA Research and Splicing (7 papers), RNA modifications and cancer (5 papers), Bone Metabolism and Diseases (5 papers), RNA and protein synthesis mechanisms (4 papers), Viral Infections and Immunology Research (3 papers), Developmental Biology and Gene Regulation (3 papers) and Cancer-related gene regulation (2 papers). The work is most often cited by research in Molecular Biology (422 citations), Cancer Research (42 citations) and Genetics (53 citations). David T. Fritz has collaborated with scholars based in United States, Ireland and Saudi Arabia. Frequent co-authors include Jeffrey Wilusz, Lance P. Ford, Min Gao, Melissa B. Rogers, Shan Jiang, Rein Strijker, Arthur D. Levinson, Donglin Liu, Young‐Woo Park and Michael G. Katze. Their work appears in journals such as Journal of Cellular Biochemistry, Journal of Biological Chemistry, Current Medicinal Chemistry, genesis and Molecular Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.