David Ray

4.3k total citations
95 papers, 3.0k citations indexed

About

David Ray is a scholar working on Cellular and Molecular Neuroscience, Plant Science and Molecular Biology. According to data from OpenAlex, David Ray has authored 95 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Cellular and Molecular Neuroscience, 26 papers in Plant Science and 23 papers in Molecular Biology. Recurrent topics in David Ray's work include Pesticide Exposure and Toxicity (25 papers), Neuroscience and Neuropharmacology Research (24 papers) and Barrier Structure and Function Studies (9 papers). David Ray is often cited by papers focused on Pesticide Exposure and Toxicity (25 papers), Neuroscience and Neuropharmacology Research (24 papers) and Barrier Structure and Function Studies (9 papers). David Ray collaborates with scholars based in United Kingdom, United States and France. David Ray's co-authors include Philip J. Forshaw, Paul G. Richards, Jeffrey R. Fry, T. Lister, Colin L. Willis, Christopher C. Nolan, J. E. Cremer, John Cavanagh, Chris Nolan and Wayne G. Carter and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Biochemistry.

In The Last Decade

David Ray

90 papers receiving 2.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David Ray United Kingdom 31 1.3k 728 539 515 486 95 3.0k
Vinay K. Khanna India 31 480 0.4× 780 1.1× 83 0.2× 505 1.0× 391 0.8× 119 3.1k
Florianne Monnet‐Tschudi Switzerland 27 383 0.3× 528 0.7× 55 0.1× 472 0.9× 320 0.7× 59 1.9k
Anna Bal‐Price Italy 39 361 0.3× 1.4k 1.9× 63 0.1× 596 1.2× 994 2.0× 63 4.3k
Xiaoli He China 29 408 0.3× 842 1.2× 727 1.3× 188 0.4× 822 1.7× 75 2.6k
Evelyn Tiffany‐Castiglioni United States 32 340 0.3× 810 1.1× 42 0.1× 1.2k 2.2× 547 1.1× 85 2.9k
Siyaram Pandey Canada 42 362 0.3× 2.1k 2.8× 58 0.1× 339 0.7× 191 0.4× 97 4.5k
Jin‐Sung Choi South Korea 36 435 0.3× 2.2k 3.0× 139 0.3× 129 0.3× 1.1k 2.2× 109 4.1k
Makoto Shibutani Japan 37 455 0.4× 1.8k 2.5× 42 0.1× 915 1.8× 381 0.8× 333 5.3k
Raquel Marín Spain 39 272 0.2× 1.7k 2.3× 152 0.3× 87 0.2× 414 0.9× 105 3.9k
Hideshi Ihara Japan 34 459 0.4× 1.8k 2.4× 111 0.2× 194 0.4× 275 0.6× 112 3.9k

Countries citing papers authored by David Ray

Since Specialization
Citations

This map shows the geographic impact of David Ray's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Ray with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Ray more than expected).

Fields of papers citing papers by David Ray

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Ray. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Ray. The network helps show where David Ray may publish in the future.

Co-authorship network of co-authors of David Ray

This figure shows the co-authorship network connecting the top 25 collaborators of David Ray. A scholar is included among the top collaborators of David Ray based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Ray. David Ray is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Smith, Paul A., Nethia Mohana‐Kumaran, Robert Layfield, et al.. (2007). Vanilloid receptor agonists and antagonists are mitochondrial inhibitors: How vanilloids cause non-vanilloid receptor mediated cell death. Biochemical and Biophysical Research Communications. 354(1). 50–55. 80 indexed citations
2.
Fowler, Maxine J, et al.. (2007). The selective neurotoxicity produced by 3-chloropropanediol in the rat is not a result of energy deprivation. Toxicology. 232(3). 268–276. 22 indexed citations
3.
Willis, Colin L., Claire J. Garwood, & David Ray. (2007). A size selective vascular barrier in the rat area postrema formed by perivascular macrophages and the extracellular matrix. Neuroscience. 150(2). 498–509. 75 indexed citations
4.
Willis, Colin L. & David Ray. (2006). Antioxidants attenuate MK-801-induced cortical neurotoxicity in the rat. NeuroToxicology. 28(1). 161–167. 22 indexed citations
5.
Ray, David & Jeffrey R. Fry. (2005). A reassessment of the neurotoxicity of pyrethroid insecticides. Pharmacology & Therapeutics. 111(1). 174–193. 221 indexed citations
6.
7.
Prior, M. J. W., et al.. (2004). MRI characterisation of a novel rat model of focal astrocyte loss. Magnetic Resonance Materials in Physics Biology and Medicine. 17(3-6). 125–132. 14 indexed citations
8.
Willis, Colin L., Christopher C. Nolan, T. Lister, et al.. (2003). Focal astrocyte loss is followed by microvascular damage, with subsequent repair of the blood‐brain barrier in the apparent absence of direct astrocytic contact. Glia. 45(4). 325–337. 100 indexed citations
9.
Guérin, Christopher J., et al.. (2001). The dynamics of blood–brain barrier breakdown in an experimental model of glial cell degeneration. Neuroscience. 103(4). 873–883. 16 indexed citations
10.
Forshaw, Philip J., T. Lister, & David Ray. (2000). The Role of Voltage-Gated Chloride Channels in Type II Pyrethroid Insecticide Poisoning. Toxicology and Applied Pharmacology. 163(1). 1–8. 53 indexed citations
11.
Ray, David. (1998). Chronic effects of low level exposure to anticholinesterases — a mechanistic review. Toxicology Letters. 102-103. 527–533. 85 indexed citations
12.
Holton, JL, Chris Nolan, S Burr, David Ray, & John Cavanagh. (1997). Increasing or decreasing nervous activity modulates the severity of the glio-vascular lesions of 1,3-dinitrobenzene in the rat: effects of the tremorgenic pyrethroid, Bifenthrin, and of anaesthesia. Acta Neuropathologica. 93(2). 159–165. 12 indexed citations
13.
Ray, David. (1994). Book Reviews: Handbook Exp. Pharmacology 103. Physiology and Pharmacology of the Blood-Brain Barrier. Human & Experimental Toxicology. 13(2). 141–141. 1 indexed citations
14.
Forshaw, Philip J., T. Lister, & David Ray. (1993). Inhibition of a neuronal voltage-dependent chloride channel by the type II pyrethroid, deltamethrin. Neuropharmacology. 32(2). 105–111. 39 indexed citations
15.
Ray, David, et al.. (1992). Functional/metabolic modulation of the brain stem lesions caused by 1,3-dinitrobenzene in the rat.. PubMed. 13(2). 379–88. 27 indexed citations
16.
Ray, David, et al.. (1989). Poems. College English. 51(6). 577–588.
17.
Joy, Robert M., Timothy E. Albertson, & David Ray. (1989). Type I and type II pyrethroids increase inhibition in the hippocampal dentate gyrus of the rat. Toxicology and Applied Pharmacology. 98(3). 398–412. 10 indexed citations
18.
Lister, T. & David Ray. (1988). The role of basal forebrain in the primary cholinergic vasodilation in rat neocortex produced by systemic administration of cismethrin. Brain Research. 450(1-2). 364–368. 10 indexed citations
19.
Ray, David, John Gattorna, & Mike Allen. (1980). Handbook of Distribution Costing and Control. International Journal of Physical Distribution & Materials Management. 10(5/6). 209–429. 4 indexed citations
20.
Ray, David. (1959). The Chicago review anthology. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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