David Q. Wang

438 total citations · 1 hit paper
8 papers, 325 citations indexed

About

David Q. Wang is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Oncology. According to data from OpenAlex, David Q. Wang has authored 8 papers receiving a total of 325 indexed citations (citations by other indexed papers that have themselves been cited), including 2 papers in Molecular Biology, 2 papers in Pulmonary and Respiratory Medicine and 2 papers in Oncology. Recurrent topics in David Q. Wang's work include Gallbladder and Bile Duct Disorders (2 papers), Pancreatic and Hepatic Oncology Research (1 paper) and Cholesterol and Lipid Metabolism (1 paper). David Q. Wang is often cited by papers focused on Gallbladder and Bile Duct Disorders (2 papers), Pancreatic and Hepatic Oncology Research (1 paper) and Cholesterol and Lipid Metabolism (1 paper). David Q. Wang collaborates with scholars based in United States, Italy and Germany. David Q. Wang's co-authors include Piero Portincasa, Saverio Marchi, Agnieszka Karkucińska‐Więckowska, Grzegorz Węgrzyn, Jerzy Duszyński, Sabine Borchard, Ziyu Ren, Karolina Pierzynowska, Hans Zischka and Alessandro Rimessi and has published in prestigious journals such as Gastroenterology, The FASEB Journal and International review of cell and molecular biology.

In The Last Decade

David Q. Wang

8 papers receiving 314 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mitochondria and Reactive Oxygen Species in Aging and Age-Related Diseases

2018 314 citations Carlotta Giorgi, Saverio Marchi et al. International review of cell and molecular biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David Q. Wang United States	3	169	72	45	27	26	8	325
Paulina Jędrak Poland	4	226 1.3×	76 1.1×	47 1.0×	27 1.0×	28 1.1×	6	388
Lígia Fão Portugal	8	264 1.6×	84 1.2×	52 1.2×	9 0.3×	26 1.0×	10	408
Irene Pradas Spain	14	309 1.8×	159 2.2×	35 0.8×	47 1.7×	18 0.7×	18	533
Hao-Fei Liu China	6	167 1.0×	77 1.1×	59 1.3×	10 0.4×	46 1.8×	7	306
Saba Ubaid India	6	162 1.0×	94 1.3×	75 1.7×	9 0.3×	40 1.5×	9	410
P. Andy Li China	9	156 0.9×	63 0.9×	69 1.5×	8 0.3×	58 2.2×	14	366
Vanesa Cepas Spain	7	127 0.8×	99 1.4×	26 0.6×	22 0.8×	13 0.5×	9	469
Lucia Cialabrini Italy	7	204 1.2×	79 1.1×	109 2.4×	23 0.9×	9 0.3×	8	497
Guoqing Wu China	11	193 1.1×	159 2.2×	27 0.6×	17 0.6×	13 0.5×	25	445
Huifang Liu China	10	194 1.1×	162 2.3×	47 1.0×	7 0.3×	29 1.1×	33	480

Countries citing papers authored by David Q. Wang

Since Specialization

Citations

This map shows the geographic impact of David Q. Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Q. Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Q. Wang more than expected).

Fields of papers citing papers by David Q. Wang

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Q. Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Q. Wang. The network helps show where David Q. Wang may publish in the future.

Co-authorship network of co-authors of David Q. Wang

This figure shows the co-authorship network connecting the top 25 collaborators of David Q. Wang. A scholar is included among the top collaborators of David Q. Wang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Q. Wang. David Q. Wang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Giorgi, Carlotta, Saverio Marchi, Inês C. M. Simões, et al.. (2018). Mitochondria and Reactive Oxygen Species in Aging and Age-Related Diseases. International review of cell and molecular biology. 340. 209–344. 314 indexed citations breakdown →

2.

Wang, Helen, Ornella de Bari, Min Liu, & David Q. Wang. (2018). 505 - Activation of G Protein-Coupled Receptor 30 (GPR30), a Novel Estrogen Receptor, by the Potent GPR30-Selective Agonist G-1 Greatly Enhances Cholesterol Cholelithogenesis in Female Mice. Gastroenterology. 154(6). S–1093. 2 indexed citations

3.

Wang, David Q., Ornella de Bari, & Helen H. Wang. (2017). G protein‐coupled receptor 30 (GPR30), a novel estrogen receptor, plays an independent role in the pathogenesis of estrogen‐induced cholesterol gallstones in female mice. The FASEB Journal. 31(S1). 1 indexed citations

4.

Bari, Ornella de, Helen H. Wang, Piero Portincasa, Min Liu, & David Q. Wang. (2015). Sa1336 Molecular Mechanisms Underlying the Critical Role of the G Protein-Coupled Receptor 30 (GPR30), a Novel Estrogen Receptor, in the Formation of Lithogenic Bile Through a Non-Transcriptional Regulatory Mode in 17β-Estradiol (E2)-Treated Mice. Gastroenterology. 148(4). S–295. 1 indexed citations

5.

Portincasa, Piero, et al.. (2012). 63 A Novel Cluster of Patients With Familial Mediterranean Fever (FMF) Displayed Abnormal Gastrointestinal Motorfunction. Gastroenterology. 142(5). S–16. 2 indexed citations

6.

Wang, Helen H., Kristina J. Liu, Deborah J. Clegg, Piero Portincasa, & David Q. Wang. (2012). 470 Deletion of the Gpr30 Gene, a Novel Estrogen Receptor, Results in Increased Susceptibility to Cholesterol Gallstone Formation in Mice. Gastroenterology. 142(5). S–98. 2 indexed citations

7.

Wang, Helen H., Piero Portincasa, Min Liu, & David Q. Wang. (2011). Rapid Postprandial Gallbladder Refilling and Increased Turnover of Bile Prevent Cholesterol Crystallization in Small Heterodimer Partner (SHP) Knockout Mice. Gastroenterology. 140(5). S–67. 1 indexed citations

8.

Wang, Helen H., Mark J. Evans, Piero Portincasa, & David Q. Wang. (2009). 346 Increased Hepatic Bile Acid (BA) Synthesis Prevents Cholesterol Gallstones in Small Heterodimer Partner (SHP) Knockout Mice. Gastroenterology. 136(5). A–800. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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