David P. McCobb
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 2%
- Cardiology and Cardiovascular Medicine top 5%
- Sensory Systems top 5%
- Social Psychology top 10%
- Co-authors
- Jiuyong XieKurt G. BeamAlice ButlerSusan TsunodaAguan WeiLawrence SalkoffPeter V. LovellMary S. Tyler
- Topics
- Ion channel regulation and function (10 papers)Cardiac electrophysiology and arrhythmias (5 papers)Neuroendocrine regulation and behavior (4 papers)
- Partner nations
- United StatesGermanyAustria
In The Last Decade
David P. McCobb
13 papers receiving 1.4k citations
Hit Papers
Peers
Comparison fields: 5 of 83
- Molecular Biology 1.2k
- Cellular and Molecular Neuroscience 755
- Cardiology and Cardiovascular Medicine 434
- Sensory Systems 123
- Social Psychology 88
Countries citing papers authored by David P. McCobb
This map shows the geographic impact of David P. McCobb's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David P. McCobb with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David P. McCobb more than expected).
Fields of papers citing papers by David P. McCobb
This network shows the impact of papers produced by David P. McCobb. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David P. McCobb. The network helps show where David P. McCobb may publish in the future.
Co-authorship network of co-authors of David P. McCobb
This figure shows the co-authorship network connecting the top 25 collaborators of David P. McCobb. A scholar is included among the top collaborators of David P. McCobb based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David P. McCobb. David P. McCobb is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 34 | |
| 2 | 28 | |
| 3 | 33 | |
| 4 | 17 | |
| 5 | 9 | |
| 6 | 51 | |
| 7 | 65 | |
| 8 | 16 | |
| 9 | 260 | |
| 10 | mSlo , a Complex Mouse Gene Encoding "Maxi" Calcium-Activated Potassium Channelsbreakdown → | 551 |
| 11 | 142 | |
| 12 | 160 | |
| 13 | 24 |
About David P. McCobb
David P. McCobb is a scholar working on Behavioral Neuroscience, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine, having authored 13 papers that have together received 1.4k indexed citations. Recurring topics across this work include Ion channel regulation and function (10 papers), Cardiac electrophysiology and arrhythmias (5 papers) and Neuroendocrine regulation and behavior (4 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (755 citations), Sensory Systems (123 citations) and Cardiology and Cardiovascular Medicine (434 citations). David P. McCobb has collaborated with scholars based in United States, Germany and Austria. Frequent co-authors include Jiuyong Xie, Kurt G. Beam, Alice Butler, Susan Tsunoda, Aguan Wei, Lawrence Salkoff, Peter V. Lovell, Mary S. Tyler, Yuko Hara and Hans‐Guenther Knaus. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Neuron.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.