David H. Rivier

637 total citations
13 papers, 564 citations indexed

About

David H. Rivier is a scholar working on Molecular Biology, Genetics and Food Science. According to data from OpenAlex, David H. Rivier has authored 13 papers receiving a total of 564 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 1 paper in Genetics and 1 paper in Food Science. Recurrent topics in David H. Rivier's work include Fungal and yeast genetics research (9 papers), Genomics and Chromatin Dynamics (8 papers) and DNA Repair Mechanisms (5 papers). David H. Rivier is often cited by papers focused on Fungal and yeast genetics research (9 papers), Genomics and Chromatin Dynamics (8 papers) and DNA Repair Mechanisms (5 papers). David H. Rivier collaborates with scholars based in United States. David H. Rivier's co-authors include Jasper Rine, Ann E. Ehrenhofer‐Murray, Lisa S. Young, Karen U. Sprague, Eugenia Y. Xu, Hanna Kim, Simone Ottonello, Joanne Ekena, Kirstin Replogle and Lorraine Pillus and has published in prestigious journals such as Science, Cell and Journal of Biological Chemistry.

In The Last Decade

David H. Rivier

13 papers receiving 556 citations

Peers

David H. Rivier
David Norris United States
Melanie Piper United Kingdom
P Laurenson United States
G. Drugeon France
David P. Aiello United States
Renzo Lucchini Switzerland
David Norris United States
David H. Rivier
Citations per year, relative to David H. Rivier David H. Rivier (= 1×) peers David Norris

Countries citing papers authored by David H. Rivier

Since Specialization
Citations

This map shows the geographic impact of David H. Rivier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David H. Rivier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David H. Rivier more than expected).

Fields of papers citing papers by David H. Rivier

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David H. Rivier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David H. Rivier. The network helps show where David H. Rivier may publish in the future.

Co-authorship network of co-authors of David H. Rivier

This figure shows the co-authorship network connecting the top 25 collaborators of David H. Rivier. A scholar is included among the top collaborators of David H. Rivier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David H. Rivier. David H. Rivier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Kamakaka, Rohinton T., et al.. (2008). The DNA End-Binding Protein Ku Regulates Silencing at the InternalHMLandHMRLoci inSaccharomyces cerevisiae. Genetics. 180(3). 1407–1418. 17 indexed citations
2.
Replogle, Kirstin, et al.. (1999). Designer deletion and prototrophic strains derived fromSaccharomyces cerevisiae strain W303-1a. Yeast. 15(11). 1141–1149. 23 indexed citations
3.
Rivier, David H., et al.. (1999). Identification of a Compound Origin of Replication at theHMR-E Locus in Saccharomyces cerevisiae. Journal of Biological Chemistry. 274(7). 4155–4159. 19 indexed citations
4.
Xu, Eugenia Y., Hanna Kim, Kirstin Replogle, Jasper Rine, & David H. Rivier. (1999). Identification of SAS4 and SAS5, Two Genes That Regulate Silencing in Saccharomyces cerevisiae. Genetics. 153(1). 13–23. 27 indexed citations
5.
Rivier, David H., Joanne Ekena, & Jasper Rine. (1999). HMR-I Is an Origin of Replication and a Silencer in Saccharomyces cerevisiae. Genetics. 151(2). 521–529. 46 indexed citations
6.
Xu, Eugenia Y., Hanna Kim, & David H. Rivier. (1999). SAS4 and SAS5 Are Locus-Specific Regulators of Silencing in Saccharomyces cerevisiae. Genetics. 153(1). 25–33. 32 indexed citations
7.
Ehrenhofer‐Murray, Ann E., David H. Rivier, & Jasper Rine. (1997). The Role of Sas2, an Acetyltransferase Homologue of Saccharomyces cerevisiae, in Silencing and ORC Function. Genetics. 145(4). 923–934. 115 indexed citations
8.
Rivier, David H. & Lorraine Pillus. (1994). Silencing speaks up. Cell. 76(6). 963–966. 21 indexed citations
9.
Rivier, David H. & Jasper Rine. (1992). Silencing: the establishment and inheritance of stable, repressed transcription states. Current Opinion in Genetics & Development. 2(2). 286–292. 37 indexed citations
10.
Rivier, David H. & Jasper Rine. (1992). An Origin of DNA Replication and a Transcription Silencer Require a Common Element. Science. 256(5057). 659–663. 129 indexed citations
11.
Rivier, David H. & Jasper Rine. (1992). Silencing of transcription. Current Biology. 2(4). 206–206. 1 indexed citations
12.
Young, Lisa S., David H. Rivier, & Karen U. Sprague. (1991). Sequences Far Downstream from the Classical tRNA Promoter Elements Bind RNA Polymerase III Transcription Factors. Molecular and Cellular Biology. 11(3). 1382–1392. 22 indexed citations
13.
Ottonello, Simone, et al.. (1987). The properties of a new polymerase III transcription factor reveal that transcription complexes can assemble by more than one pathway.. The EMBO Journal. 6(7). 1921–1927. 75 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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