David H. Barch

558 total citations
18 papers, 445 citations indexed

About

David H. Barch is a scholar working on Nutrition and Dietetics, Oncology and Rheumatology. According to data from OpenAlex, David H. Barch has authored 18 papers receiving a total of 445 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Nutrition and Dietetics, 4 papers in Oncology and 4 papers in Rheumatology. Recurrent topics in David H. Barch's work include Trace Elements in Health (6 papers), Drug Transport and Resistance Mechanisms (4 papers) and Pomegranate: compositions and health benefits (3 papers). David H. Barch is often cited by papers focused on Trace Elements in Health (6 papers), Drug Transport and Resistance Mechanisms (4 papers) and Pomegranate: compositions and health benefits (3 papers). David H. Barch collaborates with scholars based in United States, United Kingdom and Brazil. David H. Barch's co-authors include Christopher Toomajian, Anna Di Rienzo, Adrian V. S. Hill, Maria Luiza Petzl‐Erler, Peter Donnelly, Philip M. Iannaccone, Hitoshi Kawamata, Nan Li, Koji Kawai and Ryoichi Oyasu and has published in prestigious journals such as Gastroenterology, JNCI Journal of the National Cancer Institute and Genetics.

In The Last Decade

David H. Barch

17 papers receiving 415 citations

Peers

David H. Barch

GENET

ONCOL

Masakuni Sawaki Japan

Claudine Kavanaugh United States

Dilip Kumar Sinha United States

Monique Saintot France

Fred Buddingh United States

J.A. Lapré Netherlands

Catherine Pfent United States

Bruce Wr Canada

Meena Katdare United States

G. R. Wasson United Kingdom

Masakuni Sawaki Japan

David H. Barch

145 ×1.1

264 ×2.3

GENET

136 ×1.2

115 ×1.4

96 ×1.4

ONCOL

Citations per year, relative to David H. Barch David H. Barch (= 1×) peers Masakuni Sawaki

Countries citing papers authored by David H. Barch

Since Specialization

Citations

This map shows the geographic impact of David H. Barch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David H. Barch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David H. Barch more than expected).

Fields of papers citing papers by David H. Barch

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David H. Barch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David H. Barch. The network helps show where David H. Barch may publish in the future.

Co-authorship network of co-authors of David H. Barch

This figure shows the co-authorship network connecting the top 25 collaborators of David H. Barch. A scholar is included among the top collaborators of David H. Barch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David H. Barch. David H. Barch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Rienzo, Anna Di, Peter Donnelly, Christopher Toomajian, et al.. (1998). Heterogeneity of Microsatellite Mutations Within and Between Loci, and Implications for Human Demographic Histories. Genetics. 148(3). 1269–1284. 128 indexed citations

Kawamata, Hitoshi, Shuji Kameyama, Koji Kawai, et al.. (1995). Marked acceleration of the metastatic phenotype of a rat bladder carcinoma cell line by the expression of human gelatinase a. International Journal of Cancer. 63(4). 568–575. 55 indexed citations

Barch, David H., et al.. (1995). Ellagic acid induces transcription of the rat glutathione S-transferase-Ya gene. Carcinogenesis. 16(3). 665–668. 34 indexed citations

Barch, David H., et al.. (1994). Ellagic acid induces NAD(P)H:quinone reductase through activation of the antioxidant responsive element of the rat NAD(P)H:quinone reductase gene. Carcinogenesis. 15(9). 2065–2068. 28 indexed citations

Barch, David H., et al.. (1994). Dietary Ellagic Acid Inhibits the Enzymatic Activity of CYP1A1 without Altering Hepatic Concentrations of CYP1A1 or CYP1A1 mRNA. Biochemical and Biophysical Research Communications. 201(3). 1477–1482. 25 indexed citations

Barch, David H., et al.. (1992). Inhibition of rat methylbenzylnitrosamine metabolism by dietary zinc and zinc in vitro. Gastroenterology. 103(3). 800–806. 7 indexed citations

Barch, David H., et al.. (1990). The effect of mild stress on DMH-induced colorectal cancer.. PubMed. 14(5). 577–81. 1 indexed citations

Barch, David H., et al.. (1990). Sulfasalazine alters the character of dimethylhydrazine-induced colorectal carcinoma in rats.. PubMed. 9(6). 1725–8. 8 indexed citations

Roxe, David M., et al.. (1989). Phosphate-Binding Effects of Sucralfate in Patients With Chronic Renal Failure. American Journal of Kidney Diseases. 13(3). 194–199. 18 indexed citations

10.

Barch, David H., et al.. (1989). Dietary ellagic acid reduces the esophageal microsomal metabolism of methylbenzylnitrosamine. Cancer Letters. 44(1). 39–44. 19 indexed citations

11.

Barch, David H.. (1989). Esophageal cancer and microelements.. Journal of the American College of Nutrition. 8(2). 99–107. 10 indexed citations

12.

Mobarhan, Sohrab & David H. Barch. (1989). Nutritional management of patients with deglutition disorders transfer dysphagia and esophageal carcinoma. Diseases of the Esophagus. 2(1). 15–22. 1 indexed citations

13.

Barch, David H., et al.. (1988). Effect of Zinc Deficiency on Hepatic Enzymes Regulating Vitamin A Status. Journal of Nutrition. 118(8). 995–1001. 31 indexed citations

14.

Barch, David H., et al.. (1988). Selective inhibition of methylbenzylnitrosamine-induced formation of esophageal O6-methylguanine by dietary ellagic acid in rats.. PubMed. 48(24 Pt 1). 7088–92. 36 indexed citations

15.

Barch, David H., et al.. (1987). Dietary zinc deficiency increases the methylbenzylnitrosamine- induced formation of O⁶-methylguanine in the esophageal DNA of the rat. Carcinogenesis. 8(10). 1461–1464. 14 indexed citations

16.

Barch, David H., et al.. (1987). A simple system of feeding bottles for the study of zinc deficiency and ethanol consumption in the rat.. PubMed. 37(4). 504–6.

17.

Barch, David H. & Philip M. Iannaccone. (1986). Role of Zinc Deficiency in Carcinogenesis. PubMed. 206. 517–527. 26 indexed citations

18.

Barch, David H., et al.. (1986). Histopathology of Methylbenzylnitrosamine-Induced Esophageal Carcinoma in the Rat: Comparison With Cytomorphology<xref ref-type="fn" rid="FN2">2</xref>. JNCI Journal of the National Cancer Institute. 77(5). 1145–53. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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