David G. McVey

543 total citations
13 papers, 188 citations indexed

About

David G. McVey is a scholar working on Molecular Biology, Immunology and Cancer Research. According to data from OpenAlex, David G. McVey has authored 13 papers receiving a total of 188 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 5 papers in Immunology and 4 papers in Cancer Research. Recurrent topics in David G. McVey's work include RNA modifications and cancer (3 papers), RNA Research and Splicing (3 papers) and Atherosclerosis and Cardiovascular Diseases (3 papers). David G. McVey is often cited by papers focused on RNA modifications and cancer (3 papers), RNA Research and Splicing (3 papers) and Atherosclerosis and Cardiovascular Diseases (3 papers). David G. McVey collaborates with scholars based in United Kingdom, China and Singapore. David G. McVey's co-authors include Shu Ye, Xiaoxin Huang, Tom R. Webb, Nilesh J. Samani, Wei Yang, Catherine Andreadi, Peng Gong, Yang Xu, Johann Willeit and Stefan Kiechl and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and SHILAP Revista de lepidopterología.

In The Last Decade

David G. McVey

13 papers receiving 188 citations

Peers

David G. McVey
Adam W. Turner United States
Ayça Erbilgin United States
Peri Noori Sweden
David G. McVey
Citations per year, relative to David G. McVey David G. McVey (= 1×) peers Yonghui Zhao

Countries citing papers authored by David G. McVey

Since Specialization
Citations

This map shows the geographic impact of David G. McVey's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David G. McVey with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David G. McVey more than expected).

Fields of papers citing papers by David G. McVey

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David G. McVey. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David G. McVey. The network helps show where David G. McVey may publish in the future.

Co-authorship network of co-authors of David G. McVey

This figure shows the co-authorship network connecting the top 25 collaborators of David G. McVey. A scholar is included among the top collaborators of David G. McVey based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David G. McVey. David G. McVey is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
McVey, David G., Catherine Andreadi, Peng Gong, et al.. (2024). Genetic influence on vascular smooth muscle cell apoptosis. Cell Death and Disease. 15(6). 402–402. 1 indexed citations
2.
McVey, David G., et al.. (2023). Phenotypic Switching of Vascular Smooth Muscle Cells in Atherosclerosis. Journal of the American Heart Association. 12(20). e031121–e031121. 71 indexed citations
3.
Jiang, Min, Meixia Ren, Zhifeng Lu, et al.. (2023). LncRNA NIPA1-SO confers atherosclerotic protection by suppressing the transmembrane protein NIPA1. Journal of Advanced Research. 54. 29–42. 9 indexed citations
4.
Yang, Wei, Junjun Cao, David G. McVey, & Shu Ye. (2023). Allele-Specific Epigenetic Regulation of FURIN Expression at a Coronary Artery Disease Susceptibility Locus. Cells. 12(13). 1681–1681. 2 indexed citations
5.
McVey, David G., et al.. (2022). Relationship between Red Meat Metabolite Trimethylamine N-oxide and Cardiovascular Disease. SHILAP Revista de lepidopterología. 6(1). 3–9. 4 indexed citations
6.
McDonald, J., David G. McVey, Remo Guerrini, et al.. (2022). In vitro sepsis induces Nociceptin/Orphanin FQ receptor (NOP) expression in primary human vascular endothelial but not smooth muscle cells. PLoS ONE. 17(9). e0274080–e0274080. 3 indexed citations
7.
McVey, David G., Catherine Andreadi, Peng Gong, et al.. (2022). Effects of Coronary Artery Disease–Associated Variants on Vascular Smooth Muscle Cells. Circulation. 146(12). 917–929. 20 indexed citations
8.
Xu, Yang, Wei Yang, David G. McVey, et al.. (2020). FURIN Expression in Vascular Endothelial Cells Is Modulated by a Coronary Artery Disease–Associated Genetic Variant and Influences Monocyte Transendothelial Migration. Journal of the American Heart Association. 9(4). e014333–e014333. 27 indexed citations
9.
McVey, David G., Sander W. van der Laan, Kenneth Chan, et al.. (2020). The FES gene, located at the chromosome 15Q21.6 coronary-artery-disease locus, modulates atherosclerotic plaque vulnerability. Atherosclerosis. 315. e19–e19. 1 indexed citations
10.
Zhao, Guojun, Wei Yang, Jingchun Wu, et al.. (2018). Influence of a Coronary Artery Disease–Associated Genetic Variant on FURIN Expression and Effect of Furin on Macrophage Behavior. Arteriosclerosis Thrombosis and Vascular Biology. 38(8). 1837–1844. 29 indexed citations
11.
Jones, Peter D., et al.. (2016). The Coronary Artery Disease-associated Coding Variant in Zinc Finger C3HC-type Containing 1 (ZC3HC1) Affects Cell Cycle Regulation. Journal of Biological Chemistry. 291(31). 16318–16327. 12 indexed citations
12.
McVey, David G., Nilesh J. Samani, & Tom R. Webb. (2014). 214 Generation of Isogenic Cell Lines to Study a Single Disease Associated Variant at the 1P13 Cad Risk Locus. Heart. 100(Suppl 3). A117.2–A117. 1 indexed citations
13.
Veal, Colin, Hang Xu, Robert C. Free, et al.. (2013). Automated design of paralogue ratio test assays for the accurate and rapid typing of copy number variation. Bioinformatics. 29(16). 1997–2003. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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