David E. Godler

1.2k total citations
55 papers, 766 citations indexed

About

David E. Godler is a scholar working on Genetics, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, David E. Godler has authored 55 papers receiving a total of 766 indexed citations (citations by other indexed papers that have themselves been cited), including 53 papers in Genetics, 34 papers in Molecular Biology and 33 papers in Cognitive Neuroscience. Recurrent topics in David E. Godler's work include Genetics and Neurodevelopmental Disorders (47 papers), Autism Spectrum Disorder Research (33 papers) and Epigenetics and DNA Methylation (22 papers). David E. Godler is often cited by papers focused on Genetics and Neurodevelopmental Disorders (47 papers), Autism Spectrum Disorder Research (33 papers) and Epigenetics and DNA Methylation (22 papers). David E. Godler collaborates with scholars based in Australia, United States and Chile. David E. Godler's co-authors include David J. Amor, Minh Bui, Howard R. Slater, David Francis, Claudine M. Kraan, Danuta Z. Loesch, Randi J. Hagerman, Kim Cornish, Carolyn Rogers and John L. Hopper and has published in prestigious journals such as PLoS ONE, Neurology and Scientific Reports.

In The Last Decade

David E. Godler

53 papers receiving 754 citations

Peers

David E. Godler
Kathryn B. Garber United States
Kristin Herman United States
Stela Filipovic-Sadic United States
Darren Goffin United States
Anne Glicksman United States
Amy K. Sullivan United States
Angelika Rieß Germany
James Macpherson United Kingdom
Kristen M. West United States
Anthony Leotta United States
Kathryn B. Garber United States
David E. Godler
Citations per year, relative to David E. Godler David E. Godler (= 1×) peers Kathryn B. Garber

Countries citing papers authored by David E. Godler

Since Specialization
Citations

This map shows the geographic impact of David E. Godler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David E. Godler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David E. Godler more than expected).

Fields of papers citing papers by David E. Godler

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David E. Godler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David E. Godler. The network helps show where David E. Godler may publish in the future.

Co-authorship network of co-authors of David E. Godler

This figure shows the co-authorship network connecting the top 25 collaborators of David E. Godler. A scholar is included among the top collaborators of David E. Godler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David E. Godler. David E. Godler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Alshawsh, Mohammed Abdullah, Melissa Wake, Jozef Gécz, et al.. (2024). Epigenomic newborn screening for conditions with intellectual disability and autistic features in Australian newborns. Epigenomics. 16(18). 1203–1214. 2 indexed citations
2.
Crompton, Kylie, David E. Godler, Ling Ling, et al.. (2022). Umbilical Cord Blood Cell Clearance Post-Infusion in Immune-Competent Children with Cerebral Palsy. Cells Tissues Organs. 212(6). 546–553. 1 indexed citations
3.
Arpone, Marta, Lesley Bretherton, David J. Amor, et al.. (2022). Agreement between parents' and clinical researchers' ratings of behavioral problems in children with fragile X syndrome and chromosome 15 imprinting disorders. Research in Developmental Disabilities. 131. 104338–104338. 2 indexed citations
4.
5.
Archibald, Alison D., David Francis, Ling Ling, et al.. (2021). Paternal retraction of a fragile X allele to normal size, showing normal function over two generations. American Journal of Medical Genetics Part A. 188(1). 304–309. 1 indexed citations
6.
Kraan, Claudine M., Emma K. Baker, Marta Arpone, et al.. (2020). DNA Methylation at Birth Predicts Intellectual Functioning and Autism Features in Children with Fragile X Syndrome. International Journal of Molecular Sciences. 21(20). 7735–7735. 12 indexed citations
7.
Vos, Niels, et al.. (2020). Growth Trajectories in Genetic Subtypes of Prader–Willi Syndrome. Genes. 11(7). 736–736. 5 indexed citations
8.
Baker, Emma K., Merlin G. Butler, Ling Ling, et al.. (2020). Relationships between UBE3A and SNORD116 expression and features of autism in chromosome 15 imprinting disorders. Translational Psychiatry. 10(1). 362–362. 11 indexed citations
9.
Baker, Emma K., Marta Arpone, Claudine M. Kraan, et al.. (2020). FMR1 mRNA from full mutation alleles is associated with ABC-CFX scores in males with fragile X syndrome. Scientific Reports. 10(1). 11701–11701. 12 indexed citations
10.
Godler, David E. & David J. Amor. (2019). DNA methylation analysis for screening and diagnostic testing in neurodevelopmental disorders. Essays in Biochemistry. 63(6). 785–795. 8 indexed citations
11.
Prawer, Yael, Matthew F. Hunter, Ling Ling, et al.. (2018). Prenatal Diagnosis of Fragile X Syndrome in a Twin Pregnancy Complicated by a Complete Retraction. Genes. 9(6). 287–287. 6 indexed citations
12.
Kraan, Claudine M., David E. Godler, & David J. Amor. (2018). Epigenetics of fragile X syndrome and fragile X‐related disorders. Developmental Medicine & Child Neurology. 61(2). 121–127. 46 indexed citations
13.
Baker, Emma K., David E. Godler, Minh Bui, et al.. (2018). Exploring autism symptoms in an Australian cohort of patients with Prader-Willi and Angelman syndromes. Journal of Neurodevelopmental Disorders. 10(1). 24–24. 21 indexed citations
14.
Dudding, Tracy, Marta Arpone, David Francis, et al.. (2016). Molecular Inconsistencies in a Fragile X Male with Early Onset Ataxia. Genes. 7(9). 68–68. 6 indexed citations
15.
Arpone, Marta, David Francis, Xin Li, et al.. (2016). Partially methylated alleles, microdeletion, and tissue mosaicism in a fragile X male with tremor and ataxia at 30 years of age: A case report. American Journal of Medical Genetics Part A. 170(12). 3327–3332. 20 indexed citations
16.
Slater, Howard R., David Francis, Desirée du Sart, et al.. (2015). Identification of Males with Cryptic Fragile X Alleles by Methylation-Specific Quantitative Melt Analysis. Clinical Chemistry. 62(2). 343–352. 25 indexed citations
17.
18.
Loesch, Danuta Z., David E. Godler, Andrew Evans, et al.. (2011). Evidence for the toxicity of bidirectional transcripts and mitochondrial dysfunction in blood associated with small CGG expansions in the FMR1 gene in patients with parkinsonism. Genetics in Medicine. 13(5). 392–399. 65 indexed citations
19.
Loesch, Danuta Z., David E. Godler, Mahmoud Shekari Khaniani, et al.. (2009). Linking the FMR1 alleles with small CGG expansions with neurodevelopmental disorders: Preliminary data suggest an involvement of epigenetic mechanisms. American Journal of Medical Genetics Part A. 149A(10). 2306–2310. 22 indexed citations
20.
Godler, David E., et al.. (2005). Parathyroid hormone-related peptide expression in rat collagen-induced arthritis. Lara D. Veeken. 44(9). 1122–1131. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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