David E. Garcı́a

2.5k total citations · 1 hit paper
50 papers, 1.8k citations indexed

About

David E. Garcı́a is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, David E. Garcı́a has authored 50 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 35 papers in Molecular Biology, 24 papers in Cellular and Molecular Neuroscience and 12 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in David E. Garcı́a's work include Ion channel regulation and function (28 papers), Neuroscience and Neuropharmacology Research (21 papers) and Cardiac electrophysiology and arrhythmias (10 papers). David E. Garcı́a is often cited by papers focused on Ion channel regulation and function (28 papers), Neuroscience and Neuropharmacology Research (21 papers) and Cardiac electrophysiology and arrhythmias (10 papers). David E. Garcı́a collaborates with scholars based in Mexico, United States and Spain. David E. Garcı́a's co-authors include Ken Mackie, Bertil Hille, Todd Scheuer, Stefan Herlitze, William A. Catterall, Dan Lindholm, Benedikt Berninger, Sean M. Brown, Naoyuki Inagaki and Christian Hahnel and has published in prestigious journals such as Nature, Journal of Neuroscience and The Journal of Physiology.

In The Last Decade

David E. Garcı́a

45 papers receiving 1.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Modulation of Ca2+ channels βγ G-protein py subunits

1996 681 citations David E. Garcı́a, Ken Mackie et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David E. Garcı́a Mexico	14	1.0k	990	399	168	151	50	1.8k
Hiroyuki Katagiri Japan	16	577 0.6×	707 0.7×	126 0.3×	236 1.4×	215 1.4×	23	1.6k
Timothy Goodman United Kingdom	15	293 0.3×	174 0.2×	173 0.4×	115 0.7×	99 0.7×	18	936
Alexander K. Filippov United Kingdom	19	938 0.9×	666 0.7×	174 0.4×	24 0.1×	120 0.8×	29	1.3k
Yunfei Huang United States	17	1.0k 1.0×	561 0.6×	46 0.1×	81 0.5×	335 2.2×	29	1.8k
Adair J. Hotchkiss United States	12	710 0.7×	1.2k 1.2×	33 0.1×	84 0.5×	148 1.0×	14	1.4k
Céline S. Nicolas France	13	515 0.5×	346 0.3×	106 0.3×	51 0.3×	126 0.8×	23	965
Claude Walzer Switzerland	21	571 0.6×	407 0.4×	35 0.1×	78 0.5×	179 1.2×	42	1.2k
Keith S. Elmslie United States	28	1.6k 1.6×	1.0k 1.1×	392 1.0×	70 0.4×	181 1.2×	56	1.9k
Jawed Hamid Canada	25	1.9k 1.8×	1.3k 1.3×	338 0.8×	63 0.4×	449 3.0×	30	2.3k
Marco Weiergräber Germany	22	809 0.8×	812 0.8×	205 0.5×	67 0.4×	197 1.3×	65	1.4k

Countries citing papers authored by David E. Garcı́a

Since Specialization

Citations

This map shows the geographic impact of David E. Garcı́a's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David E. Garcı́a with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David E. Garcı́a more than expected).

Fields of papers citing papers by David E. Garcı́a

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David E. Garcı́a. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David E. Garcı́a. The network helps show where David E. Garcı́a may publish in the future.

Co-authorship network of co-authors of David E. Garcı́a

This figure shows the co-authorship network connecting the top 25 collaborators of David E. Garcı́a. A scholar is included among the top collaborators of David E. Garcı́a based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David E. Garcı́a. David E. Garcı́a is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Garcı́a, David E., R. Stone, Hadar Lev‐Tov, et al.. (2025). The impact of innate immunity and epigenetics in the pathogenesis of hidradenitis suppurativa. Frontiers in Immunology. 16. 1593253–1593253. 2 indexed citations

Julián-Jiménez, Agustín, et al.. (2024). Diagnostic accuracy of procalcitonin for bacterial infection in the Emergency Department: a systematic review. Revista Clínica Española (English Edition). 224(6). 400–416.

Pimentel, Jaime A., et al.. (2024). AMPK inhibits voltage-gated calcium channel-current in rat chromaffin cells. Molecular and Cellular Endocrinology. 591. 112275–112275.

Galié, Serena, et al.. (2021). Examining the Interaction of the Gut Microbiome with Host Metabolism and Cardiometabolic Health in Metabolic Syndrome. Nutrients. 13(12). 4318–4318. 8 indexed citations

Galié, Serena, Jesús García‐Gavilán, Lucía Camacho‐Barcia, et al.. (2021). Effects of the Mediterranean Diet or Nut Consumption on Gut Microbiota Composition and Fecal Metabolites and their Relationship with Cardiometabolic Risk Factors. Molecular Nutrition & Food Research. 65(19). e2000982–e2000982. 31 indexed citations

Bernáldez-Sarabia, Johanna, Tanya A. Camacho-Villegas, Luis A. Medina, et al.. (2021). Potential Therapeutic Applications of Synthetic Conotoxin s-cal14.2b, Derived from Californiconus californicus, for Treating Type 2 Diabetes. Biomedicines. 9(8). 936–936. 6 indexed citations

Elı́as-Viñas, David, et al.. (2021). Motor learning impairment in rats under a high sucrose diet. Physiology & Behavior. 234. 113384–113384. 8 indexed citations

Garcı́a, David E., et al.. (2020). Maintenance of CaV2.2 channel-current by PIP2 unveiled by neomycin in sympathetic neurons of the rat. Archives of Biochemistry and Biophysics. 682. 108261–108261. 6 indexed citations

Garduño, Julieta, et al.. (2017). Fast Inactivation of CaV2.2 Channels Is Prevented by the Gβ1 Subunit in Rat Sympathetic Neurons. Journal of Molecular Neuroscience. 63(3-4). 377–384. 1 indexed citations

10.

Elı́as-Viñas, David, et al.. (2016). Voltage-Independent Inhibition of the Tetrodotoxin-Sensitive Sodium Currents by Oxotremorine and Angiotensin II in Rat Sympathetic Neurons. Molecular Pharmacology. 89(4). 476–483. 1 indexed citations

11.

Halvorsen, Sigrun, Dan Atar, H. Yang, et al.. (2014). Efficacy and safety of apixaban compared with warfarin according to age for stroke prevention in atrial fibrillation: observations from the ARISTOTLE trial. European Heart Journal. 35(28). 1864–1872. 262 indexed citations

12.

Vivas, Oscar, et al.. (2014). Gβ2 mimics activation kinetic slowing of CaV2.2 channels by noradrenaline in rat sympathetic neurons. Biochemical and Biophysical Research Communications. 445(1). 250–254. 4 indexed citations

13.

Vivas, Oscar, et al.. (2013). Hydrolysis of PIP2 is Responsible for the Voltage-Independent Inhibition of Cav2.2 Channels. Biophysical Journal. 104(2). 460a–461a. 1 indexed citations

14.

Aláez, C., et al.. (2011). Major histocompatibility complex and strong human leukocyte antigen–DRB1 and gender association with Vogt–Koyanagi–Harada syndrome in Mexican Mestizos. Human Immunology. 72(12). 1198–1203. 15 indexed citations

15.

Garcı́a, David E., et al.. (2009). Gating charges per channel of CaV2.2 channels are modified by G protein activation in rat sympathetic neurons. Archives of Biochemistry and Biophysics. 486(1). 51–57. 7 indexed citations

16.

Garcı́a, David E., et al.. (2008). PMA counteracts G protein actions on CaV2.2 channels in rat sympathetic neurons. Archives of Biochemistry and Biophysics. 473(1). 1–7. 6 indexed citations

17.

Hernández‐Ochoa, Erick O., et al.. (2007). G protein activation inhibits gating charge movement in rat sympathetic neurons. American Journal of Physiology-Cell Physiology. 292(6). C2226–C2238. 11 indexed citations

18.

Garcı́a, David E., et al.. (2001). The antidepressant imipramine inhibits the M-type K+ current in rat sympathetic neurons. Neuroreport. 12(10). 2195–2198. 7 indexed citations

19.

Morgado‐Valle, Consuelo, et al.. (1998). The role of voltage-gated Ca 2+ channels in neurite growth of cultured chromaffin cells induced by extremely low frequency (ELF) magnetic field stimulation. Cell and Tissue Research. 291(2). 217–230. 81 indexed citations

20.

Berninger, Benedikt, David E. Garcı́a, Naoyuki Inagaki, Christian Hahnel, & Dan Lindholm. (1993). BDNF and NT-3 induce intracellular Ca2+ elevation in hippocampal neurones. Neuroreport. 4(12). 1303–1306. 171 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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