David D. Hebrink
- Immunology top 10%
- Immunotherapy and Immune Responses 4
- T-cell and B-cell Immunology 4
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 2
- Immune Cell Function and Interaction 2
- Immune Response and Inflammation 2
- Pathology and Forensic Medicine top 10%
- Multiple Sclerosis Research Studies 5
- Rheumatology top 10%
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- Cytokine Signaling Pathways and Interactions 3
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- Monoclonal and Polyclonal Antibodies Research 2
- Co-authors
- Brian G. WeinshenkerElizabeth J. AtkinsonOrhun H. KantarciCynthia T. McMurrayMariza de AndradeSara J. AchenbachJanet Schaefer-KleinMarcelo Matiello
- Partner nations
- United StatesUnited KingdomAustria
In The Last Decade
David D. Hebrink
16 papers receiving 483 citations
Peers
Comparison fields: 5 of 66
- Immunology 264
- Pathology and Forensic Medicine 207
- Rheumatology 103
- Biological Psychiatry 10
- Neurology 26
Countries citing papers authored by David D. Hebrink
This map shows the geographic impact of David D. Hebrink's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David D. Hebrink with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David D. Hebrink more than expected).
Fields of papers citing papers by David D. Hebrink
This network shows the impact of papers produced by David D. Hebrink. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David D. Hebrink. The network helps show where David D. Hebrink may publish in the future.
Co-authorship network
The 25 scholars most cited alongside David D. Hebrink, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2011 | 40 | |
| 2 | 2008 | 30 | |
| 3 | 2005 | 30 | |
| 4 | 2005 | 46 | |
| 5 | 2004 | 62 | |
| 6 | 2003 | 70 | |
| 7 | 2003 | 27 | |
| 8 | 2002 | 36 | |
| 9 | 2001 | 16 | |
| 10 | 2001 | 9 | |
| 11 | 2000 | 29 | |
| 12 | 2000 | 54 | |
| 13 | 2000 | 7 | |
| 14 | 1999 | 15 | |
| 15 | 1998 | 1 | |
| 16 | 1997 | 23 |
About David D. Hebrink
David D. Hebrink is a scholar working on Immunology, Pathology and Forensic Medicine and Physiology, having authored 16 papers that have together received 495 indexed citations. Recurring topics across this work include Multiple Sclerosis Research Studies (5 papers), Immunotherapy and Immune Responses (4 papers), T-cell and B-cell Immunology (4 papers), Cytokine Signaling Pathways and Interactions (3 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers), Immune Cell Function and Interaction (2 papers), Immune Response and Inflammation (2 papers) and Monoclonal and Polyclonal Antibodies Research (2 papers). The work is most often cited by research in Immunology (264 citations), Pathology and Forensic Medicine (207 citations) and Rheumatology (103 citations). David D. Hebrink has collaborated with scholars based in United States, United Kingdom and Austria. Frequent co-authors include Brian G. Weinshenker, Elizabeth J. Atkinson, Orhun H. Kantarci, Cynthia T. McMurray, Mariza de Andrade, Sara J. Achenbach, Janet Schaefer-Klein, Marcelo Matiello, Guy J. Buckle and Alicja Waliszewska. Their work appears in journals such as Neurology, Mayo Clinic Proceedings and Journal of Neuroimmunology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.