David Capellaro
Impact in
- Hematology top 5%
- Acute Myeloid Leukemia Research
- Chronic Myeloid Leukemia Treatments
- Genetics top 10%
- Chronic Lymphocytic Leukemia Research
Papers in
-
- Chronic Myeloid Leukemia Treatments 4
- Acute Myeloid Leukemia Research 2
- Genetics 2
- Chronic Lymphocytic Leukemia Research 2
- Co-authors
- Melvyn F. GreavesA H GoldstoneG. E. BrownJeremy R. JassGeorge JanossyPeter N. RiddleRob ShieldsRobert F. Brooks
- Journals
- British Journal of Haematology (3 papers)Journal of Clinical Pathology (1 paper)JNCI Journal of the National Cancer Institute (1 paper)Leukemia Research (1 paper)Nature (1 paper)
- Partner nations
- United KingdomUnited StatesCanada
In The Last Decade
David Capellaro
12 papers receiving 596 citations
Peers
Comparison fields: 5 of 69
- Hematology 190
- Genetics 97
- Pathology and Forensic Medicine 151
- Immunology 128
- Oncology 162
Countries citing papers authored by David Capellaro
This map shows the geographic impact of David Capellaro's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Capellaro with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Capellaro more than expected).
Fields of papers citing papers by David Capellaro
This network shows the impact of papers produced by David Capellaro. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Capellaro. The network helps show where David Capellaro may publish in the future.
Co-authorship network
The 25 scholars most cited alongside David Capellaro, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1989 | 73 | |
| 2 | 1988 | 99 | |
| 3 | 1987 | 14 | |
| 4 | 1986 | 17 | |
| 5 | 1978 | 83 | |
| 6 | 1978 | 97 | |
| 7 | 1978 | 75 | |
| 8 | 1977 | 80 | |
| 9 | 1977 | 18 | |
| 10 | 1976 | 20 | |
| 11 | 1976 | 6 | |
| 12 | 1975 | 82 |
About David Capellaro
David Capellaro is a scholar working on Hematology, Genetics, Biophysics, Immunology and Pathology and Forensic Medicine, having authored 12 papers that have together received 664 indexed citations. Recurring topics across this work include Chronic Myeloid Leukemia Treatments (4 papers), Acute Lymphoblastic Leukemia research (3 papers), Acute Myeloid Leukemia Research (2 papers), Chronic Lymphocytic Leukemia Research (2 papers), Immunotherapy and Immune Responses (2 papers), Immunodeficiency and Autoimmune Disorders (2 papers), RNA Interference and Gene Delivery (2 papers) and Glycosylation and Glycoproteins Research (1 paper). The work is most often cited by research in Hematology (190 citations), Genetics (97 citations), Pathology and Forensic Medicine (151 citations), Immunology (128 citations) and Oncology (162 citations). David Capellaro has collaborated with scholars based in United Kingdom, United States and Canada. Frequent co-authors include Melvyn F. Greaves, A H Goldstone, G. E. Brown, Jeremy R. Jass, George Janossy, Peter N. Riddle, Rob Shields, Robert F. Brooks, G. E. Francis and H. S. Goh. Their work appears in journals such as British Journal of Haematology, Journal of Clinical Pathology, JNCI Journal of the National Cancer Institute, Leukemia Research and Nature.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.