David Camerini

2.7k total citations
43 papers, 2.1k citations indexed

About

David Camerini is a scholar working on Virology, Immunology and Infectious Diseases. According to data from OpenAlex, David Camerini has authored 43 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Virology, 16 papers in Immunology and 14 papers in Infectious Diseases. Recurrent topics in David Camerini's work include HIV Research and Treatment (18 papers), HIV/AIDS drug development and treatment (9 papers) and Immune Cell Function and Interaction (8 papers). David Camerini is often cited by papers focused on HIV Research and Treatment (18 papers), HIV/AIDS drug development and treatment (9 papers) and Immune Cell Function and Interaction (8 papers). David Camerini collaborates with scholars based in United States, Netherlands and Japan. David Camerini's co-authors include Brian Seed, Adrian R.L. Gear, Stephen P. James, Gerd Walz, Wil A. M. Loenen, Jannie Borst, Shailesh K. Choudhary, Hanneke Schuitemaker, Vicente Planelles and Stuart F. Schlossman and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

David Camerini

43 papers receiving 2.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David Camerini United States 23 939 598 546 382 378 43 2.1k
Dominique Rigal France 23 779 0.8× 458 0.8× 639 1.2× 395 1.0× 227 0.6× 67 2.2k
April Chiu United States 18 1.4k 1.5× 358 0.6× 614 1.1× 383 1.0× 255 0.7× 64 2.6k
L D Shultz United States 17 1.5k 1.6× 339 0.6× 621 1.1× 399 1.0× 189 0.5× 30 2.7k
Josef Cihak Germany 23 1.9k 2.0× 329 0.6× 900 1.6× 663 1.7× 288 0.8× 44 3.1k
Yacov Ron United States 33 2.0k 2.2× 760 1.3× 1.0k 1.9× 392 1.0× 717 1.9× 79 3.8k
Gregory F. Burton United States 27 1.7k 1.8× 836 1.4× 433 0.8× 213 0.6× 426 1.1× 44 2.7k
Nicola Borthwick United Kingdom 25 2.0k 2.2× 781 1.3× 506 0.9× 425 1.1× 301 0.8× 58 2.8k
David Stephany United States 21 1.7k 1.8× 257 0.4× 404 0.7× 401 1.0× 247 0.7× 35 2.6k
Claudia Cicala United States 32 1.7k 1.8× 1.4k 2.3× 725 1.3× 311 0.8× 541 1.4× 72 3.0k
Thomas M. McHugh United States 22 806 0.9× 816 1.4× 240 0.4× 178 0.5× 489 1.3× 36 2.1k

Countries citing papers authored by David Camerini

Since Specialization
Citations

This map shows the geographic impact of David Camerini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Camerini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Camerini more than expected).

Fields of papers citing papers by David Camerini

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Camerini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Camerini. The network helps show where David Camerini may publish in the future.

Co-authorship network of co-authors of David Camerini

This figure shows the co-authorship network connecting the top 25 collaborators of David Camerini. A scholar is included among the top collaborators of David Camerini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Camerini. David Camerini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Campo, Joseph J., Rebecca M. Carpenter, Mary K. Young, et al.. (2021). Diverse Humoral Immune Responses in Younger and Older Adult COVID-19 Patients. mBio. 12(3). e0122921–e0122921. 13 indexed citations
2.
Camerini, David, et al.. (2017). NP-40 Fractionation and Nucleic Acid Extraction in Mammalian Cells. BIO-PROTOCOL. 7(20). e2584–e2584. 4 indexed citations
3.
4.
Richardson, Barbra A., Benson Singa, Vladimir E. Diaz-Ochoa, et al.. (2013). Use of Principal Components Analysis and Protein Microarray to Explore the Association of HIV-1-Specific IgG Responses with Disease Progression. AIDS Research and Human Retroviruses. 30(1). 37–44. 2 indexed citations
5.
Soto, Maira, et al.. (2012). Antibacterial Activity of Four Human Beta-Defensins: HBD-19, HBD-23, HBD-27, and HBD-29. Polymers. 4(1). 747–758. 8 indexed citations
6.
Ye, Ying, Lesley R. de Armas, Maira Soto, et al.. (2010). Cyclic and Acyclic Defensins Inhibit Human Immunodeficiency Virus Type-1 Replication by Different Mechanisms. PLoS ONE. 5(3). e9737–e9737. 68 indexed citations
7.
Choudhary, Shailesh K., Nienke Vrisekoop, Christine A. Jansen, et al.. (2007). Low Immune Activation despite High Levels of Pathogenic Human Immunodeficiency Virus Type 1 Results in Long-Term Asymptomatic Disease. Journal of Virology. 81(16). 8838–8842. 75 indexed citations
8.
Olivieri, Kevin C., Aprille L. Matthews, Mark David, et al.. (2006). The envelope gene is a cytopathic determinant of CCR5 tropic HIV-1. Virology. 358(1). 23–38. 22 indexed citations
9.
10.
Neuveut, Christine, et al.. (2003). Requirement for the Second Coding Exon of Tat in the Optimal Replication of Macrophage-Tropic HIV-1. Journal of Biomedical Science. 10(6). 651–660. 21 indexed citations
11.
Neuveut, Christine, et al.. (2003). Requirement for the second coding exon of Tat in the optimal replication of macrophage-tropic HIV-1. Journal of Biomedical Science. 10(6). 651–660. 17 indexed citations
12.
Gear, Adrian R.L. & David Camerini. (2003). Platelet Chemokines and Chemokine Receptors: Linking Hemostasis, Inflammation, and Host Defense. Microcirculation. 10(3-4). 335–350. 281 indexed citations
13.
Gear, Adrian R.L., et al.. (2001). Adenosine diphosphate strongly potentiates the ability of the chemokines MDC, TARC, and SDF-1 to stimulate platelet function. Blood. 97(4). 937–945. 81 indexed citations
14.
Taylor, James R., et al.. (2000). Pathogenesis of Primary R5 Human Immunodeficiency Virus Type 1 Clones in SCID-hu Mice. Journal of Virology. 74(7). 3205–3216. 43 indexed citations
15.
Tajima, Yasutaka, Kunio Tashiro, & David Camerini. (1997). Assignment of the possible HTLV receptor gene to chromosome 17q21-q23. Somatic Cell and Molecular Genetics. 23(3). 225–227. 7 indexed citations
16.
Hegen, Martin, David Camerini, & Bernhard Fleischer. (1993). Function of Dipeptidyl Peptidase IV (CD26, Tp103) in Transfected Human T Cells. Cellular Immunology. 146(2). 249–260. 31 indexed citations
17.
Berg, Maria, Yohko Murakawa, David Camerini, & Stephen P. James. (1991). Lamina propria lymphocytes are derived from circulating cells that lack the Leu-8 lymph node homing receptor. Gastroenterology. 101(1). 90–99. 27 indexed citations
18.
Camerini, David & Brian Seed. (1990). A CD4 domain important for HIV-mediated syncytium formation lies outside the virus binding site. Cell. 60(5). 747–754. 153 indexed citations
19.
Camerini, David, et al.. (1989). Leu-8/TQ1 is the human equivalent of the Mel-14 lymph node homing receptor. Nature. 342(6245). 78–82. 219 indexed citations
20.
Lazarovits, A I, Richard Moscicki, J T Kurnick, et al.. (1984). Lymphocyte activation antigens. I. A monoclonal antibody, anti-Act I, defines a new late lymphocyte activation antigen.. The Journal of Immunology. 133(4). 1857–1862. 127 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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