David B. Murray

2.2k total citations · 1 hit paper
24 papers, 1.8k citations indexed

About

David B. Murray is a scholar working on Cardiology and Cardiovascular Medicine, Physiology and Pathology and Forensic Medicine. According to data from OpenAlex, David B. Murray has authored 24 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Cardiology and Cardiovascular Medicine, 8 papers in Physiology and 5 papers in Pathology and Forensic Medicine. Recurrent topics in David B. Murray's work include Cardiac Fibrosis and Remodeling (8 papers), Cardiovascular Function and Risk Factors (5 papers) and Nitric Oxide and Endothelin Effects (4 papers). David B. Murray is often cited by papers focused on Cardiac Fibrosis and Remodeling (8 papers), Cardiovascular Function and Risk Factors (5 papers) and Nitric Oxide and Endothelin Effects (4 papers). David B. Murray collaborates with scholars based in United States, United Kingdom and Japan. David B. Murray's co-authors include Gökhan S. Hotamışlıgil, Adriane I. Budavari, Bruce M. Spiegelman, Gregory L. Brower, Joseph S. Janicki, Scott P. Levick, John Baynes, Jason D. Gardner, Ryoji Nagai and Thomas Metz and has published in prestigious journals such as The Lancet, Journal of Clinical Investigation and Diabetes.

In The Last Decade

David B. Murray

23 papers receiving 1.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Reduced tyrosine kinase activity of the insulin receptor in obesity-diabetes. Central role of tumor necrosis factor-alpha.

1994 658 citations Gökhan S. Hotamışlıgil, Adriane I. Budavari et al. Journal of Clinical Investigation profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David B. Murray United States	16	624	459	451	425	288	24	1.8k
Jeffrey G. Dickhout Canada	30	417 0.7×	475 1.0×	959 2.1×	488 1.1×	198 0.7×	50	2.8k
Clara Di Filippo Italy	25	593 1.0×	453 1.0×	1.0k 2.3×	195 0.5×	426 1.5×	58	2.4k
Stephan Schiekofer Germany	19	637 1.0×	602 1.3×	939 2.1×	595 1.4×	465 1.6×	47	2.6k
Tomoko Asagami United States	12	628 1.0×	582 1.3×	268 0.6×	150 0.4×	285 1.0×	15	1.6k
Yukihiro Takemura Japan	16	531 0.9×	652 1.4×	570 1.3×	953 2.2×	165 0.6×	25	2.1k
Xue-Liang Du United States	6	222 0.4×	549 1.2×	660 1.5×	334 0.8×	384 1.3×	6	2.0k
Souad Belmadani United States	20	387 0.6×	429 0.9×	577 1.3×	228 0.5×	192 0.7×	35	1.7k
Karla B Neves United Kingdom	27	574 0.9×	430 0.9×	560 1.2×	254 0.6×	301 1.0×	57	1.9k
Jee‐Young Han South Korea	29	251 0.4×	293 0.6×	670 1.5×	338 0.8×	570 2.0×	56	2.1k
Nassim Dali‐Youcef France	18	321 0.5×	492 1.1×	659 1.5×	332 0.8×	229 0.8×	37	1.8k

Countries citing papers authored by David B. Murray

Since Specialization

Citations

This map shows the geographic impact of David B. Murray's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David B. Murray with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David B. Murray more than expected).

Fields of papers citing papers by David B. Murray

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David B. Murray. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David B. Murray. The network helps show where David B. Murray may publish in the future.

Co-authorship network of co-authors of David B. Murray

This figure shows the co-authorship network connecting the top 25 collaborators of David B. Murray. A scholar is included among the top collaborators of David B. Murray based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David B. Murray. David B. Murray is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Clark, Jessica, Charity Fix, Brittany A. Law, et al.. (2013). Type II diabetes promotes a myofibroblast phenotype in cardiac fibroblasts. Life Sciences. 92(11). 669–676. 69 indexed citations

Nagai, Ryoji, David B. Murray, Thomas Metz, & John Baynes. (2012). Chelation: A Fundamental Mechanism of Action of AGE Inhibitors, AGE Breakers, and Other Inhibitors of Diabetes Complications. Diabetes. 61(3). 549–559. 182 indexed citations

Harding, Pamela & David B. Murray. (2011). The contribution of prostaglandins versus prostacyclin in ventricular remodeling during heart failure. Life Sciences. 89(19-20). 671–676. 17 indexed citations

Murray, David B., et al.. (2011). Tryptase activates isolated adult cardiac fibroblasts via protease activated receptor-2 (PAR-2). Journal of Cell Communication and Signaling. 6(1). 45–51. 18 indexed citations

Levick, Scott P., David B. Murray, Joseph S. Janicki, & Gregory L. Brower. (2010). Sympathetic Nervous System Modulation of Inflammation and Remodeling in the Hypertensive Heart. Hypertension. 55(2). 270–276. 124 indexed citations

Murray, David B., Scott P. Levick, Gregory L. Brower, & Joseph S. Janicki. (2010). Inhibition of matrix metalloproteinase activity prevents increases in myocardial tumor necrosis factor-α. Journal of Molecular and Cellular Cardiology. 49(2). 245–250. 12 indexed citations

Baynes, John & David B. Murray. (2009). Cardiac and Renal Function are Progressively Impaired with Aging in Zucker Diabetic Fatty Type II Diabetic Rats. Oxidative Medicine and Cellular Longevity. 2(5). 328–334. 35 indexed citations

Murray, David B., et al.. (2009). ET_Aselective receptor antagonism prevents ventricular remodeling in volume-overloaded rats. American Journal of Physiology-Heart and Circulatory Physiology. 297(1). H109–H116. 7 indexed citations

Baynes, John & David B. Murray. (2009). The Metal Chelators, Trientine and Citrate, Inhibit the Development of Cardiac Pathology in the Zucker Diabetic Rat. Experimental Diabetes Research. 2009. 1–6. 29 indexed citations

10.

Levick, Scott P., et al.. (2009). Cardiac Mast Cells Mediate Left Ventricular Fibrosis in the Hypertensive Rat Heart. Hypertension. 53(6). 1041–1047. 131 indexed citations

11.

Levick, Scott P., Tetyana G Voloshenyuk, David B. Murray, et al.. (2008). A novel technique for isolating functional mast cells from the heart. Inflammation Research. 57(5). 241–246. 17 indexed citations

12.

Murray, David B., Jason D. Gardner, Gregory L. Brower, & Joseph S. Janicki. (2008). Effects of nonselective endothelin-1 receptor antagonism on cardiac mast cell-mediated ventricular remodeling in rats. American Journal of Physiology-Heart and Circulatory Physiology. 294(3). H1251–H1257. 31 indexed citations

13.

Ding, Yanfeng, Gregory L. Brower, David B. Murray, et al.. (2008). DEFECTIVE INTRACELLULAR Ca²⁺ HOMEOSTASIS CONTRIBUTES TO MYOCYTE DYSFUNCTION DURING VENTRICULAR REMODELLING INDUCED BY CHRONIC VOLUME OVERLOAD IN RATS. Clinical and Experimental Pharmacology and Physiology. 35(7). 827–835. 14 indexed citations

14.

Murray, David B., et al.. (2007). Response of cardiac mast cells to atrial natriuretic peptide. American Journal of Physiology-Heart and Circulatory Physiology. 293(2). H1216–H1222. 11 indexed citations

15.

Brower, Gregory L., Jason D. Gardner, Mary F. Forman, et al.. (2006). The relationship between myocardial extracellular matrix remodeling and ventricular function☆. European Journal of Cardio-Thoracic Surgery. 30(4). 604–610. 224 indexed citations

16.

Chancey, Amanda L., Jason D. Gardner, David B. Murray, Gregory L. Brower, & Joseph S. Janicki. (2005). Modulation of cardiac mast cell-mediated extracellular matrix degradation by estrogen. American Journal of Physiology-Heart and Circulatory Physiology. 289(1). H316–H321. 25 indexed citations

17.

Murray, David B., Jason D. Gardner, Gregory L. Brower, & Joseph S. Janicki. (2004). Endothelin-1 mediates cardiac mast cell degranulation, matrix metalloproteinase activation, and myocardial remodeling in rats. American Journal of Physiology-Heart and Circulatory Physiology. 287(5). H2295–H2299. 76 indexed citations

18.

Hotamışlıgil, Gökhan S., Adriane I. Budavari, David B. Murray, & Bruce M. Spiegelman. (1994). Reduced tyrosine kinase activity of the insulin receptor in obesity-diabetes. Central role of tumor necrosis factor-alpha.. Journal of Clinical Investigation. 94(4). 1543–1549. 658 indexed citations breakdown →

19.

Murray, David B.. (1967). CORTISOL BINDING TO PLASMA PROTEINS IN MAN IN HEALTH, STRESS AND AT DEATH. Journal of Endocrinology. 39(4). 571–591. 47 indexed citations

20.

Murray, David B., et al.. (1956). EFFECT OF CARBUTAMIDE ON SERUMCHOLESTEROL LEVEL IN DIABETES MELLITUS. The Lancet. 268(6952). 1083–1084. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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